James Norton

1.9k total citations
36 papers, 1.1k citations indexed

About

James Norton is a scholar working on Otorhinolaryngology, Physiology and Immunology and Allergy. According to data from OpenAlex, James Norton has authored 36 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Otorhinolaryngology, 19 papers in Physiology and 15 papers in Immunology and Allergy. Recurrent topics in James Norton's work include Sinusitis and nasal conditions (20 papers), Asthma and respiratory diseases (18 papers) and Allergic Rhinitis and Sensitization (15 papers). James Norton is often cited by papers focused on Sinusitis and nasal conditions (20 papers), Asthma and respiratory diseases (18 papers) and Allergic Rhinitis and Sensitization (15 papers). James Norton collaborates with scholars based in United States, Japan and South Korea. James Norton's co-authors include Robert P. Schleimer, David B. Conley, Bruce K. Tan, Lydia Suh, Robert C. Kern, Rakesh K. Chandra, Leslie C. Grammer, Atsushi Kato, Anju T. Peters and Roderick Carter and has published in prestigious journals such as The Journal of Immunology, American Journal of Respiratory and Critical Care Medicine and Journal of Allergy and Clinical Immunology.

In The Last Decade

James Norton

35 papers receiving 1.1k citations

Peers

James Norton
Monet Howard United States
Faisal A. Khokhar United States
Kathryn L. Pothoven United States
Siew Shuen Chao Singapore
Andrew Whyte Australia
Keith E. Matheny United States
James Norton
Citations per year, relative to James Norton James Norton (= 1×) peers Nicholas Van Bruaene

Countries citing papers authored by James Norton

Since Specialization
Citations

This map shows the geographic impact of James Norton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James Norton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James Norton more than expected).

Fields of papers citing papers by James Norton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James Norton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James Norton. The network helps show where James Norton may publish in the future.

