James T. B. Crawley

4.5k total citations
58 papers, 3.3k citations indexed

About

James T. B. Crawley is a scholar working on Hematology, Immunology and Genetics. According to data from OpenAlex, James T. B. Crawley has authored 58 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 48 papers in Hematology, 35 papers in Immunology and 18 papers in Genetics. Recurrent topics in James T. B. Crawley's work include Blood Coagulation and Thrombosis Mechanisms (30 papers), Platelet Disorders and Treatments (28 papers) and Complement system in diseases (28 papers). James T. B. Crawley is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (30 papers), Platelet Disorders and Treatments (28 papers) and Complement system in diseases (28 papers). James T. B. Crawley collaborates with scholars based in United Kingdom, United States and Belgium. James T. B. Crawley's co-authors include David A. Lane, Rens de Groot, Brenda M. Luken, Sara Zanardelli, Marie Scully, Florea Lupu, Yaozu Xiang, Jonathan Lam, James S. O’Donnell and Frits R. Rosendaal and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

James T. B. Crawley

58 papers receiving 3.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James T. B. Crawley United Kingdom 34 1.9k 1.5k 751 547 385 58 3.3k
José A. López United States 22 1.3k 0.7× 1.0k 0.7× 490 0.7× 692 1.3× 230 0.6× 63 3.1k
Vahid Afshar‐Kharghan United States 33 1.6k 0.8× 1.3k 0.8× 350 0.5× 884 1.6× 428 1.1× 89 4.1k
Paul Browne Ireland 26 1.3k 0.7× 1.5k 0.9× 956 1.3× 858 1.6× 130 0.3× 68 3.7k
Vincent Praloran France 31 1.6k 0.8× 790 0.5× 1.2k 1.6× 1.0k 1.9× 365 0.9× 127 3.3k
Yasuo Mori Japan 20 679 0.4× 770 0.5× 378 0.5× 1.0k 1.9× 174 0.5× 102 2.9k
Karin M. Hoffmeister United States 35 2.5k 1.3× 670 0.4× 321 0.4× 907 1.7× 118 0.3× 96 4.2k
N L Esmon United States 26 2.7k 1.4× 637 0.4× 582 0.8× 645 1.2× 453 1.2× 38 3.9k
Dominique Meyer France 35 2.8k 1.5× 788 0.5× 507 0.7× 533 1.0× 150 0.4× 106 3.9k
Josef S. Smolen Austria 24 966 0.5× 1.2k 0.8× 421 0.6× 1.7k 3.1× 346 0.9× 37 5.3k
Michel Delforge Belgium 31 2.6k 1.3× 846 0.5× 646 0.9× 2.1k 3.9× 92 0.2× 150 4.4k

Countries citing papers authored by James T. B. Crawley

Since Specialization
Citations

This map shows the geographic impact of James T. B. Crawley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James T. B. Crawley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James T. B. Crawley more than expected).

Fields of papers citing papers by James T. B. Crawley

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James T. B. Crawley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James T. B. Crawley. The network helps show where James T. B. Crawley may publish in the future.

