James G. Greene

3.6k total citations
46 papers, 2.5k citations indexed

About

James G. Greene is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, James G. Greene has authored 46 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Cellular and Molecular Neuroscience, 19 papers in Neurology and 14 papers in Molecular Biology. Recurrent topics in James G. Greene's work include Parkinson's Disease Mechanisms and Treatments (12 papers), Neuroscience and Neuropharmacology Research (10 papers) and Nuclear Receptors and Signaling (7 papers). James G. Greene is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (12 papers), Neuroscience and Neuropharmacology Research (10 papers) and Nuclear Receptors and Signaling (7 papers). James G. Greene collaborates with scholars based in United States, Australia and Germany. James G. Greene's co-authors include J. Timothy Greenamyre, Raymond Dingledine, Ali Reza Noorian, Gary W. Miller, Tonya Taylor, Richard H. Porter, Kristopher J. Bough, Yoland Smith, Bjørnar Hassel and Jeremy W. Gawryluk and has published in prestigious journals such as PLoS ONE, Neurology and The Journal of Physiology.

In The Last Decade

James G. Greene

46 papers receiving 2.4k citations

Peers

James G. Greene

CMN

NEURO

PHYSI

Sarah Berman United States

Amalia C. Bruni Italy

Jignesh D. Pandya United States

V. Bonavita Italy

Jyothisri Kondapalli United States

Alberto Machado Spain

María A. Mena Spain

Victoria Trembovler Israel

Georgia Xiromerisiou Greece

B.L. Scott United States

Sarah Berman United States

James G. Greene

760 ×0.7

CMN

1.2k ×1.3

704 ×0.8

NEURO

495 ×0.8

PHYSI

165 ×0.6

Citations per year, relative to James G. Greene James G. Greene (= 1×) peers Sarah Berman

Countries citing papers authored by James G. Greene

Since Specialization

Citations

This map shows the geographic impact of James G. Greene's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Greene with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Greene more than expected).

Fields of papers citing papers by James G. Greene

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James G. Greene. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Greene. The network helps show where James G. Greene may publish in the future.

Co-authorship network of co-authors of James G. Greene

This figure shows the co-authorship network connecting the top 25 collaborators of James G. Greene. A scholar is included among the top collaborators of James G. Greene based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Greene. James G. Greene is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dattilo, Michael, Walid Bouthour, Jason H. Peragallo, et al.. (2024). The Increasing Burden of Emergency Department and Inpatient Consultations for “Papilledema”. Journal of Neuro-Ophthalmology. 44(4). 571–577. 9 indexed citations

Dattilo, Michael, Walid Bouthour, Jason H. Peragallo, et al.. (2023). Neuro-ophthalmology Emergency Department and Inpatient Consultations at a Large Academic Referral Center. Ophthalmology. 130(12). 1304–1312. 9 indexed citations

Jillella, Dinesh, Fadi Nahab, Karima Benameur, et al.. (2020). Ischemic stroke in COVID-19: An urgent need for early identification and management. PLoS ONE. 15(9). e0239443–e0239443. 28 indexed citations

Greene, James G.. (2014). Causes and Consequences of Degeneration of the Dorsal Motor Nucleus of the Vagus Nerve in Parkinson's Disease. Antioxidants and Redox Signaling. 21(4). 649–667. 52 indexed citations

Greene, James G. & Peter Yellowlees. (2013). Electronic and Remote Prescribing: Administrative, Regulatory, Technical, and Clinical Standards and Guidelines, April 2013. Telemedicine Journal and e-Health. 20(1). 63–74. 14 indexed citations

Adler, Charles H., et al.. (2012). Parkinson’s disease is not associated with gastrointestinal myenteric ganglion neuron loss. Acta Neuropathologica. 124(5). 665–680. 155 indexed citations

Noorian, Ali Reza, et al.. (2012). Alpha-synuclein transgenic mice display age-related slowing of gastrointestinal motility associated with transgene expression in the vagal system. Neurobiology of Disease. 48(1). 9–19. 67 indexed citations

Noorian, Ali Reza, et al.. (2011). Neurochemical phenotypes of myenteric neurons in the rhesus monkey. The Journal of Comparative Neurology. 519(17). 3387–3401. 24 indexed citations

Cloud, Leslie & James G. Greene. (2011). Gastrointestinal Features of Parkinson’s Disease. Current Neurology and Neuroscience Reports. 11(4). 379–384. 49 indexed citations

10.

Greene, James G.. (2011). Animal Models of Gastrointestinal Problems in Parkinson's Disease. Journal of Parkinson s Disease. 1(2). 137–149. 11 indexed citations

11.

Greene, James G.. (2010). Current status and future directions of gene expression profiling in Parkinson's disease. Neurobiology of Disease. 45(1). 76–82. 24 indexed citations

12.

Greene, James G., Karin Borges, & Raymond Dingledine. (2008). Quantitative transcriptional neuroanatomy of the rat hippocampus: Evidence for wide‐ranging, pathway‐specific heterogeneity among three principal cell layers. Hippocampus. 19(3). 253–264. 46 indexed citations

13.

Hamill, Cecily E., W. Michael Caudle, Jason R. Richardson, et al.. (2007). Exacerbation of Dopaminergic Terminal Damage in a Mouse Model of Parkinson’s Disease by the G-Protein-Coupled Receptor Protease-Activated Receptor 1. Molecular Pharmacology. 72(3). 653–664. 45 indexed citations

14.

Greene, James G., Raymond Dingledine, & J. Timothy Greenamyre. (2004). Gene expression profiling of rat midbrain dopamine neurons: implications for selective vulnerability in parkinsonism. Neurobiology of Disease. 18(1). 19–31. 151 indexed citations

15.

Collier, Timothy J., et al.. (2003). Reduced cortical noradrenergic neurotransmission is associated with increased neophobia and impaired spatial memory in aged rats. Neurobiology of Aging. 25(2). 209–221. 42 indexed citations

16.

Limousin, Patricia, et al.. (1997). Positron emission tomography (PET) study of modulation of cerebral activity by subthalamic nucleus (STN) and internal globus pallidus (GPi) stimulation in Parkinson's disease. UCL Discovery (University College London). 3 indexed citations

17.

Greene, James G. & J. Timothy Greenamyre. (1996). Bioenergetics and glutamate excitotoxicity. Progress in Neurobiology. 48(6). 613–634. 203 indexed citations

18.

Greene, James G. & J. Timothy Greenamyre. (1995). Characterization of the Excitotoxic Potential of the Reversible Succinate Dehydrogenase Inhibitor Malonate. Journal of Neurochemistry. 64(1). 430–436. 100 indexed citations

19.

Greenamyre, J. Timothy, et al.. (1994). The endogenous cofactors, thioctic acid and dihydrolipoic acid, are neuroprotective against NMDA and malonic acid lesions of striatum. Neuroscience Letters. 171(1-2). 17–20. 52 indexed citations

20.

Greene, James G., et al.. (1993). Inhibition of Succinate Dehydrogenase by Malonic Acid Produces an “Excitotoxic” Lesion in Rat Striatum. Journal of Neurochemistry. 61(3). 1151–1154. 170 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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