Ingeborg Hanbauer

5.0k total citations · 1 hit paper
97 papers, 4.1k citations indexed

About

Ingeborg Hanbauer is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Ingeborg Hanbauer has authored 97 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Cellular and Molecular Neuroscience, 42 papers in Molecular Biology and 22 papers in Physiology. Recurrent topics in Ingeborg Hanbauer's work include Neuroscience and Neuropharmacology Research (27 papers), Neuropeptides and Animal Physiology (16 papers) and Neurotransmitter Receptor Influence on Behavior (15 papers). Ingeborg Hanbauer is often cited by papers focused on Neuroscience and Neuropharmacology Research (27 papers), Neuropeptides and Animal Physiology (16 papers) and Neurotransmitter Receptor Influence on Behavior (15 papers). Ingeborg Hanbauer collaborates with scholars based in United States, Czechia and Austria. Ingeborg Hanbauer's co-authors include David A. Wink, Murali C. Krishna, Janet Gamson, James B. Mitchell, William DeGraff, Walter Lovenberg, E. Costa, Sten Hellström, Maurizio Memo and Françoise Laval and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and The Journal of Physiology.

In The Last Decade

Ingeborg Hanbauer

96 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Nitric oxide protects against cellular damage and cytotoxicity from reactive oxygen species.

1993 647 citations David A. Wink, Ingeborg Hanbauer et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ingeborg Hanbauer United States	33	1.8k	1.7k	1.4k	519	502	97	4.1k
SH Snyder United States	26	2.9k 1.6×	2.5k 1.4×	1.2k 0.9×	259 0.5×	431 0.9×	28	5.0k
C. Robin Hiley United Kingdom	36	1.4k 0.8×	1.6k 0.9×	1.5k 1.0×	326 0.6×	573 1.1×	102	4.6k
Yun‐Beom Choi United States	21	1.8k 1.0×	2.2k 1.3×	2.0k 1.4×	650 1.3×	313 0.6×	27	5.5k
Huei‐Sheng Vincent Chen United States	10	1.2k 0.7×	1.2k 0.7×	1.4k 1.0×	338 0.7×	241 0.5×	16	3.2k
James A. Ferrendelli United States	45	3.2k 1.8×	2.7k 1.6×	900 0.6×	501 1.0×	254 0.5×	137	6.3k
Kinya Kuriyama Japan	36	2.1k 1.2×	1.5k 0.9×	1.2k 0.8×	340 0.7×	168 0.3×	192	4.1k
Nobuyuki Yanagihara Japan	34	1.0k 0.6×	1.9k 1.1×	1.2k 0.9×	281 0.5×	270 0.5×	183	4.2k
Peter T.‐H. Wong Singapore	33	877 0.5×	1.2k 0.7×	932 0.6×	1.2k 2.3×	520 1.0×	107	3.8k
Antonio Giachetti Italy	45	3.6k 2.0×	3.9k 2.3×	1.5k 1.1×	164 0.3×	320 0.6×	169	6.7k
Zhuo‐Hua Pan United States	13	1.2k 0.7×	1.2k 0.7×	1.2k 0.8×	283 0.5×	224 0.4×	24	2.9k

Countries citing papers authored by Ingeborg Hanbauer

Since Specialization

Citations

This map shows the geographic impact of Ingeborg Hanbauer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ingeborg Hanbauer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ingeborg Hanbauer more than expected).

Fields of papers citing papers by Ingeborg Hanbauer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ingeborg Hanbauer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ingeborg Hanbauer. The network helps show where Ingeborg Hanbauer may publish in the future.

Co-authorship network of co-authors of Ingeborg Hanbauer

This figure shows the co-authorship network connecting the top 25 collaborators of Ingeborg Hanbauer. A scholar is included among the top collaborators of Ingeborg Hanbauer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ingeborg Hanbauer. Ingeborg Hanbauer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hanbauer, Ingeborg & Marzia Scortegagna. (2000). Molecular Markers of Oxidative Stress Vulnerability. Annals of the New York Academy of Sciences. 899(1). 182–190. 7 indexed citations

Scortegagna, Marzia, et al.. (1999). Activator protein-1 DNA binding activation by hydrogen peroxide in neuronal and astrocytic primary cultures of trisomy-16 and diploid mice. Molecular Brain Research. 73(1-2). 144–150. 13 indexed citations

Scortegagna, Marzia, Elsbeth G. Chikhale, & Ingeborg Hanbauer. (1998). Effect of lead on cytoskeletal proteins expressed in E14 mesencephalic primary cultures. Neurochemistry International. 32(4). 353–359. 7 indexed citations

Hanbauer, Ingeborg, Zygmunt Galdzicki, Stanley I. Rapoport, & Marzia Scortegagna. (1998). Evidence of Increased Oxidative Stress in Hippocampal Primary Cultures of Trisomy 16 Mouse. Restorative Neurology and Neuroscience. 12(2-3). 87–93. 1 indexed citations

