James E. Goldman

22.5k total citations · 5 hit papers
221 papers, 16.5k citations indexed

About

James E. Goldman is a scholar working on Molecular Biology, Developmental Neuroscience and Neurology. According to data from OpenAlex, James E. Goldman has authored 221 papers receiving a total of 16.5k indexed citations (citations by other indexed papers that have themselves been cited), including 94 papers in Molecular Biology, 59 papers in Developmental Neuroscience and 44 papers in Neurology. Recurrent topics in James E. Goldman's work include Neurogenesis and neuroplasticity mechanisms (58 papers), RNA Research and Splicing (42 papers) and Neuroinflammation and Neurodegeneration Mechanisms (33 papers). James E. Goldman is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (58 papers), RNA Research and Splicing (42 papers) and Neuroinflammation and Neurodegeneration Mechanisms (33 papers). James E. Goldman collaborates with scholars based in United States, Japan and United Kingdom. James E. Goldman's co-authors include Fung‐Chow Chiu, Albee Messing, Michael Brenner, Peter Canoll, Steven W. Levison, Beth Levine, A. Kume-Iwaki, Alexander A. Sosunov, Christine Peterson and Satoshi Suzuki and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

James E. Goldman

217 papers receiving 16.2k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Protection against Fatal Sindbis Virus Encephalitis by Beclin, a Novel Bcl-2-Interacting Protein

1998 949 citations James E. Goldman, Beth Levine et al. profile →
Loss of mTOR-Dependent Macroautophagy Causes Autistic-like Synaptic Pruning Deficits

2014 827 citations Guomei Tang, Kathryn Gudsnuk et al. Neuron profile →
Mutations in GFAP, encoding glial fibrillary acidic protein, are associated with Alexander disease

2001 515 citations James E. Goldman et al. profile →
White matter changes in Alzheimer’s disease: a focus on myelin and oligodendrocytes

2018 480 citations James E. Goldman et al. profile →
Non-cell-autonomous disruption of nuclear architecture as a potential cause of COVID-19-induced anosmia

2022 168 citations James E. Goldman et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
James E. Goldman United States	71	7.6k	4.2k	3.6k	3.3k	2.1k	221	16.5k
Kunlin Jin United States	70	7.5k 1.0×	5.6k 1.3×	5.2k 1.5×	4.6k 1.4×	2.7k 1.3×	226	19.1k
Mathias Bähr Germany	76	9.2k 1.2×	2.6k 0.6×	5.9k 1.6×	2.8k 0.9×	1.7k 0.8×	356	17.3k
Karl Herrup United States	66	10.6k 1.4×	2.9k 0.7×	5.1k 1.4×	2.7k 0.8×	3.4k 1.6×	214	19.7k
Giovanni Coppola United States	71	7.9k 1.0×	2.4k 0.6×	6.2k 1.7×	4.1k 1.2×	3.4k 1.6×	274	18.7k
Richard Daneman United States	36	6.3k 0.8×	1.7k 0.4×	2.8k 0.8×	6.6k 2.0×	1.9k 0.9×	58	15.4k
Richard Reynolds United Kingdom	69	5.7k 0.7×	4.5k 1.1×	2.9k 0.8×	4.7k 1.4×	1.2k 0.6×	227	17.2k
Lee L. Rubin United States	67	12.2k 1.6×	1.5k 0.4×	2.9k 0.8×	2.4k 0.7×	1.7k 0.8×	159	18.4k
Nancy Y. Ip Hong Kong	77	11.0k 1.4×	5.2k 1.2×	10.9k 3.1×	1.8k 0.6×	2.8k 1.3×	314	23.8k
Robert Nitsch Germany	61	5.4k 0.7×	2.4k 0.6×	3.8k 1.1×	3.5k 1.1×	1.4k 0.7×	176	13.1k
Mark F. Mehler United States	59	8.4k 1.1×	2.7k 0.6×	2.5k 0.7×	3.8k 1.2×	2.5k 1.2×	143	16.6k

Countries citing papers authored by James E. Goldman

Since Specialization

Citations

This map shows the geographic impact of James E. Goldman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Goldman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Goldman more than expected).

Fields of papers citing papers by James E. Goldman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James E. Goldman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Goldman. The network helps show where James E. Goldman may publish in the future.

