Itamar Barash

2.7k total citations · 1 hit paper
59 papers, 2.2k citations indexed

About

Itamar Barash is a scholar working on Genetics, Molecular Biology and Oncology. According to data from OpenAlex, Itamar Barash has authored 59 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Genetics, 28 papers in Molecular Biology and 21 papers in Oncology. Recurrent topics in Itamar Barash's work include Animal Genetics and Reproduction (23 papers), Cancer Cells and Metastasis (14 papers) and Virus-based gene therapy research (13 papers). Itamar Barash is often cited by papers focused on Animal Genetics and Reproduction (23 papers), Cancer Cells and Metastasis (14 papers) and Virus-based gene therapy research (13 papers). Itamar Barash collaborates with scholars based in Israel, United States and Germany. Itamar Barash's co-authors include Clement C. Cheung, Joseph L. Kuijper, E B Kabigting, David S. Weigle, Robert A. Steiner, Donald K. Clifton, Hong Ren, Bernd Groner, Elena Iavnilovitch and Gat Rauner and has published in prestigious journals such as Nucleic Acids Research, PLoS ONE and Biochemical and Biophysical Research Communications.

In The Last Decade

Itamar Barash

59 papers receiving 2.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Leptin is a metabolic signal to the reproductive system.

1996 887 citations Itamar Barash et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Itamar Barash Israel	22	743	677	591	440	426	59	2.2k
D. J. Flint United Kingdom	27	143 0.2×	706 1.0×	607 1.0×	446 1.0×	330 0.8×	74	2.4k
C. Morales Spain	23	388 0.5×	259 0.4×	167 0.3×	80 0.2×	220 0.5×	39	1.4k
Shinya Ohagi Japan	12	443 0.6×	802 1.2×	449 0.8×	154 0.3×	238 0.6×	23	2.0k
Louise Metherell United Kingdom	34	586 0.8×	1.6k 2.4×	947 1.6×	102 0.2×	444 1.0×	123	3.6k
Yvonne Hort Australia	19	170 0.2×	1.0k 1.5×	976 1.7×	299 0.7×	54 0.1×	32	2.4k
S. Saito Japan	21	252 0.3×	280 0.4×	103 0.2×	249 0.6×	113 0.3×	51	1.4k
Gregory W. Aponte United States	21	192 0.3×	932 1.4×	198 0.3×	137 0.3×	200 0.5×	31	1.8k
Beth S. Schachter United States	22	138 0.2×	555 0.8×	602 1.0×	167 0.4×	44 0.1×	34	1.6k
Dongsi Lu United States	14	1.1k 1.5×	387 0.6×	194 0.3×	107 0.2×	1.4k 3.4×	28	2.3k
Changjiu He China	24	545 0.7×	758 1.1×	272 0.5×	86 0.2×	48 0.1×	66	2.2k

Countries citing papers authored by Itamar Barash

Since Specialization

Citations

This map shows the geographic impact of Itamar Barash's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Itamar Barash with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Itamar Barash more than expected).

Fields of papers citing papers by Itamar Barash

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Itamar Barash. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Itamar Barash. The network helps show where Itamar Barash may publish in the future.

Co-authorship network of co-authors of Itamar Barash

This figure shows the co-authorship network connecting the top 25 collaborators of Itamar Barash. A scholar is included among the top collaborators of Itamar Barash based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Itamar Barash. Itamar Barash is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Barash, Itamar. (2025). Mammalian Species-Specific Resistance to Mammary Cancer. Journal of Mammary Gland Biology and Neoplasia. 30(1). 3–3. 1 indexed citations

Salame, Tomer‐Meir, et al.. (2021). Newly characterized bovine mammary stromal region with epithelial properties supports representative epithelial outgrowth development from transplanted stem cells. Cell and Tissue Research. 387(1). 39–61. 2 indexed citations

Barash, Itamar, et al.. (2019). Calorie restriction and rapamycin administration induce stem cell self-renewal and consequent development and production in the mammary gland. Experimental Cell Research. 382(2). 111477–111477. 6 indexed citations

Rauner, Gat, et al.. (2018). High Expression of CD200 and CD200R1 Distinguishes Stem and Progenitor Cell Populations within Mammary Repopulating Units. Stem Cell Reports. 11(1). 288–302. 9 indexed citations

