Isabelle Duroux-Richard
- Molecular Biology top 10%
- Cancer Research top 5%
- Immunology top 10%
- Rheumatology top 10%
- Oncology
- Co-authors
- Florence ApparaillyChristian JørgensenYves‐Marie PersTula Saison‐BehmoarasG SchwabFarida DjouadChristine ChavanyC. Hélène
- Topics
- MicroRNA in disease regulation (19 papers)Cancer-related molecular mechanisms research (11 papers)Extracellular vesicles in disease (7 papers)
- Partner nations
- FranceUnited StatesGermany
In The Last Decade
Isabelle Duroux-Richard
46 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 101
- Molecular Biology 835
- Cancer Research 435
- Immunology 351
- Rheumatology 147
- Oncology 123
Countries citing papers authored by Isabelle Duroux-Richard
This map shows the geographic impact of Isabelle Duroux-Richard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Isabelle Duroux-Richard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Isabelle Duroux-Richard more than expected).
Fields of papers citing papers by Isabelle Duroux-Richard
This network shows the impact of papers produced by Isabelle Duroux-Richard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Isabelle Duroux-Richard. The network helps show where Isabelle Duroux-Richard may publish in the future.
Co-authorship network of co-authors of Isabelle Duroux-Richard
This figure shows the co-authorship network connecting the top 25 collaborators of Isabelle Duroux-Richard. A scholar is included among the top collaborators of Isabelle Duroux-Richard based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Isabelle Duroux-Richard. Isabelle Duroux-Richard is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 3 | |
| 3 | 11 | |
| 4 | 12 | |
| 5 | 10 | |
| 6 | 9 | |
| 7 | 22 | |
| 8 | 45 | |
| 9 | 38 | |
| 10 | 3 | |
| 11 | 20 | |
| 12 | 22 | |
| 13 | 35 | |
| 14 | 23 | |
| 15 | 12 | |
| 16 | 20 | |
| 17 | Antisense oligonucleotides as anticancer agents | 1 |
| 18 | 7 | |
| 19 | 28 | |
| 20 | 14 |
About Isabelle Duroux-Richard
Isabelle Duroux-Richard is a scholar working on Cancer Research, Anatomy and Immunology, having authored 49 papers that have together received 1.4k indexed citations. Recurring topics across this work include MicroRNA in disease regulation (19 papers), Cancer-related molecular mechanisms research (11 papers) and Extracellular vesicles in disease (7 papers). The work is most often cited by research in Cancer Research (435 citations), Immunology (351 citations) and Molecular Biology (835 citations). Isabelle Duroux-Richard has collaborated with scholars based in France, United States and Germany. Frequent co-authors include Florence Apparailly, Christian Jørgensen, Yves‐Marie Pers, Tula Saison‐Behmoaras, G Schwab, Farida Djouad, Christine Chavany, C. Hélène, Gérard Goubin and Justine Lebeau. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.