Jan Mackenzie
Impact in
- Neurology top 5%
- Neurological diseases and metabolism
- Alcoholism and Thiamine Deficiency
-
- Prion Diseases and Protein Misfolding
- RNA regulation and disease
Papers in
-
- Prion Diseases and Protein Misfolding 10
-
- Fibromyalgia and Chronic Fatigue Syndrome Research 2
- Co-authors
- Robert Will (8 shared papers)Jean‐Philippe Brandel (2 shared papers)Richard Knight (6 shared papers)Maurizio Pocchiari (1 shared paper)Lawrence B. Schonberger (1 shared paper)Ellen Leschek (1 shared paper)Anna Ladogana (1 shared paper)Yosikazu Nakamura (1 shared paper)
- Journals
- Annals of Neurology (3 papers)Emerging infectious diseases (2 papers)Transfusion (1 paper)Transplantation (1 paper)Age and Ageing (1 paper)
- Partner nations
- United KingdomFranceTunisia
In The Last Decade
Jan Mackenzie
11 papers receiving 491 citations
Peers
Comparison fields: 5 of 56
- Neurology 229
- Molecular Biology 463
- Nutrition and Dietetics 84
- Neurology 83
- Physiology 68
Countries citing papers authored by Jan Mackenzie
This map shows the geographic impact of Jan Mackenzie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jan Mackenzie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jan Mackenzie more than expected).
Fields of papers citing papers by Jan Mackenzie
This network shows the impact of papers produced by Jan Mackenzie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jan Mackenzie. The network helps show where Jan Mackenzie may publish in the future.
Co-authors
The 25 scholars most cited alongside Jan Mackenzie, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 219 | |
| 2 | 2004 | 112 | |
| 3 | 2010 | 52 | |
| 4 | 2009 | 43 | |
| 5 | 2009 | 33 | |
| 6 | 2010 | 22 | |
| 7 | 2017 | 11 | |
| 8 | 2014 | 8 | |
| 9 | 2024 | 2 | |
| 10 | Is sporadic CJD an acquired disease? A review of the UK CJD cases. | 2016 | 1 |
| 11 | 2021 | 1 |
About Jan Mackenzie
Jan Mackenzie is a scholar working on Molecular Biology, Psychiatry and Mental health, Neurology, Rheumatology and Nutrition and Dietetics, having authored 11 papers that have together received 504 indexed citations. Recurring topics across this work include Prion Diseases and Protein Misfolding (10 papers), Fibromyalgia and Chronic Fatigue Syndrome Research (2 papers), Trace Elements in Health (1 paper), Celiac Disease Research and Management (1 paper), Glycogen Storage Diseases and Myoclonus (1 paper) and Autoimmune Neurological Disorders and Treatments (1 paper). The work is most often cited by research in Neurology (229 citations), Molecular Biology (463 citations), Nutrition and Dietetics (84 citations), Neurology (83 citations) and Physiology (68 citations). Jan Mackenzie has collaborated with scholars based in United Kingdom, France and Tunisia. Frequent co-authors include Robert Will, Jean‐Philippe Brandel, Richard Knight, Maurizio Pocchiari, Lawrence B. Schonberger, Ellen Leschek, Anna Ladogana, Yosikazu Nakamura, Paul Brown and Takeshi Sato. Their work appears in journals such as Annals of Neurology, Emerging infectious diseases, Transfusion, Transplantation and Age and Ageing.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.