Rauner, Gat & Itamar Barash. (2015). Enrichment for Repopulating Cells and Identification of Differentiation Markers in the Bovine Mammary Gland. Journal of Mammary Gland Biology and Neoplasia. 21(1-2). 41–49. 14 indexed citations

Barash, Itamar. (2012). Stat5 in breast cancer: potential oncogenic activity coincides with positive prognosis for the disease. Carcinogenesis. 33(12). 2320–2325. 27 indexed citations

Rauner, Gat, et al.. (2011). Conditional repression of STAT5 expression during lactation reveals its exclusive roles in mammary gland morphology, milk‐protein gene expression, and neonate growth. Molecular Reproduction and Development. 78(8). 585–596. 24 indexed citations

Barash, Itamar, et al.. (2010). Forced activation of Stat5 subjects mammary epithelial cells to DNA damage and preferential induction of the cellular response mechanism during proliferation. Journal of Cellular Physiology. 226(3). 616–626. 10 indexed citations

Barash, Itamar, et al.. (2009). Distinct gene-expression profiles characterize mammary tumors developed in transgenic mice expressing constitutively active and C-terminally truncated variants of STAT5. BMC Genomics. 10(1). 231–231. 7 indexed citations

10.

Barash, Itamar, et al.. (2008). Different gene-expression profiles for the poorly differentiated carcinoma and the highly differentiated papillary adenocarcinoma in mammary glands support distinct metabolic pathways. BMC Cancer. 8(1). 270–270. 9 indexed citations

11.

Desrivières, Sylvane, Christian Künz, Itamar Barash, et al.. (2006). The Biological Functions of the Versatile Transcription Factors STAT3 and STAT5 and New Strategies for their Targeted Inhibition. Journal of Mammary Gland Biology and Neoplasia. 11(1). 75–87. 67 indexed citations

12.

Barash, Itamar. (2006). Stat5 in the mammary gland: Controlling normal development and cancer. Journal of Cellular Physiology. 209(2). 305–313. 43 indexed citations

13.

Iavnilovitch, Elena, Robert D. Cardiff, Bernd Groner, & Itamar Barash. (2004). Deregulation of Stat5 expression and activation causes mammary tumors in transgenic mice. International Journal of Cancer. 112(4). 607–619. 75 indexed citations

14.

Barash, Itamar, et al.. (2003). Low-energy laser irradiation enhances de novo protein synthesis via its effects on translation-regulatory proteins in skeletal muscle myoblasts. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1593(2-3). 131–139. 72 indexed citations

15.

Ilan, Neta, et al.. (1996). β-Lactoglobulin/Human Serum Albumin Fusion Genes Do Not Respond Accurately to Signals from the Extracellular Matrix in Mammary Epithelial Cells from Transgenic Mice. Experimental Cell Research. 228(1). 146–159. 3 indexed citations

16.

Barash, Itamar, et al.. (1996). Co-integration of β-lactoglobulin/human serum albumin hybrid genes with the entire β-lactoglobulin gene or the matrix attachment region element: Repression of human serum albumin and β-lactoglobulin expression in the mammary gland and dual regulation of the transgenes. Molecular Reproduction and Development. 45(4). 421–430. 9 indexed citations

17.

Barash, Itamar, et al.. (1994). Ectopic expression of β-lactoglobulin/human serum albumin fusion genes in transgenic mice: hormonal regulation andin situ localization. Transgenic Research. 3(3). 141–151. 27 indexed citations

18.

Barash, Itamar, Zafrira Nitsan, & I. Nir. (1992). Metabolic and behavioural adaptation of light‐bodied chicks to meal feeding. British Poultry Science. 33(2). 271–278. 10 indexed citations

19.

Barash, Itamar & Barry I. Posner. (1989). Homologous induction of growth hormone receptors in cultured rat hepatocytes. Molecular and Cellular Endocrinology. 62(2). 281–286. 17 indexed citations

20.

Barash, Itamar, Zecharia Madar, & Arieh Gertler. (1983). Down-regulation of prolactin receptors in the liver, mammary gland and kidney of female virgin rat, infused with ovine prolactin or human growth hormone. Biochemical and Biophysical Research Communications. 116(2). 644–650. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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