Jay L. Grisolano

854 total citations
7 papers, 678 citations indexed

About

Jay L. Grisolano is a scholar working on Molecular Biology, Hematology and Oncology. According to data from OpenAlex, Jay L. Grisolano has authored 7 papers receiving a total of 678 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Hematology and 2 papers in Oncology. Recurrent topics in Jay L. Grisolano's work include Acute Myeloid Leukemia Research (4 papers), RNA Interference and Gene Delivery (2 papers) and Cell Adhesion Molecules Research (2 papers). Jay L. Grisolano is often cited by papers focused on Acute Myeloid Leukemia Research (4 papers), RNA Interference and Gene Delivery (2 papers) and Cell Adhesion Molecules Research (2 papers). Jay L. Grisolano collaborates with scholars based in United States, Italy and Canada. Jay L. Grisolano's co-authors include Timothy J. Ley, Pier Giuseppe Pelicci, Robin L. Wesselschmidt, Jennifer Cain, Michael H. Tomasson, Julie O’Neal, T J Ley, Jessica L. Pollock, Peter Westervelt and A. McGarry Houghton and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Blood and Cancer Research.

In The Last Decade

Jay L. Grisolano

7 papers receiving 665 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jay L. Grisolano United States	6	507	451	116	110	92	7	678
AD D'Andrea United States	13	263 0.5×	336 0.7×	182 1.6×	78 0.7×	137 1.5×	20	667
N Aoki Japan	10	173 0.3×	233 0.5×	85 0.7×	52 0.5×	84 0.9×	12	446
Tara MacRae Canada	14	466 0.9×	374 0.8×	118 1.0×	67 0.6×	159 1.7×	25	735
CR Faltynek United States	9	197 0.4×	118 0.3×	85 0.7×	35 0.3×	153 1.7×	13	385
Sarah J. Horton United Kingdom	14	500 1.0×	409 0.9×	102 0.9×	106 1.0×	72 0.8×	21	729
Weiqi Huang United States	16	450 0.9×	177 0.4×	172 1.5×	90 0.8×	208 2.3×	36	689
Donata Verdelli Italy	10	398 0.8×	346 0.8×	145 1.3×	67 0.6×	32 0.3×	14	532
Lucia Nobili Italy	15	385 0.8×	219 0.5×	120 1.0×	136 1.2×	96 1.0×	24	597
E L Madison United States	8	276 0.5×	345 0.8×	81 0.7×	442 4.0×	36 0.4×	8	660
Ann Leary United States	5	185 0.4×	108 0.2×	130 1.1×	159 1.4×	52 0.6×	8	385

Countries citing papers authored by Jay L. Grisolano

Since Specialization

Citations

This map shows the geographic impact of Jay L. Grisolano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jay L. Grisolano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jay L. Grisolano more than expected).

Fields of papers citing papers by Jay L. Grisolano

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jay L. Grisolano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jay L. Grisolano. The network helps show where Jay L. Grisolano may publish in the future.

Co-authorship network of co-authors of Jay L. Grisolano

This figure shows the co-authorship network connecting the top 25 collaborators of Jay L. Grisolano. A scholar is included among the top collaborators of Jay L. Grisolano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jay L. Grisolano. Jay L. Grisolano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Houghton, A. McGarry, Jay L. Grisolano, Mary Baumann, et al.. (2006). Macrophage Elastase (Matrix Metalloproteinase-12) Suppresses Growth of Lung Metastases. Cancer Research. 66(12). 6149–6155. 122 indexed citations

2.

Cain, Jennifer, Jay L. Grisolano, Alexander Laird, & Michael H. Tomasson. (2004). Complete remission of TEL-PDGFRB–induced myeloproliferative disease in mice by receptor tyrosine kinase inhibitor SU11657. Blood. 104(2). 561–564. 12 indexed citations

3.

Grisolano, Jay L., Julie O’Neal, Jennifer Cain, & Michael H. Tomasson. (2003). An activated receptor tyrosine kinase, TEL/PDGFβR, cooperates with AML1/ETO to induce acute myeloid leukemia in mice. Proceedings of the National Academy of Sciences. 100(16). 9506–9511. 102 indexed citations

4.

Pollock, Jessica L., et al.. (1999). A bcr-3 isoform of RARα-PML potentiates the development of PML-RARα-driven acute promyelocytic leukemia. Proceedings of the National Academy of Sciences. 96(26). 15103–15108. 89 indexed citations

5.

Grisolano, Jay L., Robin L. Wesselschmidt, Pier Giuseppe Pelicci, & Timothy J. Ley. (1997). Altered Myeloid Development and Acute Leukemia in Transgenic Mice Expressing PML-RARα Under Control of Cathepsin G Regulatory Sequences. Blood. 89(2). 376–387. 296 indexed citations

6.

Grisolano, Jay L. & Timothy J. Ley. (1996). Regulation of azurophil granule-associated serine protease genes during myelopoiesis. Current Opinion in Hematology. 3(1). 55–62. 2 indexed citations

7.

Grisolano, Jay L., et al.. (1994). Early myeloid cell-specific expression of the human cathepsin G gene in transgenic mice.. Proceedings of the National Academy of Sciences. 91(19). 8989–8993. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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