J. J. Catino
- Molecular Biology
- Organic Chemistry top 10%
- Immunology
- Oncology
- Pharmacology top 10%
- Co-authors
- B H LongJames BushWilliam T. BradnerClaude RothDavid M. OjciusIrah L. KingLie ZhengTerrence W. Doyle
- Topics
- Microbial Natural Products and Biosynthesis (3 papers)Cancer Treatment and Pharmacology (2 papers)Cancer therapeutics and mechanisms (1 paper)
- Cited by
- ToxicologyPharmacologyImmunology
- Journals
- Proceedings of the National Academy of SciencesThe Journal of Experimental MedicineJNCI Journal of the National Cancer Institute
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
J. J. Catino
8 papers receiving 518 citations
Peers
Comparison fields: 5 of 87
- Molecular Biology 251
- Organic Chemistry 160
- Immunology 134
- Oncology 127
- Pharmacology 108
Countries citing papers authored by J. J. Catino
This map shows the geographic impact of J. J. Catino's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. J. Catino with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. J. Catino more than expected).
Fields of papers citing papers by J. J. Catino
This network shows the impact of papers produced by J. J. Catino. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. J. Catino. The network helps show where J. J. Catino may publish in the future.
Co-authorship network of co-authors of J. J. Catino
This figure shows the co-authorship network connecting the top 25 collaborators of J. J. Catino. A scholar is included among the top collaborators of J. J. Catino based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. J. Catino. J. J. Catino is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 183 | |
| 2 | Two serum response elements mediate transcriptional repression of human smooth muscle alpha-actin promoter in ras-transformed cells. | 28 |
| 3 | Antitumor effect of keto-diepoxides isolated from the fungus Nattrassia mangiferae. | 1 |
| 4 | 93 | |
| 5 | 8 | |
| 6 | 217 | |
| 7 | 10 | |
| 8 | 4 |
About J. J. Catino
J. J. Catino is a scholar working on Microbiology, Toxicology and Pharmacology, having authored 8 papers that have together received 544 indexed citations. Recurring topics across this work include Microbial Natural Products and Biosynthesis (3 papers), Cancer Treatment and Pharmacology (2 papers) and Cancer therapeutics and mechanisms (1 paper). The work is most often cited by research in Toxicology (72 citations), Pharmacology (108 citations) and Immunology (134 citations). J. J. Catino has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include B H Long, James Bush, William T. Bradner, Claude Roth, David M. Ojcius, Irah L. King, Lie Zheng, Terrence W. Doyle, Jerzy Golik and Robert Rehfuss. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and JNCI Journal of the National Cancer Institute.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.