Scortegagna, Marzia, Zygmunt Galdzicki, Stanley I. Rapoport, & Ingeborg Hanbauer. (1998). In cortical cultures of trisomy 16 mouse brain the upregulated metallothionein-I/II fails to respond to H2O2 exposure or glutamate receptor stimulation. Brain Research. 787(2). 292–298. 14 indexed citations

Isaacs, Krystyna R., Ingeborg Hanbauer, & David M. Jacobowitz. (1998). A Method for the Rapid Analysis of Neuronal Proportions and Neurite Morphology in Primary Cultures. Experimental Neurology. 149(2). 464–467. 11 indexed citations

Mohanakumar, Kochupurackal P., Ingeborg Hanbauer, & Chuang C. Chiueh. (1998). Neuroprotection by nitric oxide against hydroxyl radical-induced nigral neurotoxicity. Journal of Chemical Neuroanatomy. 14(3-4). 195–205. 64 indexed citations

Cook, John A., et al.. (1996). Role of antioxidants in the nitric oxide-elicited inhibition of dopamine uptake in cultured mesencephalic neurons. Insights into potential mechanisms of nitric oxide-mediated neurotoxicity. Neurochemistry International. 28(5-6). 609–617. 34 indexed citations

Hanbauer, Ingeborg, George W. Cox, & David A. Wink. (1994). Synaptic and Extrasynaptic Action of Free Radicals on Cell‐to‐Cell Signaling. Annals of the New York Academy of Sciences. 738(1). 173–180. 7 indexed citations

10.

Wink, David A., Raymond W. Nims, John F. Darbyshire, et al.. (1994). Reaction Kinetics for Nitrosation of Cysteine and Glutathione in Aerobic Nitric Oxide Solutions at Neutral pH. Insights into the Fate and Physiological Effects of Intermediates Generated in the NO/O2 Reaction. Chemical Research in Toxicology. 7(4). 519–525. 306 indexed citations

11.

Erausquin, Gabriel A. de, et al.. (1994). Persistent AMPA receptor stimulation alters [Ca2+]i homeostasis in cultures of embryonic dopaminergic neurons. Molecular Brain Research. 21(3-4). 303–311. 21 indexed citations

12.

Hanbauer, Ingeborg & Mariagrazia Grilli. (1992). Molecular mechanisms involved in transport and release of dopamine in primary cultures of mesencephalic neurons. Neurochemistry International. 20. 101–105. 3 indexed citations

13.

Erausquin, Gabriel A. de, Gary Brooker, & Ingeborg Hanbauer. (1992). K+-evoked dopamine release depends on a cytosolic Ca2+ pool regulated by N-type Ca2+ channels. Neuroscience Letters. 145(2). 121–125. 19 indexed citations

14.

Grilli, Mariagrazia, Adrian G. Wright, & Ingeborg Hanbauer. (1991). Characterization of [³H]Dopamine Uptake Sites and [³H]Cocaine Recognition Sites in Primary Cultures of Mesencephalic Neurons During In Vitro Development. Journal of Neurochemistry. 56(6). 2108–2115. 14 indexed citations

15.

Hanbauer, Ingeborg & Enrico Sanna. (1988). Presence in Brain of an Endogenous Ligand for Nitrendipine‐Binding Sites that Modulates Ca²⁺ Channel Activity. Annals of the New York Academy of Sciences. 522(1). 96–105. 5 indexed citations

16.

Sanna, Enrico, Adrian G. Wright, & Ingeborg Hanbauer. (1986). Selective localization of /sup 3/H-nitrendipine recognition sites and voltage-sensitive Ca/sup 2 +/ channels in nerve cell bodies of caudate nuclei. Fed. Proc., Fed. Am. Soc. Exp. Biol.; (United States). 3 indexed citations

17.

Hanbauer, Ingeborg, Stefano Govoni, E.A. Majane, Hongdian Yang, & E. Costa. (1982). In vivo regulation of the release of met-enkephalin-like peptides from dog adrenal medulla.. PubMed. 33. 209–15. 14 indexed citations

18.

Hanbauer, Ingeborg, Farouk Karoum, Sten Hellström, & S. Lahiri. (1981). Effects of hypoxia lasting up to one month on the catecholamine content in rat carotid body. Neuroscience. 6(1). 81–86. 72 indexed citations

19.

Hanbauer, Ingeborg. (1976). Long-term regulatory mechanisms for tyrosine hydroxylase in sympathetic ganglia and carotid body.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 15. 475–89. 4 indexed citations

20.

Hanbauer, Ingeborg. (1976). Induction of tyrosine-3-monooxygenase in superior cervical ganglia after axotomy of postganglionic nerves. Neuropharmacology. 15(8). 509–510. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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