Isabella Spinello

587 total citations
15 papers, 492 citations indexed

About

Isabella Spinello is a scholar working on Molecular Biology, Hematology and Immunology. According to data from OpenAlex, Isabella Spinello has authored 15 papers receiving a total of 492 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 6 papers in Hematology and 6 papers in Immunology. Recurrent topics in Isabella Spinello's work include Acute Myeloid Leukemia Research (4 papers), Histone Deacetylase Inhibitors Research (4 papers) and Immune Cell Function and Interaction (4 papers). Isabella Spinello is often cited by papers focused on Acute Myeloid Leukemia Research (4 papers), Histone Deacetylase Inhibitors Research (4 papers) and Immune Cell Function and Interaction (4 papers). Isabella Spinello collaborates with scholars based in Italy. Isabella Spinello's co-authors include Catherine Labbaye, Maria Teresa Quaranta, Ugo Testa, Elvira Pelosi, Luca Pasquini, Robin Foà, Ernestina Saulle, C Peschle, Eleonora Petrucci and Eugenia Rosa Nuzzolo and has published in prestigious journals such as PLoS ONE, Nature Cell Biology and Oncogene.

In The Last Decade

Isabella Spinello

15 papers receiving 486 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Isabella Spinello Italy	12	320	248	123	102	59	15	492
Kaat Durinck Belgium	13	335 1.0×	178 0.7×	101 0.8×	98 1.0×	93 1.6×	26	548
Wouter Van Loocke Belgium	14	309 1.0×	153 0.6×	149 1.2×	103 1.0×	89 1.5×	21	518
M Zipeto United States	9	575 1.8×	167 0.7×	82 0.7×	102 1.0×	61 1.0×	13	674
Pavel Burda Czechia	10	469 1.5×	293 1.2×	113 0.9×	201 2.0×	66 1.1×	18	672
Agnieszka A. Wendorff United States	6	446 1.4×	114 0.5×	100 0.8×	179 1.8×	99 1.7×	8	642
Mingtai Chen China	12	350 1.1×	290 1.2×	69 0.6×	50 0.5×	62 1.1×	23	511
Garrett W. Rhyasen United States	8	288 0.9×	139 0.6×	205 1.7×	217 2.1×	91 1.5×	11	555
Mansi Shah United States	12	231 0.7×	108 0.4×	110 0.9×	107 1.0×	161 2.7×	24	483
Joseph R. Boyd United States	14	394 1.2×	140 0.6×	45 0.4×	61 0.6×	101 1.7×	29	529
Andrew Volk United States	11	387 1.2×	84 0.3×	114 0.9×	165 1.6×	58 1.0×	25	547

Countries citing papers authored by Isabella Spinello

Since Specialization

Citations

This map shows the geographic impact of Isabella Spinello's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Isabella Spinello with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Isabella Spinello more than expected).

Fields of papers citing papers by Isabella Spinello

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Isabella Spinello. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Isabella Spinello. The network helps show where Isabella Spinello may publish in the future.

Co-authorship network of co-authors of Isabella Spinello

This figure shows the co-authorship network connecting the top 25 collaborators of Isabella Spinello. A scholar is included among the top collaborators of Isabella Spinello based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Isabella Spinello. Isabella Spinello is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Spinello, Isabella, Catherine Labbaye, & Ernestina Saulle. (2024). Metabolic Function and Therapeutic Potential of CD147 for Hematological Malignancies: An Overview. International Journal of Molecular Sciences. 25(17). 9178–9178. 6 indexed citations

Spinello, Isabella, Ernestina Saulle, Maria Teresa Quaranta, et al.. (2024). AC-73 and Syrosingopine Inhibit SARS-CoV-2 Entry into Megakaryocytes by Targeting CD147 and MCT4. Viruses. 16(1). 82–82. 2 indexed citations

