István Gyertyán
- Cellular and Molecular Neuroscience top 1%
- Molecular Biology top 10%
- Psychiatry and Mental health top 2%
- Pharmacology top 5%
- Cognitive Neuroscience top 5%
- Co-authors
- Béla KissNika AdhamI. LaszlovszkyKrisztina GálKatalin SághyJudit LaszyAndrás Bilkei‐GorzóGyörgy Domány
- Topics
- Neurotransmitter Receptor Influence on Behavior (36 papers)Neuroscience and Neuropharmacology Research (32 papers)Receptor Mechanisms and Signaling (23 papers)
- Partner nations
- HungaryUnited StatesSweden
In The Last Decade
István Gyertyán
81 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 95
- Cellular and Molecular Neuroscience 1.2k
- Molecular Biology 762
- Psychiatry and Mental health 758
- Pharmacology 326
- Cognitive Neuroscience 279
Countries citing papers authored by István Gyertyán
This map shows the geographic impact of István Gyertyán's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by István Gyertyán with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites István Gyertyán more than expected).
Fields of papers citing papers by István Gyertyán
This network shows the impact of papers produced by István Gyertyán. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by István Gyertyán. The network helps show where István Gyertyán may publish in the future.
Co-authorship network of co-authors of István Gyertyán
This figure shows the co-authorship network connecting the top 25 collaborators of István Gyertyán. A scholar is included among the top collaborators of István Gyertyán based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with István Gyertyán. István Gyertyán is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 9 | |
| 3 | 16 | |
| 4 | 16 | |
| 5 | 14 | |
| 6 | 45 | |
| 7 | 9 | |
| 8 | 65 | |
| 9 | 11 | |
| 10 | 14 | |
| 11 | 84 | |
| 12 | 41 | |
| 13 | 3 | |
| 14 | 39 | |
| 15 | 29 | |
| 16 | 59 | |
| 17 | 32 | |
| 18 | 122 | |
| 19 | 39 | |
| 20 | 107 |
About István Gyertyán
István Gyertyán is a scholar working on Cellular and Molecular Neuroscience, Biological Psychiatry and Behavioral Neuroscience, having authored 83 papers that have together received 2.2k indexed citations. Recurring topics across this work include Neurotransmitter Receptor Influence on Behavior (36 papers), Neuroscience and Neuropharmacology Research (32 papers) and Receptor Mechanisms and Signaling (23 papers). The work is most often cited by research in Biological Psychiatry (176 citations), Cellular and Molecular Neuroscience (1.2k citations) and Psychiatry and Mental health (758 citations). István Gyertyán has collaborated with scholars based in Hungary, United States and Sweden. Frequent co-authors include Béla Kiss, Nika Adham, I. Laszlovszky, Krisztina Gál, Katalin Sághy, Judit Laszy, András Bilkei‐Gorzó, György Domány, Zsolt Szombathelyi and György Lévay. Their work appears in journals such as Scientific Reports, Brain Research and Journal of Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.