Iva Potočnjak

893 total citations
16 papers, 773 citations indexed

About

Iva Potočnjak is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Epidemiology. According to data from OpenAlex, Iva Potočnjak has authored 16 papers receiving a total of 773 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Pathology and Forensic Medicine, 6 papers in Molecular Biology and 3 papers in Epidemiology. Recurrent topics in Iva Potočnjak's work include Chemotherapy-induced organ toxicity mitigation (10 papers), Genomics, phytochemicals, and oxidative stress (5 papers) and Autophagy in Disease and Therapy (3 papers). Iva Potočnjak is often cited by papers focused on Chemotherapy-induced organ toxicity mitigation (10 papers), Genomics, phytochemicals, and oxidative stress (5 papers) and Autophagy in Disease and Therapy (3 papers). Iva Potočnjak collaborates with scholars based in Croatia and Egypt. Iva Potočnjak's co-authors include Robert Domitrović, Iva Vukelić, Marko Škoda, Lara Batičić, Ivana Gobin, Dijana Detel, Željka Crnčević-Orlić, Dalibor Broznić, Ester Pernjak Pugel and Sanda Vladimir‐Knežević and has published in prestigious journals such as Food and Chemical Toxicology, Molecular Nutrition & Food Research and Phytotherapy Research.

In The Last Decade

Iva Potočnjak

16 papers receiving 762 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Iva Potočnjak Croatia 13 270 183 173 106 87 16 773
Marko Škoda Croatia 8 219 0.8× 206 1.1× 173 1.0× 116 1.1× 68 0.8× 9 755
Yunru Peng China 18 399 1.5× 106 0.6× 123 0.7× 114 1.1× 91 1.0× 42 832
Ahmed M. Sayed Egypt 19 405 1.5× 166 0.9× 123 0.7× 105 1.0× 54 0.6× 67 997
Nahla E. El‐Ashmawy Egypt 21 380 1.4× 105 0.6× 154 0.9× 89 0.8× 83 1.0× 67 1.1k
Mukesh Nandave India 18 269 1.0× 105 0.6× 101 0.6× 150 1.4× 54 0.6× 52 848
Muobarak J. Tuorkey Egypt 13 386 1.4× 134 0.7× 115 0.7× 136 1.3× 41 0.5× 25 971
Kesavan Sabitha India 15 388 1.4× 140 0.8× 154 0.9× 149 1.4× 105 1.2× 34 1.1k
Suvara K. Wattanapitayakul Thailand 15 311 1.2× 104 0.6× 176 1.0× 112 1.1× 45 0.5× 32 1.0k
Mohamed A. Kandeil Egypt 16 304 1.1× 88 0.5× 93 0.5× 100 0.9× 127 1.5× 44 872
Jinyong Peng China 17 381 1.4× 73 0.4× 167 1.0× 84 0.8× 116 1.3× 27 788

Countries citing papers authored by Iva Potočnjak

Since Specialization
Citations

This map shows the geographic impact of Iva Potočnjak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Iva Potočnjak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Iva Potočnjak more than expected).

Fields of papers citing papers by Iva Potočnjak

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Iva Potočnjak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Iva Potočnjak. The network helps show where Iva Potočnjak may publish in the future.

Co-authorship network of co-authors of Iva Potočnjak

This figure shows the co-authorship network connecting the top 25 collaborators of Iva Potočnjak. A scholar is included among the top collaborators of Iva Potočnjak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Iva Potočnjak. Iva Potočnjak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Potočnjak, Iva, et al.. (2022). Sinomenine mitigates cisplatin-induced kidney injury by targeting multiple signaling pathways. Food and Chemical Toxicology. 171. 113538–113538. 12 indexed citations
2.
Potočnjak, Iva, et al.. (2022). Oleanolic acid induces HCT116 colon cancer cell death through the p38/FOXO3a/Sirt6 pathway. Chemico-Biological Interactions. 363. 110010–110010. 28 indexed citations
3.
Vukelić, Iva, Dijana Detel, Lara Batičić, Iva Potočnjak, & Robert Domitrović. (2020). Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy. Food and Chemical Toxicology. 145. 111680–111680. 54 indexed citations
4.
Potočnjak, Iva, et al.. (2020). Aucubin administered by either oral or parenteral route protects against cisplatin-induced acute kidney injury in mice. Food and Chemical Toxicology. 142. 111472–111472. 35 indexed citations
5.
Potočnjak, Iva, et al.. (2020). Antitumor activity of luteolin in human colon cancer SW620 cells is mediated by the ERK/FOXO3a signaling pathway. Toxicology in Vitro. 66. 104852–104852. 58 indexed citations
6.
Potočnjak, Iva, et al.. (2019). Oleanolic acid attenuates cisplatin-induced nephrotoxicity in mice and chemosensitizes human cervical cancer cells to cisplatin cytotoxicity. Food and Chemical Toxicology. 132. 110676–110676. 44 indexed citations
7.
Potočnjak, Iva, Ivana Gobin, & Robert Domitrović. (2018). Carvacrol induces cytotoxicity in human cervical cancer cells but causes cisplatin resistance: Involvement of MEK–ERK activation. Phytotherapy Research. 32(6). 1090–1097. 55 indexed citations
8.
Vukelić, Iva, et al.. (2018). Chlorogenic acid ameliorates experimental colitis in mice by suppressing signaling pathways involved in inflammatory response and apoptosis. Food and Chemical Toxicology. 121. 140–150. 77 indexed citations
9.
Potočnjak, Iva, et al.. (2017). Stevia and stevioside protect against cisplatin nephrotoxicity through inhibition of ERK1/2, STAT3, and NF-κB activation. Food and Chemical Toxicology. 107(Pt A). 215–225. 54 indexed citations
10.
Potočnjak, Iva, Robert Domitrović, & Ivana Gobin. (2016). Carvacrol attenuates acute kidney injury induced by cisplatin through suppression of MAPK and PI3K/Akt pathways. 73. 1 indexed citations
11.
Potočnjak, Iva & Robert Domitrović. (2016). Carvacrol attenuates acute kidney injury induced by cisplatin through suppression of ERK and PI3K/Akt activation. Food and Chemical Toxicology. 98(Pt B). 251–261. 74 indexed citations
12.
Domitrović, Robert & Iva Potočnjak. (2015). A comprehensive overview of hepatoprotective natural compounds: mechanism of action and clinical perspectives. Archives of Toxicology. 90(1). 39–79. 114 indexed citations
13.
Potočnjak, Iva, et al.. (2015). Oral administration of oleuropein attenuates cisplatin‐induced acute renal injury in mice through inhibition of ERK signaling. Molecular Nutrition & Food Research. 60(3). 530–541. 59 indexed citations
14.
Domitrović, Robert, et al.. (2015). Oleuropein ameliorates cisplatin-induced oxidative damage, inflammation and apoptosis in mice kidneys by modulating CYP2E1, NF-κB and ERK1/2 expression. 222. 1 indexed citations
15.
Domitrović, Robert, Iva Potočnjak, Željka Crnčević-Orlić, & Marko Škoda. (2014). Nephroprotective activities of rosmarinic acid against cisplatin-induced kidney injury in mice. Food and Chemical Toxicology. 66. 321–328. 83 indexed citations
16.
Rashed, Khaled, Iva Potočnjak, Jasminka Giacometti, Marko Škoda, & Robert Domitrović. (2014). Terminalia bellerica aerial parts ethyl acetate extract exhibits antioxidant, anti-inflammatory and antifibrotic activity in carbon tetrachloride-intoxicated mice. Journal of Functional Foods. 8. 319–330. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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