James J. Cox

8.7k total citations · 1 hit paper
50 papers, 4.6k citations indexed

About

James J. Cox is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, James J. Cox has authored 50 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Molecular Biology, 24 papers in Physiology and 16 papers in Cellular and Molecular Neuroscience. Recurrent topics in James J. Cox's work include Ion channel regulation and function (24 papers), Pain Mechanisms and Treatments (22 papers) and Neuroscience and Neuropharmacology Research (6 papers). James J. Cox is often cited by papers focused on Ion channel regulation and function (24 papers), Pain Mechanisms and Treatments (22 papers) and Neuroscience and Neuropharmacology Research (6 papers). James J. Cox collaborates with scholars based in United Kingdom, United States and Germany. James J. Cox's co-authors include C. Geoffrey Woods, John N. Wood, Adeline K. Nicholas, Gulshan Karbani, Kelly Springell, Emma Roberts, Lihadh Al‐Gazali, Frank Reimann, Fiona M. Gribble and Shaun Gorman and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

James J. Cox

47 papers receiving 4.5k citations

Hit Papers

An SCN9A channelopathy causes congenital inability to exp... 2006 2026 2012 2019 2006 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James J. Cox United Kingdom 27 2.4k 1.8k 1.2k 1.0k 584 50 4.6k
Andrew F. Russo United States 46 2.0k 0.8× 1.4k 0.7× 1.9k 1.6× 535 0.5× 445 0.8× 138 6.4k
Mami Noda Japan 39 2.0k 0.8× 1.2k 0.7× 1.7k 1.4× 314 0.3× 253 0.4× 124 6.9k
Susan A. Slaugenhaupt United States 47 3.3k 1.4× 775 0.4× 1.6k 1.3× 1.3k 1.3× 569 1.0× 92 8.1k
Rajesh Khanna United States 45 3.2k 1.3× 1.8k 1.0× 2.7k 2.3× 310 0.3× 295 0.5× 223 6.1k
José R. Naranjo Spain 44 2.9k 1.2× 1.2k 0.6× 2.6k 2.2× 370 0.4× 177 0.3× 125 5.2k
G. Cristina Brailoiu United States 35 1.1k 0.5× 589 0.3× 813 0.7× 708 0.7× 581 1.0× 82 4.3k
Maurice A. Curtis New Zealand 44 2.7k 1.1× 1.2k 0.6× 2.0k 1.7× 485 0.5× 248 0.4× 143 7.0k
Oliver von Bohlen und Halbach Germany 41 1.8k 0.7× 738 0.4× 2.1k 1.8× 304 0.3× 256 0.4× 115 5.1k
Adam J. Shaywitz United States 18 2.9k 1.2× 791 0.4× 2.1k 1.8× 528 0.5× 109 0.2× 41 5.6k
Heike Biebermann Germany 42 2.9k 1.2× 1.3k 0.7× 813 0.7× 1.4k 1.4× 209 0.4× 141 7.2k

Countries citing papers authored by James J. Cox

Since Specialization
Citations

This map shows the geographic impact of James J. Cox's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James J. Cox with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James J. Cox more than expected).

Fields of papers citing papers by James J. Cox

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James J. Cox. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James J. Cox. The network helps show where James J. Cox may publish in the future.

