Chengzhou Mao

605 total citations
16 papers, 448 citations indexed

About

Chengzhou Mao is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Chengzhou Mao has authored 16 papers receiving a total of 448 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Immunology and 5 papers in Oncology. Recurrent topics in Chengzhou Mao's work include interferon and immune responses (3 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers) and Chemokine receptors and signaling (2 papers). Chengzhou Mao is often cited by papers focused on interferon and immune responses (3 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers) and Chemokine receptors and signaling (2 papers). Chengzhou Mao collaborates with scholars based in China, United States and South Korea. Chengzhou Mao's co-authors include Somnath Datta, Sumati Rajagopalan, Ami Desai‐Mehta, Peter Baumeister, Louis Dubeau, Song Luo, B Luo, David R. Hinton, Amy S. Lee and E. Barrón and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Immunology and The FASEB Journal.

In The Last Decade

Chengzhou Mao

14 papers receiving 445 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Chengzhou Mao China 8 174 143 102 85 48 16 448
Jinhong Wang China 11 147 0.8× 183 1.3× 35 0.3× 29 0.3× 25 0.5× 25 444
Andreas Ambach Germany 15 197 1.1× 155 1.1× 148 1.5× 118 1.4× 30 0.6× 23 633
Keli Ma China 11 94 0.5× 279 2.0× 45 0.4× 28 0.3× 16 0.3× 23 395
Byungki Jang South Korea 14 145 0.8× 299 2.1× 38 0.4× 65 0.8× 51 1.1× 24 534
Katherine Sully United Kingdom 9 104 0.6× 267 1.9× 95 0.9× 36 0.4× 98 2.0× 10 542
T. Ochiai Japan 10 85 0.5× 114 0.8× 24 0.2× 44 0.5× 47 1.0× 16 364
Vinayaga S. Gnanapragassam Germany 14 160 0.9× 331 2.3× 38 0.4× 16 0.2× 42 0.9× 20 481
Karine Landry Canada 9 182 1.0× 385 2.7× 165 1.6× 20 0.2× 61 1.3× 9 575
N Takami Japan 10 77 0.4× 312 2.2× 114 1.1× 22 0.3× 56 1.2× 13 535
Kevin Brewer United States 5 119 0.7× 298 2.1× 149 1.5× 60 0.7× 52 1.1× 5 537

Countries citing papers authored by Chengzhou Mao

Since Specialization
Citations

This map shows the geographic impact of Chengzhou Mao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chengzhou Mao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chengzhou Mao more than expected).

Fields of papers citing papers by Chengzhou Mao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chengzhou Mao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chengzhou Mao. The network helps show where Chengzhou Mao may publish in the future.

Co-authorship network of co-authors of Chengzhou Mao

This figure shows the co-authorship network connecting the top 25 collaborators of Chengzhou Mao. A scholar is included among the top collaborators of Chengzhou Mao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chengzhou Mao. Chengzhou Mao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Chen, Xinyue, Jin Zhong, Chih‐Hung Lin, et al.. (2025). Glucocorticoid reduces mortality in LPS-induced sepsis mouse model by inhibiting JAK1/STAT3-mediated inflammatory response and restoring tricarboxylic acid cycle. Life Sciences. 375. 123744–123744. 2 indexed citations
2.
Mao, Chengzhou, Weiwen Fan, Jiaqi Liu, et al.. (2025). Targeting HDAC and PARP Enhances STING‐Dependent Antitumor Immunity in STING‐Deficient Tumor. Advanced Science. 12(41). e07904–e07904.
3.
Liu, Bing, Yiwen Yan, Chengzhou Mao, et al.. (2025). Disruption of cell-intrinsic PCSK9 enhances the antitumor efficacy of CD8+ T cells. Journal for ImmunoTherapy of Cancer. 13(7). e011657–e011657.
4.
Huang, Wenjun, Ya Tan, Lulu Li, et al.. (2025). DNMT inhibition epigenetically restores the cGAS-STING pathway and activates RIG-I/MDA5-MAVS to enhance antitumor immunity. Acta Pharmacologica Sinica. 47(1). 197–208. 4 indexed citations
5.
Fan, Weiwen, Wenkai Li, Chengzhou Mao, et al.. (2024). Bifunctional HDAC and DNMT inhibitor induces viral mimicry activates the innate immune response in triple-negative breast cancer. European Journal of Pharmaceutical Sciences. 197. 106767–106767. 13 indexed citations
6.
Zheng, Li, Chengzhou Mao, Zhou Zhang, et al.. (2019). Differential expression of foxo genes during embryonic development and in adult tissues of Xenopus tropicalis. Gene Expression Patterns. 35. 119091–119091. 7 indexed citations
7.
Mao, Chengzhou, Li Zheng, Yimin Zhou, et al.. (2018). CRISPR/Cas9‐mediated efficient and precise targeted integration of donor DNA harboring double cleavage sites in Xenopus tropicalis. The FASEB Journal. 32(12). 6495–6509. 5 indexed citations
8.
Xia, Jingbo, Chengzhou Mao, Guang‐Hui Liu, et al.. (2016). The CXCL10/CXCR3 axis promotes cardiac microvascular endothelial cell migration via the p38/FAK pathway in a proliferation-independent manner. Experimental and Molecular Pathology. 100(2). 257–265. 13 indexed citations
9.
Xia, Jingbo, Chengzhou Mao, Hai-Yan Wu, et al.. (2016). Hypoxia/ischemia promotes CXCL10 expression in cardiac microvascular endothelial cells by NFkB activation. Cytokine. 81. 63–70. 37 indexed citations
10.
Liu, Guang‐Hui, Chengzhou Mao, Hai-Yan Wu, et al.. (2016). Expression profile of rrbp1 genes during embryonic development and in adult tissues of Xenopus laevis. Gene Expression Patterns. 23-24. 1–6. 7 indexed citations
11.
Wang, Miao, Rui Ye, E. Barrón, et al.. (2009). Essential role of the unfolded protein response regulator GRP78/BiP in protection from neuronal apoptosis. Cell Death and Differentiation. 17(3). 488–498. 138 indexed citations
12.
Uckun, Fatih M., Chengzhou Mao, He Huang, et al.. (2001). Spongistatins as Tubulin Targeting Agents. Current Pharmaceutical Design. 7(13). 1291–1296. 15 indexed citations
13.
Venkatachalam, T.K., E. Sudbeck, Chengzhou Mao, & Fatih M. Uckun. (2000). Stereochemistry of halopyridyl and thiazolyl thiourea compounds is a major determinant of their potency as nonnucleoside inhibitors of HIV-1 reverse transcriptase. Bioorganic & Medicinal Chemistry Letters. 10(18). 2071–2074. 38 indexed citations
14.
Zhou, Ming, Chengzhou Mao, Aida Rodríguez, et al.. (1998). Crystallization and preliminary diffraction analysis of a hyperthermostable DNA polymerase from a Thermococcus archaeon. Acta Crystallographica Section D Biological Crystallography. 54(5). 994–995. 3 indexed citations
15.
Desai‐Mehta, Ami, et al.. (1995). Structure and specificity of T cell receptors expressed by potentially pathogenic anti-DNA autoantibody-inducing T cells in human lupus.. Journal of Clinical Investigation. 95(2). 531–541. 136 indexed citations
16.
Mao, Chengzhou, et al.. (1994). T cell receptor alpha-chain repertoire of pathogenic autoantibody-inducing T cells in lupus mice.. The Journal of Immunology. 152(3). 1462–1470. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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