James G. Donnelly
- Rheumatology top 5%
- Molecular Biology
- Surgery
- Pediatrics, Perinatology and Child Health top 10%
- Hematology top 10%
- Co-authors
- Phillip A. IsotaloGeorge A. WellsSteven J. SoldinLeroy ShervingtonAndrew A. HousePaul C. HébertS. P. NadlerJocelyn M. Hicks
- Topics
- Folate and B Vitamins Research (10 papers)Signaling Pathways in Disease (6 papers)Drug Transport and Resistance Mechanisms (4 papers)
- Journals
- The American Journal of Human GeneticsAmerican Journal of Obstetrics and GynecologyClinical Chemistry
- Partner nations
- United StatesCanadaEgypt
In The Last Decade
James G. Donnelly
38 papers receiving 777 citations
Peers
Comparison fields: 5 of 105
- Rheumatology 281
- Molecular Biology 194
- Surgery 165
- Pediatrics, Perinatology and Child Health 122
- Hematology 121
Countries citing papers authored by James G. Donnelly
This map shows the geographic impact of James G. Donnelly's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Donnelly with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Donnelly more than expected).
Fields of papers citing papers by James G. Donnelly
This network shows the impact of papers produced by James G. Donnelly. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Donnelly. The network helps show where James G. Donnelly may publish in the future.
Co-authorship network of co-authors of James G. Donnelly
This figure shows the co-authorship network connecting the top 25 collaborators of James G. Donnelly. A scholar is included among the top collaborators of James G. Donnelly based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Donnelly. James G. Donnelly is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 6 | |
| 3 | 6 | |
| 4 | 19 | |
| 5 | 70 | |
| 6 | 3 | |
| 7 | 39 | |
| 8 | 3 | |
| 9 | 70 | |
| 10 | 7 | |
| 11 | 6 | |
| 12 | 170 | |
| 13 | 11 | |
| 14 | 1 | |
| 15 | 40 | |
| 16 | 9 | |
| 17 | 9 | |
| 18 | 12 | |
| 19 | 12 | |
| 20 | 16 |
About James G. Donnelly
James G. Donnelly is a scholar working on Transplantation, Rheumatology and Internal Medicine, having authored 39 papers that have together received 812 indexed citations. Recurring topics across this work include Folate and B Vitamins Research (10 papers), Signaling Pathways in Disease (6 papers) and Drug Transport and Resistance Mechanisms (4 papers). The work is most often cited by research in Rheumatology (281 citations), Transplantation (31 citations) and Hematology (121 citations). James G. Donnelly has collaborated with scholars based in United States, Canada and Egypt. Frequent co-authors include Phillip A. Isotalo, George A. Wells, Steven J. Soldin, Leroy Shervington, Andrew A. House, Paul C. Hébert, S. P. Nadler, Jocelyn M. Hicks, E W Palaszynski and Jayasimha N. Murthy. Their work appears in journals such as The American Journal of Human Genetics, American Journal of Obstetrics and Gynecology and Clinical Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.