James W. Connell

1.9k total citations
18 papers, 1.3k citations indexed

About

James W. Connell is a scholar working on Cellular and Molecular Neuroscience, Cell Biology and Physiology. According to data from OpenAlex, James W. Connell has authored 18 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Cellular and Molecular Neuroscience, 9 papers in Cell Biology and 8 papers in Physiology. Recurrent topics in James W. Connell's work include Hereditary Neurological Disorders (10 papers), Alzheimer's disease research and treatments (6 papers) and Cellular transport and secretion (6 papers). James W. Connell is often cited by papers focused on Hereditary Neurological Disorders (10 papers), Alzheimer's disease research and treatments (6 papers) and Cellular transport and secretion (6 papers). James W. Connell collaborates with scholars based in United Kingdom, United States and Germany. James W. Connell's co-authors include Evan Reid, J. Paul Luzio, Christopher M. Sanderson, Thomas L. Edwards, Stephanie E. Brown, Rachel Allison, Catherine Lindon, Simon Duley, Amanda L. Thompson and Jennifer H. Lumb and has published in prestigious journals such as Journal of Neuroscience, The Journal of Cell Biology and PLoS ONE.

In The Last Decade

James W. Connell

18 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James W. Connell United Kingdom 15 695 586 553 354 203 18 1.3k
Nicholas A. DiProspero United States 8 332 0.5× 856 1.5× 515 0.9× 159 0.4× 113 0.6× 8 1.5k
Claire Haueter United States 12 375 0.5× 713 1.2× 311 0.6× 127 0.4× 36 0.2× 12 1.1k
Jane Vowles United Kingdom 19 220 0.3× 935 1.6× 542 1.0× 387 1.1× 299 1.5× 25 1.7k
Mark Kristiansen United Kingdom 16 200 0.3× 902 1.5× 352 0.6× 159 0.4× 231 1.1× 17 1.2k
Reiko Sato-Yoshitake Japan 12 1.6k 2.3× 1.5k 2.6× 509 0.9× 506 1.4× 136 0.7× 15 2.5k
Elizabeth L. Tudor United Kingdom 7 296 0.4× 752 1.3× 239 0.4× 196 0.6× 129 0.6× 7 1.2k
Ismael Al‐Ramahi United States 22 272 0.4× 1.2k 2.1× 956 1.7× 229 0.6× 93 0.5× 41 1.6k
Elizabeth P. Garcia United States 12 493 0.7× 933 1.6× 521 0.9× 100 0.3× 74 0.4× 12 1.4k
Martine Girard Canada 18 803 1.2× 1.1k 1.9× 361 0.7× 173 0.5× 96 0.5× 27 1.5k
Lakhu Keshvara United States 16 329 0.5× 847 1.4× 549 1.0× 191 0.5× 147 0.7× 20 1.7k

Countries citing papers authored by James W. Connell

Since Specialization
Citations

This map shows the geographic impact of James W. Connell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James W. Connell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James W. Connell more than expected).

Fields of papers citing papers by James W. Connell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James W. Connell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James W. Connell. The network helps show where James W. Connell may publish in the future.

Co-authorship network of co-authors of James W. Connell

This figure shows the co-authorship network connecting the top 25 collaborators of James W. Connell. A scholar is included among the top collaborators of James W. Connell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James W. Connell. James W. Connell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Connell, James W., et al.. (2019). ESCRT-III-associated proteins and spastin inhibit protrudin-dependent polarised membrane traffic. Cellular and Molecular Life Sciences. 77(13). 2641–2658. 21 indexed citations
2.
Allison, Rachel, James R. Edgar, Tania Rizo, et al.. (2017). Defects in ER–endosome contacts impact lysosome function in hereditary spastic paraplegia. The Journal of Cell Biology. 216(5). 1337–1355. 124 indexed citations
3.
Connell, James W., Rachel Allison, & Evan Reid. (2016). Quantitative Gait Analysis Using a Motorized Treadmill System Sensitively Detects Motor Abnormalities in Mice Expressing ATPase Defective Spastin. PLoS ONE. 11(3). e0152413–e0152413. 17 indexed citations
4.
Allison, Rachel, Jennifer H. Lumb, Coralie Fassier, et al.. (2013). An ESCRT–spastin interaction promotes fission of recycling tubules from the endosome. The Journal of Cell Biology. 202(3). 527–543. 120 indexed citations
5.
Lumb, Jennifer H., James W. Connell, Rachel Allison, & Evan Reid. (2011). The AAA ATPase spastin links microtubule severing to membrane modelling. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1823(1). 192–197. 57 indexed citations
6.
Edwards, Thomas L., Xinnan Wang, James W. Connell, et al.. (2009). The hereditary spastic paraplegia proteins NIPA1, spastin and spartin are inhibitors of mammalian BMP signalling. Human Molecular Genetics. 18(20). 3805–3821. 103 indexed citations
7.
Edwards, Thomas L., et al.. (2009). Endogenous spartin (SPG20) is recruited to endosomes and lipid droplets and interacts with the ubiquitin E3 ligases AIP4 and AIP5. Biochemical Journal. 423(1). 31–39. 57 indexed citations
8.
Connell, James W., Catherine Lindon, J. Paul Luzio, & Evan Reid. (2008). Spastin Couples Microtubule Severing to Membrane Traffic in Completion of Cytokinesis and Secretion. Traffic. 10(1). 42–56. 183 indexed citations
9.
10.
Connell, James W., Teresa Rodríguez-Martín, Graham Gibb, et al.. (2005). Quantitative analysis of tau isoform transcripts in sporadic tauopathies. Molecular Brain Research. 137(1-2). 104–109. 56 indexed citations
11.
Sanderson, Christopher M., James W. Connell, Thomas L. Edwards, et al.. (2005). Spastin and atlastin, two proteins mutated in autosomal-dominant hereditary spastic paraplegia, are binding partners. Human Molecular Genetics. 15(2). 307–318. 120 indexed citations
12.
Reid, Evan, James W. Connell, Thomas L. Edwards, et al.. (2004). The hereditary spastic paraplegia protein spastin interacts with the ESCRT-III complex-associated endosomal protein CHMP1B. Human Molecular Genetics. 14(1). 19–38. 154 indexed citations
13.
Utton, Michelle A., James W. Connell, Ayodeji A. Asuni, et al.. (2002). The mechanism of slow axonal transport of the microtubule-associated protein tau and the transport rates of different isoforms and mutants. UCL Discovery (University College London). 1 indexed citations
14.
Anderton, Brian, Michelle A. Utton, Diane P. Hanger, et al.. (2002). The pathological importance of microtubules and tau. Movement Disorders. 17(6). 1402–1402. 1 indexed citations
15.
Lovestone, Simon, Michael Philpot, James W. Connell, et al.. (2002). Genetics, molecular biology, neuropathology and phenotype of frontal lobe dementia. The British Journal of Psychiatry. 180(5). 455–460. 1 indexed citations
16.
Utton, Michelle A., James W. Connell, Ayodeji A. Asuni, et al.. (2002). The Slow Axonal Transport of the Microtubule-Associated Protein Tau and the Transport Rates of Different Isoforms and Mutants in Cultured Neurons. Journal of Neuroscience. 22(15). 6394–6400. 60 indexed citations
17.
18.
Anderton, Brian H., Joanna Betts, Walter Blackstock, et al.. (2001). Sites of phosphorylation in tau and factors affecting their regulation. Biochemical Society Symposia. 67(67). 73–80. 87 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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