Ismail H. Al-Abdullah
- Surgery top 5%
- Molecular Biology
- Endocrinology, Diabetes and Metabolism top 5%
- Genetics top 5%
- Pharmacology top 5%
- Co-authors
- Fouad KandeelYoko MullenKevin FerreriИван ТодоровHsun Teresa KuAkio KurodaGerd P. PfeiferTibor A. Rauch
- Topics
- Pancreatic function and diabetes (59 papers)Diabetes Management and Research (20 papers)Diabetes and associated disorders (20 papers)
- Journals
- Nature CommunicationsSHILAP Revista de lepidopterologíaPLoS ONE
- Partner nations
- United StatesIranKuwait
In The Last Decade
Ismail H. Al-Abdullah
70 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 95
- Surgery 792
- Molecular Biology 485
- Endocrinology, Diabetes and Metabolism 389
- Genetics 368
- Pharmacology 194
Countries citing papers authored by Ismail H. Al-Abdullah
This map shows the geographic impact of Ismail H. Al-Abdullah's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ismail H. Al-Abdullah with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ismail H. Al-Abdullah more than expected).
Fields of papers citing papers by Ismail H. Al-Abdullah
This network shows the impact of papers produced by Ismail H. Al-Abdullah. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ismail H. Al-Abdullah. The network helps show where Ismail H. Al-Abdullah may publish in the future.
Co-authorship network of co-authors of Ismail H. Al-Abdullah
This figure shows the co-authorship network connecting the top 25 collaborators of Ismail H. Al-Abdullah. A scholar is included among the top collaborators of Ismail H. Al-Abdullah based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ismail H. Al-Abdullah. Ismail H. Al-Abdullah is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 2 | |
| 4 | 5 | |
| 5 | 50 | |
| 6 | 1 | |
| 7 | 9 | |
| 8 | 41 | |
| 9 | 8 | |
| 10 | 9 | |
| 11 | 14 | |
| 12 | 24 | |
| 13 | 226 | |
| 14 | 3 | |
| 15 | 4 | |
| 16 | 5 | |
| 17 | 115 | |
| 18 | 20 | |
| 19 | 3 | |
| 20 | 1 |
About Ismail H. Al-Abdullah
Ismail H. Al-Abdullah is a scholar working on Transplantation, Endocrinology, Diabetes and Metabolism and Surgery, having authored 73 papers that have together received 1.3k indexed citations. Recurring topics across this work include Pancreatic function and diabetes (59 papers), Diabetes Management and Research (20 papers) and Diabetes and associated disorders (20 papers). The work is most often cited by research in Transplantation (102 citations), Endocrinology, Diabetes and Metabolism (389 citations) and Surgery (792 citations). Ismail H. Al-Abdullah has collaborated with scholars based in United States, Iran and Kuwait. Frequent co-authors include Fouad Kandeel, Yoko Mullen, Kevin Ferreri, Иван Тодоров, Hsun Teresa Ku, Akio Kuroda, Gerd P. Pfeifer, Tibor A. Rauch, M. S. A. Kumar and Jeffrey Rawson. Their work appears in journals such as Nature Communications, SHILAP Revista de lepidopterología and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.