Co-authorship network of co-authors of James E. Goldman

This figure shows the co-authorship network connecting the top 25 collaborators of James E. Goldman. A scholar is included among the top collaborators of James E. Goldman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James E. Goldman. James E. Goldman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Xia, Shengnan, Susumu Antoku, David Chen, et al.. (2025). The spatial landscape of glial pathology and T cell response in Parkinson’s disease substantia nigra. Nature Communications. 16(1). 7146–7146. 3 indexed citations

Sosunov, Alexander A., Guy M. McKhann, Guomei Tang, & James E. Goldman. (2024). Cytoplasmic vacuolization and ectopic formation of perineuronal nets are characteristic pathologies of cytomegalic neurons in tuberous sclerosis. Journal of Neuropathology & Experimental Neurology. 83(12). 1047–1059. 1 indexed citations

Ham, Ahrom, Hongyu Li, Jennifer Bain, et al.. (2024). Impaired macroautophagy confers substantial risk for intellectual disability in children with autism spectrum disorders. Molecular Psychiatry. 30(2). 810–824. 2 indexed citations

Al‐Dalahmah, Osama, Alexander A. Sosunov, Yu Sun, et al.. (2024). The Matrix Receptor CD44 Is Present in Astrocytes throughout the Human Central Nervous System and Accumulates in Hypoxia and Seizures. Cells. 13(2). 129–129. 5 indexed citations

Shafit‐Zagardo, Bridget, Simone Sidoli, James E. Goldman, et al.. (2023). TMEM106B Puncta Is Increased in Multiple Sclerosis Plaques, and Reduced Protein in Mice Results in Delayed Lipid Clearance Following CNS Injury. Cells. 12(13). 1734–1734. 2 indexed citations

Heaven, Michael R., Anthony W. Herren, Natasha L. Pacheco, et al.. (2021). Metabolic Enzyme Alterations and Astrocyte Dysfunction in a Murine Model of Alexander Disease With Severe Reactive Gliosis. Molecular & Cellular Proteomics. 21(1). 100180–100180. 8 indexed citations

Shapiro, Steven D., James E. Goldman, Susan Morgello, et al.. (2018). Pathological correlates of brain arterial calcifications. Cardiovascular Pathology. 38. 7–13. 9 indexed citations

Griffiths, Ian R., et al.. (2015). Modeling the natural history of Pelizaeus–Merzbacher disease. Neurobiology of Disease. 75. 115–130. 13 indexed citations

Gutierrez, José, Marco González, James E. Goldman, et al.. (2015). Brain large artery inflammation associated with HIV and large artery remodeling. AIDS. 30(3). 1–1. 33 indexed citations

10.

Tang, Guping, Kathryn Gudsnuk, Sheng‐Han Kuo, et al.. (2014). Loss of mTOR-Dependent Macroautophagy Causes Autistic-like Synaptic Pruning Deficits. Neuron. 83(6). 1482–1482. 30 indexed citations

11.

Tang, Guomei, Kathryn Gudsnuk, Sheng‐Han Kuo, et al.. (2014). Loss of mTOR-Dependent Macroautophagy Causes Autistic-like Synaptic Pruning Deficits. Neuron. 83(5). 1131–1143. 827 indexed citations breakdown →

12.

Goldman, James E., et al.. (2011). Malware analysis & reverse engineering quick evaluation system. 17. 1 indexed citations

13.

Hedley‐Whyte, E. Tessa, James E. Goldman, Maiken Nedergaard, et al.. (2009). Hyaline Protoplasmic Astrocytopathy of Neocortex. Journal of Neuropathology & Experimental Neurology. 68(2). 136–147. 22 indexed citations

14.

Goldman, James E., et al.. (2009). Integration of COBIT, balanced scorecard & SSE-CMM as a strategic information security management (ISM) framework. 19. 3 indexed citations

15.

Novitch, Bennett G., et al.. (2005). Olig2 Directs Astrocyte and Oligodendrocyte Formation in Postnatal Subventricular Zone Cells. Journal of Neuroscience. 25(32). 7289–7298. 207 indexed citations

16.

Mason, Jeffrey L., Arrel D. Toews, Janell Hostettler, et al.. (2004). Oligodendrocytes and Progenitors Become Progressively Depleted within Chronically Demyelinated Lesions. American Journal Of Pathology. 164(5). 1673–1682. 174 indexed citations

17.

Berkel, Victor van, Beth Levine, Sharookh B. Kapadia, et al.. (2002). Critical role for a high-affinity chemokine-binding protein in γ-herpesvirus–induced lethal meningitis. Journal of Clinical Investigation. 109(7). 905–914. 73 indexed citations

18.

Goldman, James E., et al.. (2002). SubpallialDlx2-Expressing Cells Give Rise to Astrocytes and Oligodendrocytes in the Cerebral Cortex and White Matter. Journal of Neuroscience. 22(22). 9821–9830. 101 indexed citations

19.

Goldman, James E., et al.. (1997). Endogenous Progenitors Remyelinate Demyelinated Axons in the Adult CNS. Neuron. 19(1). 197–203. 457 indexed citations

20.

Goldman, James E.. (1994). Design and Implementation of a Practical Business Oriented Undergraduate Data Communications Curriculum. Journal of the Association for Information Systems. 6(2). 80–85. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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