Jar‐Yi Ho

1.2k total citations
32 papers, 892 citations indexed

About

Jar‐Yi Ho is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Jar‐Yi Ho has authored 32 papers receiving a total of 892 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 13 papers in Cancer Research and 6 papers in Oncology. Recurrent topics in Jar‐Yi Ho's work include Cancer-related molecular mechanisms research (8 papers), MicroRNA in disease regulation (6 papers) and Epigenetics and DNA Methylation (4 papers). Jar‐Yi Ho is often cited by papers focused on Cancer-related molecular mechanisms research (8 papers), MicroRNA in disease regulation (6 papers) and Epigenetics and DNA Methylation (4 papers). Jar‐Yi Ho collaborates with scholars based in Taiwan, Austria and United States. Jar‐Yi Ho's co-authors include Cheng‐Ping Yu, Dah‐Shyong Yü, Ren‐Jun Hsu, Chia‐Lun Wu, Tong‐Hong Wang, Tai‐Lung Cha, Jui‐Ming Liu, Chau‐Ting Yeh, Sheng‐Chieh Chou and Tse‐Ching Chen and has published in prestigious journals such as Journal of Clinical Oncology, PLoS ONE and Cancer Research.

In The Last Decade

Jar‐Yi Ho

29 papers receiving 881 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jar‐Yi Ho Taiwan 19 605 333 150 114 90 32 892
Ruth Sánchez‐Martínez Spain 15 667 1.1× 404 1.2× 147 1.0× 116 1.0× 64 0.7× 21 966
Ece Konaç Türkiye 19 468 0.8× 280 0.8× 145 1.0× 85 0.7× 88 1.0× 59 941
Baoen Shan China 18 626 1.0× 264 0.8× 188 1.3× 68 0.6× 84 0.9× 44 851
Yu Ding China 16 564 0.9× 317 1.0× 93 0.6× 113 1.0× 83 0.9× 49 862
Ying‐Erh Chou Taiwan 18 488 0.8× 233 0.7× 173 1.2× 67 0.6× 62 0.7× 45 733
Jie Lin China 21 749 1.2× 473 1.4× 185 1.2× 169 1.5× 75 0.8× 55 1.0k
Chundi Gao China 19 656 1.1× 426 1.3× 155 1.0× 194 1.7× 80 0.9× 47 944
Hua-Tao Wu China 15 476 0.8× 242 0.7× 221 1.5× 147 1.3× 73 0.8× 38 846
Huayao Li China 22 731 1.2× 422 1.3× 199 1.3× 178 1.6× 131 1.5× 55 1.2k
Neda Shajari Iran 10 616 1.0× 255 0.8× 148 1.0× 90 0.8× 85 0.9× 14 833

Countries citing papers authored by Jar‐Yi Ho

Since Specialization
Citations

This map shows the geographic impact of Jar‐Yi Ho's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jar‐Yi Ho with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jar‐Yi Ho more than expected).

Fields of papers citing papers by Jar‐Yi Ho

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jar‐Yi Ho. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jar‐Yi Ho. The network helps show where Jar‐Yi Ho may publish in the future.

