Iqbal Dulloo

469 total citations
12 papers, 362 citations indexed

About

Iqbal Dulloo is a scholar working on Molecular Biology, Oncology and Biotechnology. According to data from OpenAlex, Iqbal Dulloo has authored 12 papers receiving a total of 362 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Oncology and 4 papers in Biotechnology. Recurrent topics in Iqbal Dulloo's work include Cancer-related Molecular Pathways (6 papers), Cancer Research and Treatments (4 papers) and RNA modifications and cancer (3 papers). Iqbal Dulloo is often cited by papers focused on Cancer-related Molecular Pathways (6 papers), Cancer Research and Treatments (4 papers) and RNA modifications and cancer (3 papers). Iqbal Dulloo collaborates with scholars based in Singapore, United Kingdom and Japan. Iqbal Dulloo's co-authors include Kanaga Sabapathy, Matthew Freeman, Gerry Melino, Kai Lin, Karen H. Vousden, Clémence Levet, Lakshmanane Boominathan, Huck‐Hui Ng, Qiang Wu and Faina Vikhanskaya and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Iqbal Dulloo

12 papers receiving 360 citations

Peers

Iqbal Dulloo

MB

ONCOL

CR

BIOTE

CB

Amir Zalcenstein Israel

Sonia Voiculescu United States

Michael D. Brewer United States

Annette Vu United States

David Michod Switzerland

Katharina Jannasch Germany

Hana Kunkel Germany

Jaebon Lee South Korea

Bon‐Hong Min South Korea

Chuanqing Wu China

Amir Zalcenstein Israel

Iqbal Dulloo

221 ×0.8

MB

195 ×1.0

ONCOL

71 ×0.9

CR

48 ×0.7

BIOTE

26 ×0.7

CB

Citations per year, relative to Iqbal Dulloo Iqbal Dulloo (= 1×) peers Amir Zalcenstein

Countries citing papers authored by Iqbal Dulloo

Since Specialization

Citations

This map shows the geographic impact of Iqbal Dulloo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Iqbal Dulloo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Iqbal Dulloo more than expected).

Fields of papers citing papers by Iqbal Dulloo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Iqbal Dulloo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Iqbal Dulloo. The network helps show where Iqbal Dulloo may publish in the future.

Co-authorship network of co-authors of Iqbal Dulloo

This figure shows the co-authorship network connecting the top 25 collaborators of Iqbal Dulloo. A scholar is included among the top collaborators of Iqbal Dulloo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Iqbal Dulloo. Iqbal Dulloo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

1.

Dulloo, Iqbal, Michael Tellier, Clémence Levet, et al.. (2024). Cleavage of the pseudoprotease iRhom2 by the signal peptidase complex reveals an ER-to-nucleus signaling pathway. Molecular Cell. 84(2). 277–292.e9. 4 indexed citations

2.

Dulloo, Iqbal, Yi‐Chun Yeh, Clémence Levet, et al.. (2022). iRhom pseudoproteases regulate ER stress-induced cell death through IP3 receptors and BCL-2. Nature Communications. 13(1). 1257–1257. 15 indexed citations

3.

Levet, Clémence, et al.. (2020). ADAM 17‐triggered TNF signalling protects the ageing Drosophila retina from lipid droplet‐mediated degeneration. The EMBO Journal. 39(17). e104415–e104415. 24 indexed citations

4.

Dulloo, Iqbal, et al.. (2019). The molecular, cellular and pathophysiological roles of iRhom pseudoproteases. Open Biology. 9(3). 190003–190003. 48 indexed citations

5.

Li, Dan, Iqbal Dulloo, & Kanaga Sabapathy. (2018). Context-dependent AMPK activation distinctly regulates TAp73 stability and transcriptional activity. Signal Transduction and Targeted Therapy. 3(1). 20–20. 4 indexed citations

6.

Dulloo, Iqbal, Soo‐Yong Tan, Liang Kee Goh, et al.. (2015). Hypoxia-inducible TAp73 supports tumorigenesis by regulating the angiogenic transcriptome. Nature Cell Biology. 17(4). 511–523. 49 indexed citations

7.

Dulloo, Iqbal, et al.. (2015). Hypoxia-induced DNp73 stabilization regulates Vegf-A expression and tumor angiogenesis similar to TAp73. Cell Cycle. 14(22). 3533–3539. 26 indexed citations

8.

Dulloo, Iqbal, et al.. (2014). Suppression of acetylpolyamine oxidase by selected AP-1 members regulates DNp73 abundance: mechanistic insights for overcoming DNp73-mediated resistance to chemotherapeutic drugs. Cell Death and Differentiation. 21(8). 1240–1249. 32 indexed citations

9.

Dulloo, Iqbal, et al.. (2010). The antiapoptotic DeltaNp73 is degraded in a c-Jun–dependent manner upon genotoxic stress through the antizyme-mediated pathway. Proceedings of the National Academy of Sciences. 107(11). 4902–4907. 51 indexed citations

10.

Vikhanskaya, Faina, Iqbal Dulloo, Qiang Wu, et al.. (2007). p73 supports cellular growth through c-Jun-dependent AP-1 transactivation. Nature Cell Biology. 9(6). 698–706. 46 indexed citations

11.

Dulloo, Iqbal & Kanaga Sabapathy. (2005). Transactivation-dependent and -independent Regulation of p73 Stability. Journal of Biological Chemistry. 280(31). 28203–28214. 18 indexed citations

12.

Lin, Kai, et al.. (2004). Multiple Stress Signals Induce p73β Accumulation. Neoplasia. 6(5). 546–557. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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