Inmaculada López-Sánchez

1.2k total citations
25 papers, 825 citations indexed

About

Inmaculada López-Sánchez is a scholar working on Molecular Biology, Cell Biology and Surgery. According to data from OpenAlex, Inmaculada López-Sánchez has authored 25 papers receiving a total of 825 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 9 papers in Cell Biology and 6 papers in Surgery. Recurrent topics in Inmaculada López-Sánchez's work include Protein Kinase Regulation and GTPase Signaling (9 papers), Pancreatic function and diabetes (6 papers) and Nuclear Structure and Function (5 papers). Inmaculada López-Sánchez is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (9 papers), Pancreatic function and diabetes (6 papers) and Nuclear Structure and Function (5 papers). Inmaculada López-Sánchez collaborates with scholars based in United States, Spain and United Kingdom. Inmaculada López-Sánchez's co-authors include Pedro A. Lazo, Alberto Valbuena, Pradipta Ghosh, Marta Sanz-García, Nicolas Aznar, Krishna Midde, Mikel Garcia‐Marcos, Francisco M. De La Vega, Marilyn G. Farquhar and Ying Dunkel and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and PLoS ONE.

In The Last Decade

Inmaculada López-Sánchez

25 papers receiving 822 citations

Peers

Inmaculada López-Sánchez
Xun Shang United States
Casey L. Moulson United States
Sarah Löw Singapore
Xun Shang United States
Inmaculada López-Sánchez
Citations per year, relative to Inmaculada López-Sánchez Inmaculada López-Sánchez (= 1×) peers Xun Shang

Countries citing papers authored by Inmaculada López-Sánchez

Since Specialization
Citations

This map shows the geographic impact of Inmaculada López-Sánchez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Inmaculada López-Sánchez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Inmaculada López-Sánchez more than expected).

Fields of papers citing papers by Inmaculada López-Sánchez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Inmaculada López-Sánchez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Inmaculada López-Sánchez. The network helps show where Inmaculada López-Sánchez may publish in the future.

