J Barbosa

1.0k total citations
20 papers, 857 citations indexed

About

J Barbosa is a scholar working on Genetics, Immunology and Surgery. According to data from OpenAlex, J Barbosa has authored 20 papers receiving a total of 857 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Genetics, 9 papers in Immunology and 5 papers in Surgery. Recurrent topics in J Barbosa's work include Diabetes and associated disorders (13 papers), Immune Cell Function and Interaction (6 papers) and T-cell and B-cell Immunology (5 papers). J Barbosa is often cited by papers focused on Diabetes and associated disorders (13 papers), Immune Cell Function and Interaction (6 papers) and T-cell and B-cell Immunology (5 papers). J Barbosa collaborates with scholars based in United States, Canada and Germany. J Barbosa's co-authors include Stephen S. Rich, Barbara E. Bierer, Steven J. Burakoff, G. Thomson, Wendy P. Robinson, Mary K. Kuhner, Sung-Hong Joe, J. Bertrams, Michael J. MacDonald and JL Gottschall and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

J Barbosa

20 papers receiving 789 citations

Peers

J Barbosa

GENET

IMMUN

SURGE

EDM

V. Lepage France

Merana Tamir Israel

M S Pollack United States

Rupert Kenefeck United Kingdom

Daniel Rodríguez‐Pinto United States

Peter Eerligh Netherlands

Joanna D. Davies United States

Kevin Goudy United States

Alan W. Steele United States

D K Didier United States

V. Lepage France

J Barbosa

473 ×0.9

GENET

739 ×1.9

IMMUN

245 ×0.8

SURGE

195 ×0.9

EDM

87 ×0.7

Citations per year, relative to J Barbosa J Barbosa (= 1×) peers V. Lepage

Countries citing papers authored by J Barbosa

Since Specialization

Citations

This map shows the geographic impact of J Barbosa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J Barbosa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J Barbosa more than expected).

Fields of papers citing papers by J Barbosa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J Barbosa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J Barbosa. The network helps show where J Barbosa may publish in the future.

Co-authorship network of co-authors of J Barbosa

This figure shows the co-authorship network connecting the top 25 collaborators of J Barbosa. A scholar is included among the top collaborators of J Barbosa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J Barbosa. J Barbosa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Barbosa, J, et al.. (2021). Human Immunodeficiency Virus Type 1 Vpr Mediates Degradation of APC1, a Scaffolding Component of the Anaphase-Promoting Complex/Cyclosome. Journal of Virology. 95(15). e0097120–e0097120. 6 indexed citations

Barbosa, J, et al.. (2018). HIV-1 Vpr hijacks EDD-DYRK2-DDB1DCAF1 to disrupt centrosome homeostasis. Journal of Biological Chemistry. 293(24). 9448–9460. 17 indexed citations

Lodge, Robert, J Barbosa, Julian C. Gilmore, et al.. (2017). Host MicroRNAs-221 and -222 Inhibit HIV-1 Entry in Macrophages by Targeting the CD4 Viral Receptor. Cell Reports. 21(1). 141–153. 57 indexed citations

Lodge, Robert, et al.. (2017). Regulation of CD4 Receptor and HIV-1 Entry by MicroRNAs-221 and -222 during Differentiation of THP-1 Cells. Viruses. 10(1). 13–13. 11 indexed citations

Barbosa, J, Josée Labrie, Francis Beaudry, Carl A. Gagnon, & Mario Jacques. (2015). Actinobacillus pleuropneumoniae induces SJPL cell cycle arrest in G2/M-phase and inhibits porcine reproductive and respiratory syndrome virus replication. Virology Journal. 12(1). 188–188. 6 indexed citations

Rich, Stephen S., S. Scott Panter, F. C. Goetz, Bo E. Hedlund, & J Barbosa. (1991). Shared genetic susceptibility of Type 1 (insulin-dependent) and Type 2 (non-insulin-dependent) diabetes mellitus: contributions of HLA and haptoglobin. Diabetologia. 34(5). 350–355. 17 indexed citations

Bierer, Barbara E., J Barbosa, S H Herrmann, & Steven J. Burakoff. (1988). Interaction of CD2 with its ligand, LFA-3, in human T cell proliferation.. The Journal of Immunology. 140(10). 3358–3363. 81 indexed citations

Bierer, Barbara E., Andrew S. Peterson, J Barbosa, Brian Seed, & Steven J. Burakoff. (1988). Expression of the T-cell surface molecule CD2 and an epitope-loss CD2 mutant to define the role of lymphocyte function-associated antigen 3 (LFA-3) in T-cell activation.. Proceedings of the National Academy of Sciences. 85(4). 1194–1198. 71 indexed citations

Thomson, G., Wendy P. Robinson, Mary K. Kuhner, et al.. (1988). Genetic heterogeneity, modes of inheritance, and risk estimates for a joint study of Caucasians with insulin-dependent diabetes mellitus.. PubMed. 43(6). 799–816. 302 indexed citations

10.

Rich, Stephen S., et al.. (1987). A combined segregation and linkage analysis of insulin-dependent diabetes mellitus.. PubMed. 40(3). 237–49. 11 indexed citations

11.

Rich, Stephen S., L.R. Weitkamp, S.A. Guttormsen, & J Barbosa. (1986). Gm, Km, and HLA in Insulin-Dependent Type I Diabetes Mellitus: A Log-Linear Analysis of Association. Diabetes. 35(8). 927–932. 19 indexed citations

12.

Rich, Stephen S., G. J. O’Neill, Agustin P. Dalmasso, C. Nerl, & J Barbosa. (1985). Complement and HLA: Further Definition of High-Risk Haplotypes in Insulin-dependent Diabetes. Diabetes. 34(5). 504–509. 29 indexed citations

13.

Rich, Stephen S., L.R. Weitkamp, & J Barbosa. (1984). Genetic heterogeneity of insulin-dependent (type I) diabetes mellitus: evidence from a study of extended haplotypes.. PubMed. 36(5). 1015–23. 32 indexed citations

14.

Barbosa, J, et al.. (1984). Do genetic factors play a role in the pathogenesis of diabetic microangiopathy?. Diabetologia. 27(5). 487–492. 56 indexed citations

15.

Morton, Newton E., Arne Svejgaard, J Barbosa, et al.. (1983). Heterozygous expression of insulin-dependent diabetes mellitus (IDDM) determinants in the HLA system.. PubMed. 35(2). 201–13. 42 indexed citations

16.

Barbosa, J. (1981). Is diabetic microangiopathy genetically heterogeneous? HLA and diabetic nephropathy.. PubMed. 11. 77–80. 3 indexed citations

17.

Barbosa, J, et al.. (1979). HLA—Dw Antigens in Unrelated Juvenile, Insulin‐Dependent Diabetics. Tissue Antigens. 14(5). 426–436. 18 indexed citations

18.

Barbosa, J, M M Chern, H. Noreen, & Vernon Anderson. (1978). Analysis of linkage between the major histocompatibility system and juvenile, insulin-dependent diabetes in multiplex families. Reanalysis of data.. Journal of Clinical Investigation. 62(2). 492–495. 45 indexed citations

19.

Barbosa, J, et al.. (1978). HLA-DR antigens and unique serologic reactions in juvenile-onset diabetes mellitus.. PubMed. 10(4). 967–9. 11 indexed citations

20.

Barbosa, J, et al.. (1976). Histocompatibility (HLA) Antigens and Diabetic Microangiopathy*. Tissue Antigens. 7(4). 233–237. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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