Janine Mett

1.1k total citations
20 papers, 898 citations indexed

About

Janine Mett is a scholar working on Physiology, Molecular Biology and Surgery. According to data from OpenAlex, Janine Mett has authored 20 papers receiving a total of 898 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Physiology, 7 papers in Molecular Biology and 6 papers in Surgery. Recurrent topics in Janine Mett's work include Alzheimer's disease research and treatments (16 papers), Cholesterol and Lipid Metabolism (5 papers) and Nuclear Receptors and Signaling (3 papers). Janine Mett is often cited by papers focused on Alzheimer's disease research and treatments (16 papers), Cholesterol and Lipid Metabolism (5 papers) and Nuclear Receptors and Signaling (3 papers). Janine Mett collaborates with scholars based in Germany, Finland and Israel. Janine Mett's co-authors include Marcus O.W. Grimm, Tobias Hartmann, Heike S. Grimm, Viola J. Haupenthal, Christoph P. Stahlmann, Valerie C. Zimmer, Uli Müller, Benjamin Hundsdörfer, Sven Grösgen and Tatjana L. Rothhaar and has published in prestigious journals such as Journal of Neuroscience, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Janine Mett

20 papers receiving 885 citations

Peers

Janine Mett
Janine Mett
Citations per year, relative to Janine Mett Janine Mett (= 1×) peers Viola J. Haupenthal

Countries citing papers authored by Janine Mett

Since Specialization
Citations

This map shows the geographic impact of Janine Mett's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Janine Mett with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Janine Mett more than expected).

Fields of papers citing papers by Janine Mett

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Janine Mett. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Janine Mett. The network helps show where Janine Mett may publish in the future.

Co-authorship network of co-authors of Janine Mett

This figure shows the co-authorship network connecting the top 25 collaborators of Janine Mett. A scholar is included among the top collaborators of Janine Mett based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Janine Mett. Janine Mett is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mett, Janine, et al.. (2024). Metabolic Profiling of SH-SY5Y and Neuro2A Cells in Relation to Fetal Calf Serum (FCS) Concentration in Culture Media. Metabolites. 14(4). 188–188. 1 indexed citations
2.
Zimmer, Valerie C., Anna Andrea Lauer, Viola J. Haupenthal, et al.. (2023). A bidirectional link between sulfatide and Alzheimer’s disease. Cell chemical biology. 31(2). 265–283.e7. 7 indexed citations
3.
Mett, Janine, Anna Andrea Lauer, Daniel Janitschke, et al.. (2021). Medium-Chain Length Fatty Acids Enhance Aβ Degradation by Affecting Insulin-Degrading Enzyme. Cells. 10(11). 2941–2941. 12 indexed citations
4.
Mett, Janine & Uli Müller. (2021). The medium-chain fatty acid decanoic acid reduces oxidative stress levels in neuroblastoma cells. Scientific Reports. 11(1). 6135–6135. 48 indexed citations
6.
Lauer, Anna Andrea, Janine Mett, Daniel Janitschke, et al.. (2020). Regulatory feedback cycle of the insulin‐degrading enzyme and the amyloid precursor protein intracellular domain: Implications for Alzheimer’s disease. Aging Cell. 19(11). e13264–e13264. 8 indexed citations
7.
Grimm, Marcus O.W., Anna Andrea Lauer, Daniel Janitschke, et al.. (2020). The impact of capsaicinoids on APP processing in Alzheimer’s disease in SH-SY5Y cells. Scientific Reports. 10(1). 9164–9164. 8 indexed citations
8.
Grimm, Marcus O.W., Janine Mett, Heike S. Grimm, & Tobias Hartmann. (2017). APP Function and Lipids: A Bidirectional Link. Frontiers in Molecular Neuroscience. 10. 63–63. 87 indexed citations
9.
Grimm, Marcus O.W., Janine Mett, Christoph P. Stahlmann, et al.. (2016). Eicosapentaenoic acid and docosahexaenoic acid increase the degradation of amyloid-β by affecting insulin-degrading enzyme. Biochemistry and Cell Biology. 94(6). 534–542. 51 indexed citations
10.
Grimm, Marcus O.W., Janine Mett, & Tobias Hartmann. (2016). The Impact of Vitamin E and Other Fat-Soluble Vitamins on Alzheimer´s Disease. International Journal of Molecular Sciences. 17(11). 1785–1785. 83 indexed citations
11.
Grimm, Marcus O.W., Janine Mett, Christoph P. Stahlmann, et al.. (2016). Tocotrienol Affects Oxidative Stress, Cholesterol Homeostasis and the Amyloidogenic Pathway in Neuroblastoma Cells: Consequences for Alzheimer’s Disease. International Journal of Molecular Sciences. 17(11). 1809–1809. 39 indexed citations
12.
Grimm, Marcus O.W., Christoph P. Stahlmann, Janine Mett, et al.. (2015). Vitamin E: Curse or benefit in Alzheimer's disease? A systematic investigation of the impact of α-, γ- and δ-tocopherol on Aβ generation and degradation in neuroblastoma cells. The journal of nutrition health & aging. 19(6). 646–654. 27 indexed citations
13.
Grimm, Marcus O.W., Janine Mett, Christoph P. Stahlmann, et al.. (2015). APP intracellular domain derived from amyloidogenic β- and γ-secretase cleavage regulates neprilysin expression. Frontiers in Aging Neuroscience. 7. 77–77. 58 indexed citations
14.
Grimm, Marcus O.W., Viola J. Haupenthal, Janine Mett, et al.. (2015). Oxidized Docosahexaenoic Acid Species and Lipid Peroxidation Products Increase Amyloidogenic Amyloid Precursor Protein Processing. Neurodegenerative Diseases. 16(1-2). 44–54. 49 indexed citations
15.
Grimm, Marcus O.W., Benjamin Hundsdörfer, Sven Grösgen, et al.. (2014). PS Dependent APP Cleavage Regulates Glucosylceramide Synthase and is Affected in Alzheimer's Disease. Cellular Physiology and Biochemistry. 34(1). 92–110. 26 indexed citations
16.
Burg, Verena K., Heike S. Grimm, Tatjana L. Rothhaar, et al.. (2013). Plant Sterols the Better Cholesterol in Alzheimer's Disease? A Mechanistical Study. Journal of Neuroscience. 33(41). 16072–16087. 97 indexed citations
17.
Grimm, Marcus O.W., Johannes Lehmann, Janine Mett, et al.. (2013). Impact of Vitamin D on Amyloid Precursor Protein Processing and Amyloid-β Peptide Degradation in Alzheimer's Disease. Neurodegenerative Diseases. 13(2-3). 75–81. 48 indexed citations
18.
Grimm, Marcus O.W., Janine Mett, Christoph P. Stahlmann, et al.. (2013). Neprilysin and Aβ Clearance: Impact of the APP Intracellular Domain in NEP Regulation and Implications in Alzheimer’s Disease. Frontiers in Aging Neuroscience. 5. 98–98. 119 indexed citations
19.
Grösgen, Sven, Janine Mett, Valerie C. Zimmer, et al.. (2013). Upregulation of PGC‐1α expression by Alzheimer's disease‐associated pathway: presenilin 1/amyloid precursor protein (APP)/intracellular domain of APP. Aging Cell. 13(2). 263–272. 45 indexed citations
20.
Rothhaar, Tatjana L., Sven Grösgen, Viola J. Haupenthal, et al.. (2012). Plasmalogens Inhibit APP Processing by Directly Affectingγ-Secretase Activity in Alzheimer’s Disease. The Scientific World JOURNAL. 2012. 1–15. 61 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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