Itai Strominger

663 total citations
8 papers, 459 citations indexed

About

Itai Strominger is a scholar working on Neurology, Physiology and Immunology. According to data from OpenAlex, Itai Strominger has authored 8 papers receiving a total of 459 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Neurology, 3 papers in Physiology and 3 papers in Immunology. Recurrent topics in Itai Strominger's work include Neuroinflammation and Neurodegeneration Mechanisms (6 papers), T-cell and B-cell Immunology (3 papers) and Alzheimer's disease research and treatments (3 papers). Itai Strominger is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (6 papers), T-cell and B-cell Immunology (3 papers) and Alzheimer's disease research and treatments (3 papers). Itai Strominger collaborates with scholars based in Israel. Itai Strominger's co-authors include Alon Monsonego, Anna Nemirovsky, Yehezqel Elyahu, Omer Berner, Ekaterina Eremenko, Idan Hekselman, Vered Chalifa‐Caspi, Nir Friedman, Esti Yeger‐Lotem and Maya Schiller and has published in prestigious journals such as The Journal of Immunology, Science Advances and Frontiers in Immunology.

In The Last Decade

Itai Strominger

8 papers receiving 457 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Itai Strominger Israel	7	214	198	131	102	101	8	459
Yehezqel Elyahu Israel	9	204 1.0×	232 1.2×	114 0.9×	121 1.2×	79 0.8×	10	518
Omer Berner Israel	5	167 0.8×	180 0.9×	104 0.8×	95 0.9×	68 0.7×	7	396
Carolina Manganeli Polonio Brazil	7	171 0.8×	97 0.5×	62 0.5×	139 1.4×	63 0.6×	13	374
Shumei Man United States	8	340 1.6×	122 0.6×	179 1.4×	122 1.2×	75 0.7×	10	626
Jesús Amo‐Aparicio Spain	11	154 0.7×	155 0.8×	49 0.4×	158 1.5×	25 0.2×	16	467
Alanna G. Spiteri Australia	11	199 0.9×	161 0.8×	46 0.4×	103 1.0×	21 0.2×	22	421
Xianyuan Song United States	12	182 0.9×	154 0.8×	43 0.3×	150 1.5×	32 0.3×	23	597
Nehal Narayan United Kingdom	6	113 0.5×	74 0.4×	40 0.3×	61 0.6×	50 0.5×	19	317
Aoife McGinley Ireland	6	91 0.4×	233 1.2×	37 0.3×	95 0.9×	37 0.4×	8	383
Michael Fassler Israel	8	161 0.8×	76 0.4×	57 0.4×	94 0.9×	21 0.2×	15	351

Countries citing papers authored by Itai Strominger

Since Specialization

Citations

This map shows the geographic impact of Itai Strominger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Itai Strominger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Itai Strominger more than expected).

Fields of papers citing papers by Itai Strominger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Itai Strominger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Itai Strominger. The network helps show where Itai Strominger may publish in the future.

Co-authorship network of co-authors of Itai Strominger

This figure shows the co-authorship network connecting the top 25 collaborators of Itai Strominger. A scholar is included among the top collaborators of Itai Strominger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Itai Strominger. Itai Strominger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Strominger, Itai, et al.. (2020). Protein kinase C eta is activated in reactive astrocytes of an Alzheimer's disease mouse model: Evidence for its immunoregulatory function in primary astrocytes. Glia. 69(3). 697–714. 9 indexed citations

2.

Eremenko, Ekaterina, Omer Berner, Yehezqel Elyahu, et al.. (2019). CD4 T Cells Induce A Subset of MHCII-Expressing Microglia that Attenuates Alzheimer Pathology. iScience. 16. 298–311. 82 indexed citations

3.

Elyahu, Yehezqel, Idan Hekselman, Omer Berner, et al.. (2019). Aging promotes reorganization of the CD4 T cell landscape toward extreme regulatory and effector phenotypes. Science Advances. 5(8). eaaw8330–eaaw8330. 190 indexed citations

4.

Strominger, Itai, Yehezqel Elyahu, Omer Berner, et al.. (2018). The Choroid Plexus Functions as a Niche for T-Cell Stimulation Within the Central Nervous System. Frontiers in Immunology. 9. 1066–1066. 82 indexed citations

5.

Bhattacharya, Udayan, et al.. (2017). Old Mice Accumulate Activated Effector CD4 T Cells Refractory to Regulatory T Cell-Induced Immunosuppression. Frontiers in Immunology. 8. 283–283. 16 indexed citations

6.

Strominger, Itai, et al.. (2016). TGF-β affinity-bound to a macroporous alginate scaffold generates local and peripheral immunotolerant responses and improves allocell transplantation. Acta Biomaterialia. 45. 196–209. 22 indexed citations

7.

Strominger, Itai, et al.. (2014). Th1 polarization of T cells injected into the cerebrospinal fluid induces brain immunosurveillance. Journal of Neuroimmunology. 275(1-2). 72–72. 1 indexed citations

8.

Fisher, Yair, et al.. (2013). Th1 Polarization of T Cells Injected into the Cerebrospinal Fluid Induces Brain Immunosurveillance. The Journal of Immunology. 192(1). 92–102. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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