J. David McDonald

3.4k total citations · 1 hit paper
34 papers, 2.7k citations indexed

About

J. David McDonald is a scholar working on Molecular Biology, Clinical Biochemistry and Genetics. According to data from OpenAlex, J. David McDonald has authored 34 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 20 papers in Clinical Biochemistry and 4 papers in Genetics. Recurrent topics in J. David McDonald's work include Metabolism and Genetic Disorders (20 papers), Mitochondrial Function and Pathology (7 papers) and Metabolomics and Mass Spectrometry Studies (4 papers). J. David McDonald is often cited by papers focused on Metabolism and Genetic Disorders (20 papers), Mitochondrial Function and Pathology (7 papers) and Metabolomics and Mass Spectrometry Studies (4 papers). J. David McDonald collaborates with scholars based in United States, Canada and Australia. J. David McDonald's co-authors include William F. Dove, Jon M. Kornhauser, Phillip L. Lowrey, Martha Hotz Vitaterna, David P. King, Lawrence H. Pinto, Joseph S. Takahashi, Fred W. Turek, Alexandra Shedlovsky and Vernon C. Bode and has published in prestigious journals such as Science, New England Journal of Medicine and Proceedings of the National Academy of Sciences.

In The Last Decade

J. David McDonald

34 papers receiving 2.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mutagenesis and Mapping of a Mouse Gene, Clock , Essential for Circadian Behavior

1994 1.3k citations Martha Hotz Vitaterna, David P. King et al. Science profile →

Peers

J. David McDonald

EAS

MB

PHYSI

CB

GENET

Steven J. Swoap United States

Jerry Vriend Canada

M.E. Stoeckel France

Felino R. Cagampang United Kingdom

Dan Zhou United States

Atsuhiko Hattori Japan

Jon J. Ramsey United States

G. Csaba Hungary

Qing‐Jun Meng United Kingdom

Kinji Inoue Japan

Steven J. Swoap United States

J. David McDonald

603 ×0.6

EAS

1.4k ×1.4

MB

1.6k ×2.3

PHYSI

61 ×0.1

CB

242 ×0.6

GENET

Citations per year, relative to J. David McDonald J. David McDonald (= 1×) peers Steven J. Swoap

Countries citing papers authored by J. David McDonald

Since Specialization

Citations

This map shows the geographic impact of J. David McDonald's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. David McDonald with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. David McDonald more than expected).

Fields of papers citing papers by J. David McDonald

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. David McDonald. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. David McDonald. The network helps show where J. David McDonald may publish in the future.

Co-authorship network of co-authors of J. David McDonald

This figure shows the co-authorship network connecting the top 25 collaborators of J. David McDonald. A scholar is included among the top collaborators of J. David McDonald based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. David McDonald. J. David McDonald is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Sidell, Neil, Lijuan Hao, Marzia Pasquali, & J. David McDonald. (2009). Carcinogenic Effects in a Phenylketonuria Mouse Model. PLoS ONE. 4(1). e4292–e4292. 17 indexed citations

2.

McDonald, J. David, et al.. (2008). Recovery and STR Amplification of DNA from RFLP Membranes*. Journal of Forensic Sciences. 53(2). 349–358. 2 indexed citations

3.

Lund, Amie K., Travis L. Knuckles, Ralph V. Shohet, et al.. (2006). Gasoline Exhaust Emissions Induce Vascular Remodeling Pathways Involved in Atherosclerosis. Toxicological Sciences. 95(2). 485–494. 86 indexed citations

4.

Gudi, Ramadevi, et al.. (2006). Health effects of subchronic exposure to diesel-water-methanol emulsion emission. Toxicology and Industrial Health. 22(2). 65–85. 9 indexed citations

5.

Reed, Matthew D., et al.. (2005). Health Effects of Subchronic Exposure to Diesel–Water Emulsion Emission. Inhalation Toxicology. 17(14). 851–870. 7 indexed citations

6.

Surendran, Sankar, Gerald A. Campbell, Stephen K. Tyring, et al.. (2003). High Levels of Orexin A in the Brain of the Mouse Model for Phenylketonuria: Possible Role of Orexin A in Hyperactivity Seen in Children with PKU. Neurochemical Research. 28(12). 1891–1894. 10 indexed citations

7.

Erçal, Nuran, Nükhet Aykin‐Burns, Hande Gürer-Orhan, & J. David McDonald. (2002). Oxidative stress in a phenylketonuria animal model. Free Radical Biology and Medicine. 32(9). 906–911. 44 indexed citations

8.

Herron, Bruce J., Lu W, Shanming Liu, et al.. (2002). Efficient generation and mapping of recessive developmental mutations using ENU mutagenesis. Nature Genetics. 30(2). 185–189. 151 indexed citations

9.

McDonald, J. David, et al.. (2001). Effect of Maternal Blood Phenylalanine Level on Mouse Maternal Phenylketonuria Offspring. Molecular Genetics and Metabolism. 74(4). 420–425. 12 indexed citations

10.

McDonald, J. David, et al.. (2001). Characterization of the Mouse Phenylalanine Hydroxylase Mutation Pahenu3. Molecular Genetics and Metabolism. 72(1). 27–30. 12 indexed citations

11.

McDonald, J. David. (2000). Production of Mouse Models for the Study of Human Inborn Errors of Metabolism. Molecular Genetics and Metabolism. 71(1-2). 240–244. 4 indexed citations

12.

Sarkissian, Christineh N., et al.. (2000). A Heteroallelic Mutant Mouse Model: A New Orthologue for Human Hyperphenylalaninemia. Molecular Genetics and Metabolism. 69(3). 188–194. 40 indexed citations

13.

McDonald, J. David, et al.. (1999). Linkage Analysis of the hph-1 Mutation and the GTP Cyclohydrolase I Structural Gene. Molecular Genetics and Metabolism. 68(1). 91–92. 5 indexed citations

14.

McDonald, J. David, et al.. (1997). Characterization of Mutations at the Mouse Phenylalanine Hydroxylase Locus. Genomics. 39(3). 402–405. 75 indexed citations

15.

McDonald, J. David. (1994). The PKU mouse project: its history, potential and implications. Acta Paediatrica. 83(s407). 122–123. 7 indexed citations

16.

Vitaterna, Martha Hotz, David P. King, Jon M. Kornhauser, et al.. (1994). Mutagenesis and Mapping of a Mouse Gene, Clock , Essential for Circadian Behavior. Science. 264(5159). 719–725. 1274 indexed citations breakdown →

17.

Gütlich, Markus, Irmgard Ziegler, K. Witter, et al.. (1994). Molecular Characterization of HPH-1: A Mouse Mutant Deficient in GTP Cyclohydrolase I Activity. Biochemical and Biophysical Research Communications. 203(3). 1675–1681. 19 indexed citations

18.

Shedlovsky, Alexandra, J. David McDonald, Derek J. Symula, & William F. Dove. (1993). Mouse models of human phenylketonuria.. Genetics. 134(4). 1205–1210. 192 indexed citations

19.

McDonald, J. David, Richard G.H. Cotton, Ian G. Jennings, et al.. (1988). Biochemical Defect of the hph‐1 Mouse Mutant Is a Deficiency in GTP‐Cyclohydrolase Activity. Journal of Neurochemistry. 50(2). 655–657. 51 indexed citations

20.

Bode, Vernon C., J. David McDonald, Jean‐Louis Guénet, & Daniel Simon. (1988). hph-1: a mouse mutant with hereditary hyperphenylalaninemia induced by ethylnitrosourea mutagenesis.. Genetics. 118(2). 299–305. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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