Jake T. Neumann

751 total citations
27 papers, 601 citations indexed

About

Jake T. Neumann is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Developmental Neuroscience. According to data from OpenAlex, Jake T. Neumann has authored 27 papers receiving a total of 601 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 8 papers in Developmental Neuroscience. Recurrent topics in Jake T. Neumann's work include Anesthesia and Neurotoxicity Research (8 papers), Ion channel regulation and function (7 papers) and Neuroscience and Neuropharmacology Research (6 papers). Jake T. Neumann is often cited by papers focused on Anesthesia and Neurotoxicity Research (8 papers), Ion channel regulation and function (7 papers) and Neuroscience and Neuropharmacology Research (6 papers). Jake T. Neumann collaborates with scholars based in United States, Italy and Taiwan. Jake T. Neumann's co-authors include Miguel A. Pérez‐Pinzón, Charles H. Cohan, Clinton B. Wright, Hung Wen Lin, Julio A. Copello, Kunjan R. Dave, Kevin B. Koronowski, Paula Díaz‐Sylvester, Sidney Fleischer and Alexandre Couto e Silva and has published in prestigious journals such as Journal of the American College of Cardiology, PLoS ONE and Stroke.

In The Last Decade

Jake T. Neumann

26 papers receiving 590 citations

Peers

Jake T. Neumann

NEURO

CMN

PHYSI

Charles H. Cohan United States

Hung Wen Lin United States

Jeong Hwi Cho South Korea

Sandeep K. Agrawal United States

Myoung Cheol Shin South Korea

Chun-Feng Liu China

Kusum Sinha India

Jingye Wang China

Reggie Hui‐Chao Lee United States

Charles H. Cohan United States

Jake T. Neumann

103 ×0.5

160 ×1.0

NEURO

88 ×0.7

CMN

98 ×1.0

PHYSI

44 ×0.7

Citations per year, relative to Jake T. Neumann Jake T. Neumann (= 1×) peers Charles H. Cohan

Countries citing papers authored by Jake T. Neumann

Since Specialization

Citations

This map shows the geographic impact of Jake T. Neumann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jake T. Neumann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jake T. Neumann more than expected).

Fields of papers citing papers by Jake T. Neumann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jake T. Neumann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jake T. Neumann. The network helps show where Jake T. Neumann may publish in the future.

Co-authorship network of co-authors of Jake T. Neumann

This figure shows the co-authorship network connecting the top 25 collaborators of Jake T. Neumann. A scholar is included among the top collaborators of Jake T. Neumann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jake T. Neumann. Jake T. Neumann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Neumann, Jake T., et al.. (2024). Geographical Impact of Human Gift Registries in West Virginia: A Model for Centralized Resources in Human Anatomy Education. 95(1). 1 indexed citations

Silva, Alexandre Couto e, Vesna Tešić, Krista M. Rodgers, et al.. (2022). Protein arginine methyltransferase 4 modulates nitric oxide synthase uncoupling and cerebral blood flow in Alzheimer's disease. Journal of Cellular Physiology. 239(6). e30858–e30858. 13 indexed citations

Lee, Reggie Hui‐Chao, et al.. (2021). Activation of Neuropeptide Y2 Receptor Can Inhibit Global Cerebral Ischemia-Induced Brain Injury. NeuroMolecular Medicine. 24(2). 97–112. 6 indexed citations

Wu, Celeste Yin‐Chieh, Miguel A. López‐Toledano, Ahmed Daak, et al.. (2020). SC411 treatment can enhance survival in a mouse model of sickle cell disease. Prostaglandins Leukotrienes and Essential Fatty Acids. 158. 102110–102110. 3 indexed citations

Wu, Celeste Yin‐Chieh, Alexandre Couto e Silva, Chin‐Hung Liu, et al.. (2020). Stearic acid methyl ester affords neuroprotection and improves functional outcomes after cardiac arrest. Prostaglandins Leukotrienes and Essential Fatty Acids. 159. 102138–102138. 12 indexed citations

Lee, Reggie Hui‐Chao, et al.. (2018). Palmitic acid methyl ester is a novel neuroprotective agent against cardiac arrest. Prostaglandins Leukotrienes and Essential Fatty Acids. 147. 6–14. 30 indexed citations

