Jae-Ung Lee

606 total citations
13 papers, 345 citations indexed

About

Jae-Ung Lee is a scholar working on Immunology, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Jae-Ung Lee has authored 13 papers receiving a total of 345 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Molecular Biology and 2 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Jae-Ung Lee's work include Immune Cell Function and Interaction (5 papers), Immunotherapy and Immune Responses (3 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). Jae-Ung Lee is often cited by papers focused on Immune Cell Function and Interaction (5 papers), Immunotherapy and Immune Responses (3 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). Jae-Ung Lee collaborates with scholars based in South Korea, United States and Mexico. Jae-Ung Lee's co-authors include Je‐Min Choi, Do-Hyun Kim, Hong‐Gyun Lee, Sangho Lim, Insoo Kang, Alfred L.M. Bothwell, Hyeon‐Soo Park, Myung Chan Gye, CheMyong Ko and Gabsik Yang and has published in prestigious journals such as Nature Communications, The Journal of Immunology and Scientific Reports.

In The Last Decade

Jae-Ung Lee

12 papers receiving 344 citations

Peers

Jae-Ung Lee
Chengzhou Mao China
Yijie Wang China
Denise Sickert Switzerland
Hendrika W. Grievink Netherlands
Zsuzsanna Kolostyák Hungary
Cynthia Laflamme Canada
Ruth Seelige United States
Lowella V. Fortuno United States
Ankita Saini United States
Stefano Maio United Kingdom
Chengzhou Mao China
Jae-Ung Lee
Citations per year, relative to Jae-Ung Lee Jae-Ung Lee (= 1×) peers Chengzhou Mao

Countries citing papers authored by Jae-Ung Lee

Since Specialization
Citations

This map shows the geographic impact of Jae-Ung Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jae-Ung Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jae-Ung Lee more than expected).

Fields of papers citing papers by Jae-Ung Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jae-Ung Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jae-Ung Lee. The network helps show where Jae-Ung Lee may publish in the future.

Co-authorship network of co-authors of Jae-Ung Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Jae-Ung Lee. A scholar is included among the top collaborators of Jae-Ung Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jae-Ung Lee. Jae-Ung Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
2.
So, Y., et al.. (2024). The Potentiality of Natural Products and Herbal Medicine as Novel Medications for Parkinson’s Disease: A Promising Therapeutic Approach. International Journal of Molecular Sciences. 25(2). 1071–1071. 18 indexed citations
3.
Lee, Sung‐Hoon, et al.. (2023). In vivo safety and biodistribution profile of Klotho-enhanced human urine-derived stem cells for clinical application. Stem Cell Research & Therapy. 14(1). 355–355. 3 indexed citations
4.
Lee, Sung Hoon, Tae‐Wook Kang, Jae-Ung Lee, et al.. (2022). Urine-Derived Stem Cell-Secreted Klotho Plays a Crucial Role in the HK-2 Fibrosis Model by Inhibiting the TGF-β Signaling Pathway. International Journal of Molecular Sciences. 23(9). 5012–5012. 11 indexed citations
5.
Kim, Do-Hyun, et al.. (2019). Estrogen receptor α in T cells suppresses follicular helper T cell responses and prevents autoimmunity. Experimental & Molecular Medicine. 51(4). 1–9. 32 indexed citations
6.
Lee, Hong‐Gyun, Jae-Ung Lee, Do-Hyun Kim, et al.. (2019). Pathogenic function of bystander-activated memory-like CD4+ T cells in autoimmune encephalomyelitis. Nature Communications. 10(1). 709–709. 59 indexed citations
7.
Lim, Sangho, et al.. (2019). The cytoplasmic domain of CTLA-4 control autoimmunity via inducing regulatory T cells. The Journal of Immunology. 202(1_Supplement). 193.4–193.4. 1 indexed citations
8.
Lee, Jae-Ung, et al.. (2018). Revisiting the Concept of Targeting NFAT to Control T Cell Immunity and Autoimmune Diseases. Frontiers in Immunology. 9. 2747–2747. 133 indexed citations
9.
10.
Park, Hyeon‐Soo, et al.. (2016). Gender-specific differences in PPARγ regulation of follicular helper T cell responses with estrogen. Scientific Reports. 6(1). 28495–28495. 36 indexed citations
11.
Lee, Jae-Ung, et al.. (2016). Sex-Based Selectivity of PPARγ Regulation in Th1, Th2, and Th17 Differentiation. International Journal of Molecular Sciences. 17(8). 1347–1347. 35 indexed citations
12.
Lee, JE, Seok‐Hwan Kim, Jae-Ung Lee, et al.. (2007). Association with Left Ventricular Diastolic Function Parameters and Right Brachial-ankle Pulse Wave Velocity in Hemodialysis Patients. Kidney Research and Clinical Practice. 26(2). 204–211. 2 indexed citations
13.
Lee, Jae-Ung, et al.. (1998). Nonlinear Cardiac Dynamics and Morning Dip: An Unsound Circadian Rhythm. Sunhwan'gi. 28(3). 382–382.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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