Jakob Gierten

1.0k total citations
23 papers, 655 citations indexed

About

Jakob Gierten is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jakob Gierten has authored 23 papers receiving a total of 655 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 6 papers in Cardiology and Cardiovascular Medicine and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Jakob Gierten's work include Ion channel regulation and function (11 papers), Cardiac electrophysiology and arrhythmias (6 papers) and Genetic Associations and Epidemiology (3 papers). Jakob Gierten is often cited by papers focused on Ion channel regulation and function (11 papers), Cardiac electrophysiology and arrhythmias (6 papers) and Genetic Associations and Epidemiology (3 papers). Jakob Gierten collaborates with scholars based in Germany, United States and United Kingdom. Jakob Gierten's co-authors include Joachim Wittbrodt, Hugo A. Katus, Dierk Thomas, Nils Wagner, Nils Norlin, Robert Prevedel, Lars Hufnagel, Patrick A. Schweizer, Edgar Zitron and Eberhard P. Scholz and has published in prestigious journals such as Nature, Nature Communications and PLoS ONE.

In The Last Decade

Jakob Gierten

21 papers receiving 642 citations

Peers

Jakob Gierten

CCM

BIOPH

CMN

Lennart Hilbert Germany

Diwakar Turaga United States

Yingli Fan United States

Xianhao Chen Canada

Michaela Mickoleit Germany

Eric Lin Canada

Nicholas A. Frost United States

Marie Caroline Müllenbroich Italy

Vladimir P. Nikolski United States

Katsuhiko Sakurada Japan

Lennart Hilbert Germany

Jakob Gierten

410 ×1.3

44 ×0.2

CCM

73 ×0.5

BIOPH

21 ×0.2

CMN

90 ×1.1

Citations per year, relative to Jakob Gierten Jakob Gierten (= 1×) peers Lennart Hilbert

Countries citing papers authored by Jakob Gierten

Since Specialization

Citations

This map shows the geographic impact of Jakob Gierten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jakob Gierten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jakob Gierten more than expected).

Fields of papers citing papers by Jakob Gierten

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jakob Gierten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jakob Gierten. The network helps show where Jakob Gierten may publish in the future.

Co-authorship network of co-authors of Jakob Gierten

This figure shows the co-authorship network connecting the top 25 collaborators of Jakob Gierten. A scholar is included among the top collaborators of Jakob Gierten based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jakob Gierten. Jakob Gierten is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Thumberger, Thomas, et al.. (2026). Discovery and characterization of gene-by-environment and epistatic genetic effects in a vertebrate model. Cell Genomics. 101164–101164.

Gierten, Jakob, Tomas Fitzgerald, Thomas Thumberger, et al.. (2025). Natural genetic variation quantitatively regulates heart rate and dimension. Nature Communications. 16(1). 4062–4062. 1 indexed citations

Doering, L.C., Thomas Thumberger, Beate Wittbrodt, et al.. (2023). CRISPR-based knockout and base editing confirm the role of MYRF in heart development and congenital heart disease. Disease Models & Mechanisms. 16(8). 3 indexed citations

Gierten, Jakob, et al.. (2022). Precise in vivo functional analysis of DNA variants with base editing using ACEofBASEs target prediction. eLife. 11. 20 indexed citations

Gierten, Jakob, et al.. (2021). In vivo identification and validation of novel potential predictors for human cardiovascular diseases. PLoS ONE. 16(12). e0261572–e0261572. 6 indexed citations

Wagner, Nils, Fynn Beuttenmueller, Nils Norlin, et al.. (2021). Deep learning-enhanced light-field imaging with continuous validation. Nature Methods. 18(5). 557–563. 91 indexed citations

Meyer, Hannah V., Timothy J. W. Dawes, Marta Serrani, et al.. (2020). Genetic and functional insights into the fractal structure of the heart. Nature. 584(7822). 589–594. 78 indexed citations

Gierten, Jakob, Christian Pylatiuk, Johannes Stegmaier, et al.. (2020). Automated high-throughput heartbeat quantification in medaka and zebrafish embryos under physiological conditions. Scientific Reports. 10(1). 2046–2046. 65 indexed citations

Just, Steffen, et al.. (2019). Machine Learning Methods for Automated Quantification of Ventricular Dimensions. Zebrafish. 16(6). 542–545. 10 indexed citations

10.

Wagner, Nils, Nils Norlin, Jakob Gierten, et al.. (2019). Instantaneous isotropic volumetric imaging of fast biological processes. Nature Methods. 16(6). 497–500. 83 indexed citations

11.

Staudacher, Ingo, et al.. (2018). Cloning and characterization of zebrafish K2P13.1 (THIK-1) two-pore-domain K+ channels. Journal of Molecular and Cellular Cardiology. 126. 96–104. 4 indexed citations

12.

Staudacher, Ingo, et al.. (2018). Identification and functional characterization of zebrafish K 2P 17.1 (TASK-4, TALK-2) two-pore-domain K + channels. European Journal of Pharmacology. 831. 94–102. 3 indexed citations

13.

Rahm, Ann‐Kathrin, Felix Wiedmann, Jakob Gierten, et al.. (2013). Functional characterization of zebrafish K2P18.1 (TRESK) two-pore-domain K+ channels. Naunyn-Schmiedeberg s Archives of Pharmacology. 387(3). 291–300. 23 indexed citations

14.

Staudacher, Ingo, Eckhard Ficker, Claudia Seyler, et al.. (2011). Carvedilol targets human K_2P3.1 (TASK1) K⁺leak channels. British Journal of Pharmacology. 163(5). 1099–1110. 38 indexed citations

15.

Rahm, Ann‐Kathrin, Jakob Gierten, Ingo Staudacher, et al.. (2011). PKC‐dependent activation of human K_2P18.1 K⁺ channels. British Journal of Pharmacology. 166(2). 764–773. 37 indexed citations

16.

Gierten, Jakob, et al.. (2011). Identification and functional characterization of zebrafish K2P10.1 (TREK2) two-pore-domain K+ channels. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1818(1). 33–41. 27 indexed citations

17.

Seyler, Claudia, Edgar Zitron, Jakob Gierten, et al.. (2011). TASK1 (K_2P3.1) K⁺channel inhibition by endothelin‐1 is mediated through Rho kinase‐dependent phosphorylation. British Journal of Pharmacology. 165(5). 1467–1475. 42 indexed citations

18.

Gierten, Jakob, Eckhard Ficker, Ramona Bloehs, et al.. (2009). Inhibition of hK2P3.1 (TASK-1) Potassium Channels by the Tyrosine Kinase Inhibitor Genistein. Biophysical Journal. 96(3). 563a–563a. 1 indexed citations

19.

Gierten, Jakob, Eckhard Ficker, Ramona Bloehs, et al.. (2008). Regulation of two‐pore‐domain (K_2P) potassium leak channels by the tyrosine kinase inhibitor genistein. British Journal of Pharmacology. 154(8). 1680–1690. 56 indexed citations

20.

Thomas, Dierk, Ramona Bloehs, Ronald Koschny, et al.. (2007). Doxazosin induces apoptosis of cells expressing hERG K+ channels. European Journal of Pharmacology. 579(1-3). 98–103. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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