Saulle, Ernestina, Isabella Spinello, Maria Teresa Quaranta, & Catherine Labbaye. (2023). Advances in Understanding the Links between Metabolism and Autophagy in Acute Myeloid Leukemia: From Biology to Therapeutic Targeting. Cells. 12(11). 1553–1553. 7 indexed citations

Butera, Alessia, Maria Teresa Quaranta, Luca Crippa, et al.. (2022). CD147 Targeting by AC-73 Induces Autophagy and Reduces Intestinal Fibrosis Associated with TNBS Chronic Colitis. Journal of Crohn s and Colitis. 16(11). 1751–1761. 25 indexed citations

Saulle, Ernestina, Isabella Spinello, Maria Teresa Quaranta, et al.. (2021). Targeting Lactate Metabolism by Inhibiting MCT1 or MCT4 Impairs Leukemic Cell Proliferation, Induces Two Different Related Death-Pathways and Increases Chemotherapeutic Sensitivity of Acute Myeloid Leukemia Cells. Frontiers in Oncology. 10. 621458–621458. 41 indexed citations

Spinello, Isabella, Ernestina Saulle, Maria Teresa Quaranta, et al.. (2018). The small-molecule compound AC-73 targeting CD147 inhibits leukemic cell proliferation, induces autophagy and increases the chemotherapeutic sensitivity of acute myeloid leukemia cells. Haematologica. 104(5). 973–985. 41 indexed citations

Quaranta, Maria Teresa, Isabella Spinello, Gianfranco Macchia, et al.. (2016). Identification of β-Dystrobrevin as a Direct Target of miR-143: Involvement in Early Stages of Neural Differentiation. PLoS ONE. 11(5). e0156325–e0156325. 11 indexed citations

Quaranta, Maria Teresa, Eleonora Olivetta, Massimo Sanchez, et al.. (2015). miR-146a controls CXCR4 expression in a pathway that involves PLZF and can be used to inhibit HIV-1 infection of CD4+ T lymphocytes. Virology. 478. 27–38. 28 indexed citations

Spinello, Isabella, Maria Teresa Quaranta, Luca Pasquini, et al.. (2015). Human TM9SF4 Is a New Gene Down-Regulated by Hypoxia and Involved in Cell Adhesion of Leukemic Cells. PLoS ONE. 10(5). e0126968–e0126968. 17 indexed citations

10.

Spinello, Isabella, Maria Teresa Quaranta, Elvira Pelosi, et al.. (2015). Differential hypoxic regulation of the microRNA-146a/CXCR4 pathway in normal and leukemic monocytic cells: impact on response to chemotherapy. Haematologica. 100(9). 1160–1171. 23 indexed citations

11.

Saulle, Ernestina, Raffaella Guerriero, Alessia Petronelli, et al.. (2012). Autocrine Role of Angiopoietins during Megakaryocytic Differentiation. PLoS ONE. 7(7). e39796–e39796. 15 indexed citations

12.

Spinello, Isabella, Maria Teresa Quaranta, Roberta Riccioni, et al.. (2011). MicroRNA-146a and AMD3100, two ways to control CXCR4 expression in acute myeloid leukemias. Blood Cancer Journal. 1(6). e26–e26. 54 indexed citations

13.

Spinello, Isabella, Maria Teresa Quaranta, Luca Pasquini, et al.. (2009). PLZF-mediated control on c-kit expression in CD34+ cells and early erythropoiesis. Oncogene. 28(23). 2276–2288. 23 indexed citations

14.

Labbaye, Catherine, Isabella Spinello, Maria Teresa Quaranta, et al.. (2008). A three-step pathway comprising PLZF/miR-146a/CXCR4 controls megakaryopoiesis. Nature Cell Biology. 10(7). 788–801. 185 indexed citations

15.

Quaranta, Maria Teresa, Isabella Spinello, Ugo Testa, et al.. (2005). PLZF-mediated control on VLA-4 expression in normal and leukemic myeloid cells. Oncogene. 25(3). 399–408. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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