Co-authorship network of co-authors of Inmaculada López-Sánchez

This figure shows the co-authorship network connecting the top 25 collaborators of Inmaculada López-Sánchez. A scholar is included among the top collaborators of Inmaculada López-Sánchez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Inmaculada López-Sánchez. Inmaculada López-Sánchez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
El‐Hafeez, Amer Ali Abd, Krishna Midde, Tony Ngo, et al.. (2023). A circuit for secretion‐coupled cellular autonomy in multicellular eukaryotic cells. Molecular Systems Biology. 19(4). e11127–e11127. 9 indexed citations
2.
Castillo, Vanessa, Gajanan D. Katkar, Inmaculada López-Sánchez, et al.. (2021). GIV/Girdin, a non-receptor modulator for Gαi/s, regulates spatiotemporal signaling during sperm capacitation and is required for male fertility. eLife. 10. 4 indexed citations
3.
Kalogriopoulos, Nicholas A., Inmaculada López-Sánchez, Chang‐Shen Lin, et al.. (2020). Receptor tyrosine kinases activate heterotrimeric G proteins via phosphorylation within the interdomain cleft of Gαi. Proceedings of the National Academy of Sciences. 117(46). 28763–28774. 22 indexed citations
4.
Rohena, C.C., Nicholas A. Kalogriopoulos, Suchismita Roy, et al.. (2020). GIV•Kindlin Interaction Is Required for Kindlin-Mediated Integrin Recognition and Activation. iScience. 23(6). 101209–101209. 8 indexed citations
5.
Aznar, Nicolas, Jason Ear, Ying Dunkel, et al.. (2018). Convergence of Wnt, growth factor, and heterotrimeric G protein signals on the guanine nucleotide exchange factor Daple. Science Signaling. 11(519). 20 indexed citations
6.
Bengoa‐Vergniory, Nora, et al.. (2016). Identification of Noncanonical Wnt Receptors Required for Wnt-3a-Induced Early Differentiation of Human Neural Stem Cells. Molecular Neurobiology. 54(8). 6213–6224. 18 indexed citations
7.
Moura, David S., et al.. (2016). Oncogenic Sox2 regulates and cooperates with VRK1 in cell cycle progression and differentiation. Scientific Reports. 6(1). 28532–28532. 12 indexed citations
8.
Gupta, Vijay, Deepali Bhandari, Anthony Leyme, et al.. (2016). GIV/Girdin activates Gαi and inhibits Gαs via the same motif. Proceedings of the National Academy of Sciences. 113(39). E5721–30. 29 indexed citations
9.
Gupta, Vijay, Krishna Midde, Vanessa Taupin, et al.. (2015). Activation of Gαi at the Golgi by GIV/Girdin Imposes Finiteness in Arf1 Signaling. Developmental Cell. 33(2). 189–203. 34 indexed citations
10.
Aznar, Nicolas, Krishna Midde, Ying Dunkel, et al.. (2015). Daple is a novel non-receptor GEF required for trimeric G protein activation in Wnt signaling. eLife. 4. e07091–e07091. 93 indexed citations
11.
López-Sánchez, Inmaculada, et al.. (2015). Focal adhesions are foci for tyrosine-based signal transduction via GIV/Girdin and G proteins. Molecular Biology of the Cell. 26(24). 4313–4324. 22 indexed citations
12.
Gary, S., Inmaculada López-Sánchez, Nicolas Aznar, et al.. (2015). Activation of G proteins by GIV-GEF is a pivot point for insulin resistance and sensitivity. Molecular Biology of the Cell. 26(23). 4209–4223. 13 indexed citations
13.
Lin, Chang‐Shen, Jason Ear, Krishna Midde, et al.. (2014). Structural basis for activation of trimeric Gi proteins by multiple growth factor receptors via GIV/Girdin. Molecular Biology of the Cell. 25(22). 3654–3671. 47 indexed citations
14.
López-Sánchez, Inmaculada, Ying Dunkel, Yash Mittal, et al.. (2014). GIV/Girdin is a central hub for profibrogenic signalling networks during liver fibrosis. Nature Communications. 5(1). 4451–4451. 76 indexed citations
15.
López-Sánchez, Inmaculada, Alberto Valbuena, Marta Vázquez-Cedeira, et al.. (2014). VRK1 interacts with p53 forming a basal complex that is activated by UV‐induced DNA damage. FEBS Letters. 588(5). 692–700. 40 indexed citations
16.
López-Sánchez, Inmaculada, Mikel Garcia‐Marcos, Yash Mittal, et al.. (2013). Protein kinase C-theta (PKCθ) phosphorylates and inhibits the guanine exchange factor, GIV/Girdin. Proceedings of the National Academy of Sciences. 110(14). 5510–5515. 33 indexed citations
17.
Valbuena, Alberto, Marta Sanz-García, Inmaculada López-Sánchez, Francisco M. De La Vega, & Pedro A. Lazo. (2011). Roles of VRK1 as a new player in the control of biological processes required for cell division. Cellular Signalling. 23(8). 1267–1272. 66 indexed citations
18.
Valbuena, Alberto, Inmaculada López-Sánchez, & Pedro A. Lazo. (2008). Human VRK1 Is an Early Response Gene and Its Loss Causes a Block in Cell Cycle Progression. PLoS ONE. 3(2). e1642–e1642. 84 indexed citations
19.
Valbuena, Alberto, Inmaculada López-Sánchez, Francisco M. De La Vega, et al.. (2007). Identification of a dominant epitope in human vaccinia-related kinase 1 (VRK1) and detection of different intracellular subpopulations. Archives of Biochemistry and Biophysics. 465(1). 219–226. 43 indexed citations
20.
Lazo, Pedro A., Francisco M. De La Vega, Ana Sevilla, et al.. (2005). Atlas of Genetics and Cytogenetics in Oncology and Haematology. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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