Wu, Celeste Yin‐Chieh, Alexandre Couto e Silva, Tsung‐Han Hsieh, et al.. (2018). Utilizing the Modified T-Maze to Assess Functional Memory Outcomes After Cardiac Arrest. Journal of Visualized Experiments. 23 indexed citations

Cohan, Charles H., Jake T. Neumann, Kunjan R. Dave, et al.. (2015). Effect of Cardiac Arrest on Cognitive Impairment and Hippocampal Plasticity in Middle-Aged Rats. PLoS ONE. 10(5). e0124918–e0124918. 54 indexed citations

Lee, Reggie Hui‐Chao, et al.. (2015). Fatty acid methyl esters as a potential therapy against cerebral ischemia. OCL. 23(1). D108–D108. 9 indexed citations

10.

Díaz‐Sylvester, Paula, Jake T. Neumann, Sidney Fleischer, & Julio A. Copello. (2014). Coupled Gating of Ryanodine Receptors: Evidence for a Role of Physical RyR-RyR Interactions. Biophysical Journal. 106(2). 109a–110a. 1 indexed citations

11.

Lin, Hung Wen, et al.. (2014). Protein Kinase C Delta Modulates Endothelial Nitric Oxide Synthase after Cardiac Arrest. Journal of Cerebral Blood Flow & Metabolism. 34(4). 613–620. 12 indexed citations

12.

Neumann, Jake T., John W. Thompson, Ami P. Raval, et al.. (2014). Increased BDNF Protein Expression after Ischemic Or PKC Epsilon Preconditioning Promotes Electrophysiologic Changes That Lead to Neuroprotection. Journal of Cerebral Blood Flow & Metabolism. 35(1). 121–130. 50 indexed citations

13.

Neumann, Jake T., et al.. (2013). Global Cerebral Ischemia: Synaptic and Cognitive Dysfunction. Current Drug Targets. 14(1). 20–35. 102 indexed citations

14.

Lin, Hung Wen, et al.. (2013). Fatty Acid Methyl Esters and Solutol HS 15 Confer Neuroprotection after Focal and Global Cerebral Ischemia. Translational Stroke Research. 5(1). 109–117. 31 indexed citations

15.

Neumann, Jake T., Charles H. Cohan, Kunjan R. Dave, Clinton B. Wright, & Miguel A. Pérez‐Pinzón. (2012). Global Cerebral Ischemia: Synaptic and Cognitive Dysfunction. Current Drug Targets. 14(1). 20–35. 6 indexed citations

16.

Neumann, Jake T., Paula Díaz‐Sylvester, Sidney Fleischer, & Julio A. Copello. (2011). CGP37157 is an Inhibitor of the Sarcoplasmic Reticulum Calcium ATPase and an Activator of Ryanodine Receptors in Striated Muscle. Biophysical Journal. 100(3). 416a–416a. 1 indexed citations

17.

Neumann, Jake T. & Julio A. Copello. (2011). Cross-Reactivity of Ryanodine Receptors with Plasma Membrane Ion Channel Modulators. Molecular Pharmacology. 80(3). 509–517. 14 indexed citations

18.

Neumann, Jake T. & Julio A. Copello. (2010). Modulation of Synchronous Gating in Skeletal Muscle Ryanodine Receptor Channels (RyR1) by Nucleotides or Phosphorylation. Biophysical Journal. 98(3). 301a–301a. 1 indexed citations

19.

Neumann, Jake T., Paula Díaz‐Sylvester, Sidney Fleischer, & Julio A. Copello. (2010). CGP-37157 Inhibits the Sarcoplasmic Reticulum Ca2+ ATPase and Activates Ryanodine Receptor Channels in Striated Muscle. Molecular Pharmacology. 79(1). 141–147. 41 indexed citations

20.

Neumann, Jake T. & Julio A. Copello. (2009). Cross-Reactivity of Ryanodine Receptors with Putative Modulators of Ion Channels in the Plasma Membrane. Biophysical Journal. 96(3). 107a–107a. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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