Co-authorship network of co-authors of James J. Cox

This figure shows the co-authorship network connecting the top 25 collaborators of James J. Cox. A scholar is included among the top collaborators of James J. Cox based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James J. Cox. James J. Cox is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wood, John N., Nieng Yan, Jian Huang, et al.. (2025). Sensory neuron sodium channels as pain targets; from cocaine to Journavx (VX-548, suzetrigine). The Journal of General Physiology. 157(4). 5 indexed citations
2.
3.
Habib, Abdella M., Shengnan Li, Chenjing Zhang, et al.. (2025). MDFIC2 is a PIEZO channel modulator that can alleviate mechanical allodynia associated with neuropathic pain. Proceedings of the National Academy of Sciences. 122(45). e2512426122–e2512426122.
4.
Gossage, Samuel J., Sara Caxaria, Bazbek Davletov, et al.. (2024). Novel therapies for cancer-induced bone pain. PubMed. 16. 100167–100167. 3 indexed citations
5.
Bangash, Muhammad, Cankut Çubuk, Ana Paula Luiz, et al.. (2024). Analgesic targets identified in mouse sensory neuron somata and terminal pain translatomes. Cell Reports. 43(8). 114614–114614. 2 indexed citations
6.
Gossage, Samuel J., Ana Paula Luiz, Shafaq Sikandar, et al.. (2023). Chemogenetic Silencing of NaV1.8-Positive Sensory Neurons Reverses Chronic Neuropathic and Bone Cancer Pain in FLEx PSAM4-GlyR Mice. eNeuro. 10(9). ENEURO.0151–23.2023. 6 indexed citations
7.
Israel, Mathilde R., Lotten Ragnarsson, James J. Cox, et al.. (2022). Low potency inhibition of NaV1.7 by externally applied QX-314 via a depolarizing shift in the voltage-dependence of activation. European Journal of Pharmacology. 925. 175013–175013.
8.
Alles, Sascha R.A., Filipe Nascimento, Rafael Luján, et al.. (2020). Sensory neuron–derived Na V 1.7 contributes to dorsal horn neuron excitability. Science Advances. 6(8). eaax4568–eaax4568. 19 indexed citations
9.
Koenig, Jennifer, Honglei Huang, Martina Pyrski, et al.. (2018). Mapping protein interactions of sodium channel Na V 1.7 using epitope‐tagged gene‐targeted mice. The EMBO Journal. 37(3). 427–445. 53 indexed citations
10.
Emery, Edward C., Abdella M. Habib, James J. Cox, et al.. (2015). NovelSCN9AMutations Underlying Extreme Pain Phenotypes: Unexpected Electrophysiological and Clinical Phenotype Correlations. Journal of Neuroscience. 35(20). 7674–7681. 41 indexed citations
11.
Waxman, Stephen G., Ingemar S. J. Merkies, Monique M. Gerrits, et al.. (2014). Sodium channel genes in pain-related disorders: phenotype–genotype associations and recommendations for clinical use. The Lancet Neurology. 13(11). 1152–1160. 140 indexed citations
12.
Lutes, Lesley D., Keeley J. Pratt, Amy Gross McMillan, et al.. (2014). Camp-Based Immersion Treatment for Obese, Low Socioeconomic Status, Multi-Ethnic Adolescents. Childhood Obesity. 10(2). 122–131. 19 indexed citations
13.
Quick, Kathy, Jing Zhao, Niels Eijkelkamp, et al.. (2012). TRPC3 and TRPC6 are essential for normal mechanotransduction in subsets of sensory neurons and cochlear hair cells. Open Biology. 2(5). 120068–120068. 129 indexed citations
14.
Carvalho, Ofélia P., Jozef Hertecant, Henry Houlden, et al.. (2010). A novel NGF mutation clarifies the molecular mechanism and extends the phenotypic spectrum of the HSAN5 neuropathy. Journal of Medical Genetics. 48(2). 131–135. 66 indexed citations
15.
Jacoby, Monique, James J. Cox, Stéphanie Gayral, et al.. (2009). INPP5E mutations cause primary cilium signaling defects, ciliary instability and ciliopathies in human and mouse. Nature Genetics. 41(9). 1027–1031. 277 indexed citations
16.
Nicholas, Adeline K., Eric A. Swanson, James J. Cox, et al.. (2008). The molecular landscape of ASPM mutations in primary microcephaly. Journal of Medical Genetics. 46(4). 249–253. 68 indexed citations
17.
Cox, James J., Frank Reimann, Adeline K. Nicholas, et al.. (2006). An SCN9A channelopathy causes congenital inability to experience pain. Nature. 444(7121). 894–898. 1094 indexed citations breakdown →
18.
Cox, James J., Andrew P. Jackson, Jacquelyn Bond, & C. Geoffrey Woods. (2006). What primary microcephaly can tell us about brain growth. Trends in Molecular Medicine. 12(8). 358–366. 128 indexed citations
19.
Woods, C. Geoffrey, James J. Cox, Kelly Springell, et al.. (2006). Quantification of Homozygosity in Consanguineous Individuals with Autosomal Recessive Disease. The American Journal of Human Genetics. 78(5). 889–896. 192 indexed citations
20.
Woods, C. Geoffrey, Sigmar Stricker, Petra Seemann, et al.. (2006). Mutations in WNT7A Cause a Range of Limb Malformations, Including Fuhrmann Syndrome and Al-Awadi/Raas-Rothschild/Schinzel Phocomelia Syndrome. The American Journal of Human Genetics. 79(2). 402–408. 116 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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