Javier Alcácer

459 total citations
11 papers, 292 citations indexed

About

Javier Alcácer is a scholar working on Molecular Biology, Oncology and Geriatrics and Gerontology. According to data from OpenAlex, Javier Alcácer has authored 11 papers receiving a total of 292 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Oncology and 3 papers in Geriatrics and Gerontology. Recurrent topics in Javier Alcácer's work include Genomics, phytochemicals, and oxidative stress (4 papers), Estrogen and related hormone effects (3 papers) and Sirtuins and Resveratrol in Medicine (3 papers). Javier Alcácer is often cited by papers focused on Genomics, phytochemicals, and oxidative stress (4 papers), Estrogen and related hormone effects (3 papers) and Sirtuins and Resveratrol in Medicine (3 papers). Javier Alcácer collaborates with scholars based in Spain. Javier Alcácer's co-authors include Elena Obrador, María Benlloch, José M. Estrela, Soraya L. Vallés, José A. Pellicer, Rosario Salvador‐Palmer, Manuel Mata, J. Antoni Sirerol, María Sancho‐Tello and Lara Milián and has published in prestigious journals such as PLoS ONE, Cancer Research and American Journal Of Pathology.

In The Last Decade

Javier Alcácer

11 papers receiving 289 citations

Peers

Javier Alcácer
Bing Wen China
Yi‐Hsuan Lin Taiwan
Shengdan Nie China
Qiang Xue China
Minghua Li China
Qiong Jiang China
Bor‐Ren Huang Taiwan
Ju Hie Lee South Korea
Sara Bisetto United States
Israel López-Valero Spain
Bing Wen China
Javier Alcácer
Citations per year, relative to Javier Alcácer Javier Alcácer (= 1×) peers Bing Wen

Countries citing papers authored by Javier Alcácer

Since Specialization
Citations

This map shows the geographic impact of Javier Alcácer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Javier Alcácer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Javier Alcácer more than expected).

Fields of papers citing papers by Javier Alcácer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Javier Alcácer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Javier Alcácer. The network helps show where Javier Alcácer may publish in the future.

Co-authorship network of co-authors of Javier Alcácer

This figure shows the co-authorship network connecting the top 25 collaborators of Javier Alcácer. A scholar is included among the top collaborators of Javier Alcácer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Javier Alcácer. Javier Alcácer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Obrador, Elena, Rosario Salvador‐Palmer, Rafael López‐Blanch, et al.. (2020). Melanoma in the liver: Oxidative stress and the mechanisms of metastatic cell survival. Seminars in Cancer Biology. 71. 109–121. 17 indexed citations
2.
Obrador, Elena, Rosario Salvador‐Palmer, Patricia Marchio, et al.. (2020). Nicotinamide Riboside and Pterostilbene Cooperatively Delay Motor Neuron Failure in ALS SOD1G93A Mice. Molecular Neurobiology. 58(4). 1345–1371. 28 indexed citations
3.
4.
Obrador, Elena, José M. Estrela, Rosario Salvador‐Palmer, et al.. (2020). Glucocorticoid receptor antagonism overcomes resistance to BRAF inhibition in BRAFV600E-mutated metastatic melanoma. European Journal of Cancer. 138. S41–S41. 15 indexed citations
5.
Carretero, Julián, Antonio Cremades, Javier Alcácer, et al.. (2019). Clinical significance of epithelial–mesenchymal transition-related markers expression in the micrometastatic sentinel lymph node of NSCLC. Clinical & Translational Oncology. 22(3). 381–391. 8 indexed citations
6.
Estrela, José M., Rosario Salvador‐Palmer, Patricia Marchio, et al.. (2019). Glucocorticoid receptor antagonism overcomes resistance to BRAF inhibition in BRAFV600E-mutated metastatic melanoma.. PubMed. 9(12). 2580–2598. 15 indexed citations
7.
Revert, Fernando, Roberto Gozalbo‐Rovira, Jerónimo Forteza, et al.. (2018). Selective targeting of collagen IV in the cancer cell microenvironment reduces tumor burden. Oncotarget. 9(13). 11020–11045. 26 indexed citations
8.
Benlloch, María, Soraya L. Vallés, María L. Rodríguez, et al.. (2016). Abstract 2810: Pterostilbene, a natural phytoalexin, weakens the antioxidant defenses of aggressive cancer cells in vivo: a pituitary gland- and Nrf2-dependent mechanism. Cancer Research. 76(14_Supplement). 2810–2810. 1 indexed citations
9.
Benlloch, María, Elena Obrador, Soraya L. Vallés, et al.. (2015). Pterostilbene Decreases the Antioxidant Defenses of Aggressive Cancer Cells In Vivo : A Physiological Glucocorticoids- and Nrf2-Dependent Mechanism. Antioxidants and Redox Signaling. 24(17). 974–990. 56 indexed citations
10.
11.
Revert, Fernando, Ramón Merino, Carlos Monteagudo, et al.. (2007). Increased Goodpasture Antigen-Binding Protein Expression Induces Type IV Collagen Disorganization and Deposit of Immunoglobulin A in Glomerular Basement Membrane. American Journal Of Pathology. 171(5). 1419–1430. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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