Irene Iscla

945 total citations
24 papers, 685 citations indexed

About

Irene Iscla is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Irene Iscla has authored 24 papers receiving a total of 685 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 11 papers in Physiology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Irene Iscla's work include Ion channel regulation and function (19 papers), Erythrocyte Function and Pathophysiology (11 papers) and Lipid Membrane Structure and Behavior (8 papers). Irene Iscla is often cited by papers focused on Ion channel regulation and function (19 papers), Erythrocyte Function and Pathophysiology (11 papers) and Lipid Membrane Structure and Behavior (8 papers). Irene Iscla collaborates with scholars based in United States, Argentina and China. Irene Iscla's co-authors include Paul Blount, Robin Wray, Junmei Wang, G. M. Levin, Lidia Szczupak, Shuguang Wei, Bruce A. Posner, Sanguk Kim, Samantha Miller and Yuezhou Li and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and PLoS ONE.

In The Last Decade

Irene Iscla

24 papers receiving 683 citations

Peers

Irene Iscla

PHYSI

GENET

CMN

Robin Wray United States

Wendy Bartlett United Kingdom

Natalia Levina Russia

Michelle D. Edwards United Kingdom

Paul C. Moe United States

Mariam Bayoumi Netherlands

Matthew Buechner United States

Vinesh Vijayan India

Youzhong Guo United States

Chuanqi Sun China

Robin Wray United States

Irene Iscla

504 ×0.9

261 ×0.9

PHYSI

76 ×0.8

66 ×0.7

GENET

53 ×1.0

CMN

Citations per year, relative to Irene Iscla Irene Iscla (= 1×) peers Robin Wray

Countries citing papers authored by Irene Iscla

Since Specialization

Citations

This map shows the geographic impact of Irene Iscla's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Irene Iscla with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Irene Iscla more than expected).

Fields of papers citing papers by Irene Iscla

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Irene Iscla. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Irene Iscla. The network helps show where Irene Iscla may publish in the future.

Co-authorship network of co-authors of Irene Iscla

This figure shows the co-authorship network connecting the top 25 collaborators of Irene Iscla. A scholar is included among the top collaborators of Irene Iscla based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Irene Iscla. Irene Iscla is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wray, Robin, Junmei Wang, Paul Blount, & Irene Iscla. (2022). Activation of a Bacterial Mechanosensitive Channel, MscL, Underlies the Membrane Permeabilization of Dual-Targeting Antibacterial Compounds. Antibiotics. 11(7). 970–970. 6 indexed citations

Wray, Robin, Paul Blount, Junmei Wang, & Irene Iscla. (2022). In Silico Screen Identifies a New Family of Agonists for the Bacterial Mechanosensitive Channel MscL. Antibiotics. 11(4). 433–433. 4 indexed citations

Wray, Robin, Irene Iscla, & Paul Blount. (2021). Curcumin activation of a bacterial mechanosensitive channel underlies its membrane permeability and adjuvant properties. PLoS Pathogens. 17(12). e1010198–e1010198. 13 indexed citations

Wray, Robin, Junmei Wang, Irene Iscla, & Paul Blount. (2020). Novel MscL agonists that allow multiple antibiotics cytoplasmic access activate the channel through a common binding site. PLoS ONE. 15(1). e0228153–e0228153. 14 indexed citations

Wray, Robin, Irene Iscla, Ya Gao, et al.. (2016). Dihydrostreptomycin Directly Binds to, Modulates, and Passes through the MscL Channel Pore. PLoS Biology. 14(6). e1002473–e1002473. 33 indexed citations

Iscla, Irene, et al.. (2015). Scanning MscL Channels with Targeted Post-Translational Modifications for Functional Alterations. PLoS ONE. 10(9). e0137994–e0137994. 22 indexed citations

Iscla, Irene, Robin Wray, Shuguang Wei, Bruce A. Posner, & Paul Blount. (2014). Streptomycin potency is dependent on MscL channel expression. Nature Communications. 5(1). 4891–4891. 46 indexed citations

Iscla, Irene, et al.. (2013). MscL as a Triggered Nanovalve: New Modifications to Improve Design. Biophysical Journal. 104(2). 469a–469a. 1 indexed citations

Iscla, Irene, et al.. (2013). Improving the Design of a MscL-Based Triggered Nanovalve. Biosensors. 3(1). 171–184. 29 indexed citations

10.

Zhang, Xiaozhe, Jingjing Wang, Yue Feng, et al.. (2012). Structure and molecular mechanism of an anion-selective mechanosensitive channel of small conductance. Proceedings of the National Academy of Sciences. 109(44). 18180–18185. 49 indexed citations

11.

Iscla, Irene & Paul Blount. (2012). Sensing and Responding to Membrane Tension: The Bacterial MscL Channel as a Model System. Biophysical Journal. 103(2). 169–174. 63 indexed citations

12.

Iscla, Irene, Robin Wray, & Paul Blount. (2012). The dynamics of protein-protein interactions between domains of MscL at the cytoplasmic-lipid interface. Channels. 6(4). 255–261. 12 indexed citations

13.

Iscla, Irene, Robin Wray, & Paul Blount. (2011). The oligomeric state of the truncated mechanosensitive channel of large conductance shows no variance in vivo. Protein Science. 20(9). 1638–1642. 29 indexed citations

14.

Iscla, Irene, Robin Wray, & Paul Blount. (2010). An in vivo screen reveals protein‐lipid interactions crucial for gating a mechanosensitive channel. The FASEB Journal. 25(2). 694–702. 23 indexed citations

15.

Iscla, Irene, Robin Wray, & Paul Blount. (2008). On the Structure of the N-Terminal Domain of the MscL Channel: Helical Bundle or Membrane Interface. Biophysical Journal. 95(5). 2283–2291. 60 indexed citations

16.

Iscla, Irene, G. M. Levin, Robin Wray, & Paul Blount. (2006). Disulfide Trapping the Mechanosensitive Channel MscL into a Gating-Transition State. Biophysical Journal. 92(4). 1224–1232. 28 indexed citations

17.

Edwards, Michelle D., Yuezhou Li, Sanguk Kim, et al.. (2005). Pivotal role of the glycine-rich TM3 helix in gating the MscS mechanosensitive channel. Nature Structural & Molecular Biology. 12(2). 113–119. 96 indexed citations

18.

Iscla, Irene, et al.. (2005). Selective Serotonin Reuptake Inhibitors Induce Spontaneous Interneuronal Activity in the Leech Nervous System. Journal of Neurophysiology. 93(5). 2644–2655. 15 indexed citations

19.

Iscla, Irene, G. M. Levin, Robin Wray, Robert D. Reynolds, & Paul Blount. (2004). Defining the Physical Gate of a Mechanosensitive Channel, MscL, by Engineering Metal-Binding Sites. Biophysical Journal. 87(5). 3172–3180. 41 indexed citations

20.

Iscla, Irene, Pedro David Arini, & Lidia Szczupak. (1999). Differential channeling of sensory stimuli onto a motor neuron in the leech. Journal of Comparative Physiology A. 184(2). 233–241. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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