István Likó
- Molecular Biology
- Cancer Research top 5%
- Endocrinology, Diabetes and Metabolism top 5%
- Surgery
- Genetics top 10%
- Co-authors
- Attila PatócsKàroly RáczPéter IgazHenriett ButzSándor CzirjákMárta KorbonitsPéter M. SzabóMiklós Sárvári
- Topics
- MicroRNA in disease regulation (10 papers)Adrenal and Paraganglionic Tumors (10 papers)Cancer, Hypoxia, and Metabolism (10 papers)
- Partner nations
- HungaryUnited StatesUnited Kingdom
In The Last Decade
István Likó
69 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 669
- Cancer Research 483
- Endocrinology, Diabetes and Metabolism 348
- Surgery 242
- Genetics 182
Countries citing papers authored by István Likó
This map shows the geographic impact of István Likó's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by István Likó with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites István Likó more than expected).
Fields of papers citing papers by István Likó
This network shows the impact of papers produced by István Likó. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by István Likó. The network helps show where István Likó may publish in the future.
Co-authorship network of co-authors of István Likó
This figure shows the co-authorship network connecting the top 25 collaborators of István Likó. A scholar is included among the top collaborators of István Likó based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with István Likó. István Likó is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 3 | |
| 5 | 8 | |
| 6 | 1 | |
| 7 | 8 | |
| 8 | 7 | |
| 9 | 4 | |
| 10 | 3 | |
| 11 | 10 | |
| 12 | 34 | |
| 13 | 75 | |
| 14 | 23 | |
| 15 | 12 | |
| 16 | [Investigation of CNTF, COMT, DDR1, DISC1, DRD2, DRD3, and DTNBP1 candidate genes in schizophrenia: Results from the Hungarian SCHIZOBANK Consortium]. | 7 |
| 17 | 9 | |
| 18 | 3 | |
| 19 | 15 | |
| 20 | MicroRNA expression in human sporadic pituitary adenomas | 2 |
About István Likó
István Likó is a scholar working on Cancer Research, Behavioral Neuroscience and Endocrinology, Diabetes and Metabolism, having authored 71 papers that have together received 1.4k indexed citations. Recurring topics across this work include MicroRNA in disease regulation (10 papers), Adrenal and Paraganglionic Tumors (10 papers) and Cancer, Hypoxia, and Metabolism (10 papers). The work is most often cited by research in Cancer Research (483 citations), Behavioral Neuroscience (80 citations) and Endocrinology, Diabetes and Metabolism (348 citations). István Likó has collaborated with scholars based in Hungary, United States and United Kingdom. Frequent co-authors include Attila Patócs, Kàroly Rácz, Péter Igaz, Henriett Butz, Sándor Czirják, Márta Korbonits, Péter M. Szabó, Miklós Sárvári, Zsófia Tömböl and Zsolt Liposits. Their work appears in journals such as PLoS ONE, The Journal of Clinical Endocrinology & Metabolism and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.