James L. Slack
- Hematology top 0.2%
- Molecular Biology top 5%
- Genetics top 2%
- Public Health, Environmental and Occupational Health top 5%
- Oncology top 10%
- Co-authors
- Hagop M. KantarjianJohn F. DiPersioMaher AlbitarPaul BörnsteinC RosenfeldJean‐Pierre J. IssaHussain I. SabaCarlos de Castro Lozano
- Topics
- Acute Myeloid Leukemia Research (42 papers)Acute Lymphoblastic Leukemia research (19 papers)Retinoids in leukemia and cellular processes (18 papers)
- Cited by
- HematologyGeneticsMolecular Biology
- Partner nations
- United StatesCanadaGermany
In The Last Decade
James L. Slack
82 papers receiving 3.6k citations
Hit Papers
Peers
Comparison fields: 5 of 125
- Hematology 2.1k
- Molecular Biology 1.9k
- Genetics 569
- Public Health, Environmental and Occupational Health 548
- Oncology 405
Countries citing papers authored by James L. Slack
This map shows the geographic impact of James L. Slack's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James L. Slack with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James L. Slack more than expected).
Fields of papers citing papers by James L. Slack
This network shows the impact of papers produced by James L. Slack. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James L. Slack. The network helps show where James L. Slack may publish in the future.
Co-authorship network of co-authors of James L. Slack
This figure shows the co-authorship network connecting the top 25 collaborators of James L. Slack. A scholar is included among the top collaborators of James L. Slack based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James L. Slack. James L. Slack is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 3 | |
| 3 | 28 | |
| 4 | 28 | |
| 5 | 5 | |
| 6 | 7 | |
| 7 | 16 | |
| 8 | 13 | |
| 9 | 91 | |
| 10 | 8 | |
| 11 | 19 | |
| 12 | Decitabine improves patient outcomes in myelodysplastic syndromesbreakdown → | 1139 |
| 13 | 18 | |
| 14 | 57 | |
| 15 | 33 | |
| 16 | 26 | |
| 17 | 16 | |
| 18 | 9 | |
| 19 | 20 | |
| 20 | 15 |
About James L. Slack
James L. Slack is a scholar working on Hematology, Genetics and Transplantation, having authored 84 papers that have together received 3.6k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (42 papers), Acute Lymphoblastic Leukemia research (19 papers) and Retinoids in leukemia and cellular processes (18 papers). The work is most often cited by research in Hematology (2.1k citations), Genetics (569 citations) and Molecular Biology (1.9k citations). James L. Slack has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include Hagop M. Kantarjian, John F. DiPersio, Maher Albitar, Paul Börnstein, C Rosenfeld, Jean‐Pierre J. Issa, Hussain I. Saba, Carlos de Castro Lozano, Azra Raza and Virginia M. Klimek. Their work appears in journals such as Journal of Clinical Oncology, Blood and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.