Co-authorship network of co-authors of Jar‐Yi Ho

This figure shows the co-authorship network connecting the top 25 collaborators of Jar‐Yi Ho. A scholar is included among the top collaborators of Jar‐Yi Ho based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jar‐Yi Ho. Jar‐Yi Ho is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Siu, Brian, Alex Qinyang Liu, Chi‐Ho Leung, et al.. (2025). Treatment cycles per unit prostate volume (CPV) for transurethral water vapor therapy (Rezūm) in catheter-dependent patients. Prostate Cancer and Prostatic Diseases. 29(1). 132–137. 1 indexed citations
2.
Siu, Brian, Jar‐Yi Ho, Alex Qinyang Liu, et al.. (2025). Transurethral water vapor therapy (Rezūm) for acute urinary retention with obstructive uropathy: a prospective cohort study with one-year follow-up. Prostate International. 13(4). 258–263.
3.
Hsieh, Tsung-Hsun, Jar‐Yi Ho, Chih‐Chien Wang, et al.. (2025). Fucoidan Modulates Osteoarthritis Progression Through miR-22/HO-1 Pathway. Cells. 14(15). 1208–1208.
4.
Yü, Dah‐Shyong, et al.. (2025). Extracellular Vesicle‐Mediated Regulation of H3C14 Contributes to Gemcitabine Resistance in Bladder Cancer. Journal of Extracellular Vesicles. 14(11). e70179–e70179.
7.
Yang, Ching‐Wei, Chia‐Lun Wu, Jar‐Yi Ho, et al.. (2019). The modulation study of multiple drug resistance in bladder cancer by curcumin and resveratrol. Oncology Letters. 18(6). 6869–6876. 33 indexed citations
8.
Wu, Chia‐Lun, Jar‐Yi Ho, Shun‐Hsing Hung, & Dah‐Shyong Yü. (2018). miR‐429 expression in bladder cancer and its correlation with tumor behavior and clinical outcome. The Kaohsiung Journal of Medical Sciences. 34(6). 335–340. 19 indexed citations
9.
Chen, Chih‐Cheng, Chi‐Yuan Chen, Shir‐Hwa Ueng, et al.. (2018). Corylin increases the sensitivity of hepatocellular carcinoma cells to chemotherapy through long noncoding RNA RAD51-AS1-mediated inhibition of DNA repair. Cell Death and Disease. 9(5). 543–543. 46 indexed citations
10.
Ho, Jar‐Yi, Fung-Wei Chang, Jui‐Ming Liu, et al.. (2016). Estrogen Enhances the Cell Viability and Motility of Breast Cancer Cells through the ERα-ΔNp63-Integrin β4 Signaling Pathway. PLoS ONE. 11(2). e0148301–e0148301. 17 indexed citations
11.
Tang, Shou‐Hung, Sheng‐Tang Wu, Yu‐Chi Chen, et al.. (2015). Novel Cancer Therapeutics with Allosteric Modulation of the Mitochondrial C-Raf–DAPK Complex by Raf Inhibitor Combination Therapy. Cancer Research. 75(17). 3568–3582. 18 indexed citations
12.
Hsu, Ren‐Jun, Jyh-Cherng Yu, Fung-Wei Chang, et al.. (2015). The triterpenoids of Hibiscus syriacus induce apoptosis and inhibit cell migration in breast cancer cells. BMC Complementary and Alternative Medicine. 15(1). 65–65. 52 indexed citations
13.
Tang, Shye-Jye, Cheng‐Ping Yu, Jar‐Yi Ho, et al.. (2014). Galectin-1 Upregulates CXCR4 to Promote Tumor Progression and Poor Outcome in Kidney Cancer. Journal of the American Society of Nephrology. 25(7). 1486–1495. 42 indexed citations
14.
Cha, Tai‐Lung, Shou‐Hung Tang, Sheng‐Tang Wu, et al.. (2013). Emodin modulates epigenetic modifications and suppresses bladder carcinoma cell growth. Molecular Carcinogenesis. 54(3). 167–177. 35 indexed citations
15.
Yu, Cheng‐Ping, Jar‐Yi Ho, Yiting Huang, et al.. (2013). Estrogen Inhibits Renal Cell Carcinoma Cell Progression through Estrogen Receptor-β Activation. PLoS ONE. 8(2). e56667–e56667. 96 indexed citations
16.
Liu, Jui‐Ming, Ren‐Jun Hsu, Horng‐Jyh Harn, et al.. (2012). Angiotensin converting enzyme DD genotype is associated with acute coronary syndrome severity and sudden cardiac death in Taiwan: a case-control emergency room study. BMC Cardiovascular Disorders. 12(1). 6–6. 15 indexed citations
17.
Hsu, Ren‐Jun, Jar‐Yi Ho, Tai‐Lung Cha, et al.. (2012). WNT10A Plays an Oncogenic Role in Renal Cell Carcinoma by Activating WNT/β-catenin Pathway. PLoS ONE. 7(10). e47649–e47649. 58 indexed citations
18.
Wang, Tong‐Hong, et al.. (2011). EphrinA5 suppresses colon cancer development by negatively regulating epidermal growth factor receptor stability. FEBS Journal. 279(2). 251–263. 22 indexed citations
19.
Ding, Shian-ling, Jyh‐Cherng Yu, Shou‐Tung Chen, et al.. (2010). Diverse Associations between ESR1 Polymorphism and Breast Cancer Development and Progression. Clinical Cancer Research. 16(13). 3473–3484. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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