Co-authorship network of co-authors of James T. B. Crawley

This figure shows the co-authorship network connecting the top 25 collaborators of James T. B. Crawley. A scholar is included among the top collaborators of James T. B. Crawley based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James T. B. Crawley. James T. B. Crawley is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sasikumar, Parvathy, et al.. (2024). TFPIα anticoagulant function is highly dependent on protein S in vivo. Science Advances. 10(5). eadk5836–eadk5836. 6 indexed citations
2.
Ahnström, Josefin, et al.. (2024). Tissue factor pathway inhibitor – cofactor-dependent regulation of the initiation of coagulation. Current Opinion in Hematology. 31(6). 315–320. 4 indexed citations
3.
Crawley, James T. B., et al.. (2023). Platelet–Neutrophil Crosstalk in Thrombosis. International Journal of Molecular Sciences. 24(2). 1266–1266. 27 indexed citations
4.
Gierula, Magdalena, David Jones, Salvatore Santamaria, et al.. (2021). Laminin G1 residues of protein S mediate its TFPI cofactor function and are competitively regulated by C4BP. Blood Advances. 6(2). 704–715. 8 indexed citations
5.
Kim, Hyo Jung, et al.. (2021). Crystal structure of ADAMTS13 CUB domains reveals their role in global latency. Science Advances. 7(16). 24 indexed citations
6.
Schelpe, An‐Sofie, Elien Roose, Inge Pareyn, et al.. (2020). Antibodies that conformationally activate ADAMTS13 allosterically enhance metalloprotease domain function. Blood Advances. 4(6). 1072–1080. 30 indexed citations
7.
Grassi, Luigi, et al.. (2020). Activated αIIbβ3 on platelets mediates flow-dependent NETosis via SLC44A2. eLife. 9. 71 indexed citations
8.
Gierula, Magdalena, Isabelle I. Salles‐Crawley, Salvatore Santamaria, et al.. (2019). The roles of factor Va and protein S in formation of the activated protein C/protein S/factor Va inactivation complex. Journal of Thrombosis and Haemostasis. 17(12). 2056–2068. 15 indexed citations
9.
Colige, Alain, et al.. (2019). Proteomic discovery of substrates of the cardiovascular protease ADAMTS7. Journal of Biological Chemistry. 294(20). 8037–8045. 40 indexed citations
10.
Santamaria, Salvatore, Magdalena Gierula, Rodney M. Camire, et al.. (2017). Factor V has an anticoagulant cofactor activity that targets the early phase of coagulation. Journal of Biological Chemistry. 292(22). 9335–9344. 38 indexed citations
11.
Maino, Alberto, Bob Siegerink, Luca A. Lotta, et al.. (2015). Plasma ADAMTS‐13 levels and the risk of myocardial infarction: an individual patient data meta‐analysis. Journal of Thrombosis and Haemostasis. 13(8). 1396–1404. 49 indexed citations
12.
Crawley, James T. B., Rens de Groot, Yaozu Xiang, Brenda M. Luken, & David A. Lane. (2011). Unraveling the scissile bond: how ADAMTS13 recognizes and cleaves von Willebrand factor. Blood. 118(12). 3212–3221. 214 indexed citations
13.
Crawley, James T. B., David A. Lane, Mark Woodward, Ann Rumley, & Gordon Lowe. (2008). Evidence that high von Willebrand factor and low ADAMTS‐13 levels independently increase the risk of a non‐fatal heart attack. Journal of Thrombosis and Haemostasis. 6(4). 583–588. 80 indexed citations
14.
Lam, Jonathan, et al.. (2007). Further characterization of ADAMTS‐13 inactivation by thrombin. Journal of Thrombosis and Haemostasis. 5(5). 1010–1018. 29 indexed citations
15.
Crawley, James T. B., et al.. (2007). The central role of thrombin in hemostasis. Journal of Thrombosis and Haemostasis. 5. 95–101. 272 indexed citations
16.
O’Donnell, James S., Thomas A. J. McKinnon, James T. B. Crawley, David A. Lane, & Michael Laffan. (2005). Bombay phenotype is associated with reduced plasma-VWF levels and an increased susceptibility to ADAMTS13 proteolysis. Blood. 106(6). 1988–1991. 78 indexed citations
17.
Zanardelli, Sara, et al.. (2005). ADAMTS13 Substrate Recognition of von Willebrand Factor A2 Domain. Journal of Biological Chemistry. 281(3). 1555–1563. 69 indexed citations
18.
Crawley, James T. B., et al.. (2004). Proteolytic inactivation of ADAMTS13 by thrombin and plasmin. Blood. 105(3). 1085–1093. 176 indexed citations
19.
Crawley, James T. B. & Jennifer T. Grant Weinandy. (2002). Transference and projection : mirrors to the self. Australasian Journal of Paramedicine. 22 indexed citations
20.
Gu, Jian-Ming, James T. B. Crawley, Gary Ferrell, et al.. (2002). Disruption of the Endothelial Cell Protein C Receptor Gene in Mice Causes Placental Thrombosis and Early Embryonic Lethality. Journal of Biological Chemistry. 277(45). 43335–43343. 102 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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