Co-authorship network of co-authors of James Norton

This figure shows the co-authorship network connecting the top 25 collaborators of James Norton. A scholar is included among the top collaborators of James Norton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James Norton. James Norton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Stevens, Whitney W., Kathryn E. Hulse, Roderick Carter, et al.. (2020). A Novel Role for 15-Lipoxygenase Metabolites in Aspirin Exacerbated Respiratory Disease. Journal of Allergy and Clinical Immunology. 145(2). AB242–AB242. 2 indexed citations
2.
Stevens, Whitney W., Kathryn E. Hulse, Deborah R. Winter, et al.. (2020). Selective Activation of the 15-Lipoxgyenase Pathway in Aspirin Exacerbated Respiratory Disease. A1262–A1262. 3 indexed citations
3.
Stevens, Whitney W., Julie A. Poposki, Atsushi Kato, et al.. (2019). Investigation of Basophils in Aspirin Exacerbated Respiratory Disease Pathogenesis. A2370–A2370. 2 indexed citations
4.
Cho, Seong, Dae Woo Kim, Sun H. Lee, et al.. (2015). Age-Related Increased Prevalence of Asthma and Nasal Polyps in Chronic Rhinosinusitis and Its Association with Altered IL-6 Trans-Signaling. American Journal of Respiratory Cell and Molecular Biology. 53(5). 601–606. 48 indexed citations
5.
Homma, Tetsuya, Bharat Bhushan, Atsushi Kato, et al.. (2014). Aspergillus Fumigatus May Promote Th2 Activation By Suppression Of Interferon Signaling. Journal of Allergy and Clinical Immunology. 133(2). AB136–AB136. 2 indexed citations
6.
Pothoven, Kathryn L., James Norton, Christopher Ocampo, et al.. (2014). Oncostatin M Is Elevated In Chronic Rhinosinusitis and Decreases Barrier Function In Human Airway Epithelium. Journal of Allergy and Clinical Immunology. 133(2). AB237–AB237. 4 indexed citations
7.
Poposki, Julie A., Sarah Peterson, Kevin C. Welch, et al.. (2014). Elevated presence of myeloid dendritic cells in nasal polyps of patients with chronic rhinosinusitis. Clinical & Experimental Allergy. 45(2). 384–393. 28 indexed citations
8.
Kern, Robert C., Kathryn E. Hulse, Rakesh K. Chandra, et al.. (2014). Evidence For Immunoglobulin D In Patients With Chronic Rhinosinusitis. Journal of Allergy and Clinical Immunology. 133(2). AB236–AB236. 4 indexed citations
9.
Kato, Atsushi, Anjeni Keswani, Julie Kim, et al.. (2014). Post-Translational Modification By Serine Proteases Controls The CCL23 Activity In Nasal Polyps Of Chronic Rhinosinusitis. Journal of Allergy and Clinical Immunology. 133(2). AB129–AB129. 1 indexed citations
10.
Takabayashi, Tetsuji, Atsushi Kato, Anju T. Peters, et al.. (2013). Increased expression of factor XIII-A in patients with chronic rhinosinusitis with nasal polyps. Journal of Allergy and Clinical Immunology. 132(3). 584–592.e4. 103 indexed citations
11.
Peterson, Sarah, Julie A. Poposki, Lydia Suh, et al.. (2013). Elevated Presence of Dendritic Cell Subsets in Chronic Rhinosinusitis. Journal of Allergy and Clinical Immunology. 131(2). AB60–AB60. 1 indexed citations
12.
Nagarkar, Deepti R., Julie A. Poposki, Bruce K. Tan, et al.. (2013). Thymic stromal lymphopoietin activity is increased in nasal polyps of patients with chronic rhinosinusitis. Journal of Allergy and Clinical Immunology. 132(3). 593–600.e12. 199 indexed citations
13.
Takabayashi, Tetsuji, Atsushi Kato, Anju T. Peters, et al.. (2012). Excessive Fibrin Deposition in Nasal Polyps Caused by Fibrinolytic Impairment through Reduction of Tissue Plasminogen Activator Expression. American Journal of Respiratory and Critical Care Medicine. 187(1). 49–57. 125 indexed citations
14.
Takabayashi, Tetsuji, Atsushi Kato, Anju T. Peters, et al.. (2012). Glandular mast cells with distinct phenotype are highly elevated in chronic rhinosinusitis with nasal polyps. Journal of Allergy and Clinical Immunology. 130(2). 410–420.e5. 121 indexed citations
15.
Poposki, Julie A., Ashraf Uzzaman, Deepti R. Nagarkar, et al.. (2011). Increased expression of the chemokine CCL23 in eosinophilic chronic rhinosinusitis with nasal polyps. Journal of Allergy and Clinical Immunology. 128(1). 73–81.e4. 82 indexed citations
16.
Peterson, Sarah, Julie A. Poposki, Deepti R. Nagarkar, et al.. (2011). Increased expression of CC chemokine ligand 18 in patients with chronic rhinosinusitis with nasal polyps. Journal of Allergy and Clinical Immunology. 129(1). 119–127.e9. 70 indexed citations
17.
Tan, Bruce K., Quan‐Zhen Li, Lydia Suh, et al.. (2011). Evidence for intranasal antinuclear autoantibodies in patients with chronic rhinosinusitis with nasal polyps. Journal of Allergy and Clinical Immunology. 128(6). 1198–1206.e1. 139 indexed citations
18.
Seshadri, Sudarshan, David C. Lin, James Norton, et al.. (2011). Reduced Expression of Antimicrobial Palate, Lung and Nasal Epithelial Clone (PLUNC) protein in Polyps from Patients with Chronic Rhinosinusitis is Due to Decreased Number of Glands. Journal of Allergy and Clinical Immunology. 127(2). AB141–AB141. 1 indexed citations
19.
Peters, Anju T., Atsushi Kato, Lydia Suh, et al.. (2009). Analysis of the Th17 Pathway in Chronic Rhinosinusitis. Journal of Allergy and Clinical Immunology. 123(2). S145–S145. 2 indexed citations
20.
Friedman, Beth S., Gail Darling, James Norton, Leigh S. Hamby, & Dean D. Metcalfe. (1990). Splenectomy in the management of systemic mast cell disease.. PubMed. 107(1). 94–100. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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