Imen Boumaiza

546 total citations
22 papers, 434 citations indexed

About

Imen Boumaiza is a scholar working on Epidemiology, Endocrinology, Diabetes and Metabolism and Surgery. According to data from OpenAlex, Imen Boumaiza has authored 22 papers receiving a total of 434 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Epidemiology, 10 papers in Endocrinology, Diabetes and Metabolism and 6 papers in Surgery. Recurrent topics in Imen Boumaiza's work include Adipokines, Inflammation, and Metabolic Diseases (8 papers), Diabetes, Cardiovascular Risks, and Lipoproteins (7 papers) and Lipoproteins and Cardiovascular Health (5 papers). Imen Boumaiza is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (8 papers), Diabetes, Cardiovascular Risks, and Lipoproteins (7 papers) and Lipoproteins and Cardiovascular Health (5 papers). Imen Boumaiza collaborates with scholars based in Tunisia, France and United States. Imen Boumaiza's co-authors include Asma Omezzine, Ahmed Ben Abdelaziz, Nabila Ben Rejeb, Lamia Rebhi, Jihène Rejeb, Naoufel Nabli, Ali Bouslama, Amani Ouedrani, Zouhaïr Tabka and Monia Zaouali and has published in prestigious journals such as Gene, Lipids in Health and Disease and Molecular Biology Reports.

In The Last Decade

Imen Boumaiza

22 papers receiving 426 citations

Peers

Imen Boumaiza
C.R. Milner Australia
Jaspal S. Kooner United Kingdom
Pierre-Yves Martin United States
Lamia Rebhi Tunisia
Haude Cogo France
Greeshma K. Shetty United States
C.R. Milner Australia
Imen Boumaiza
Citations per year, relative to Imen Boumaiza Imen Boumaiza (= 1×) peers C.R. Milner

Countries citing papers authored by Imen Boumaiza

Since Specialization
Citations

This map shows the geographic impact of Imen Boumaiza's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Imen Boumaiza with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Imen Boumaiza more than expected).

Fields of papers citing papers by Imen Boumaiza

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Imen Boumaiza. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Imen Boumaiza. The network helps show where Imen Boumaiza may publish in the future.

Co-authorship network of co-authors of Imen Boumaiza

This figure shows the co-authorship network connecting the top 25 collaborators of Imen Boumaiza. A scholar is included among the top collaborators of Imen Boumaiza based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Imen Boumaiza. Imen Boumaiza is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Boumaiza, Imen, et al.. (2017). Resistin polymorphims, plasma resistin levels and obesity in Tunisian volunteers. Journal of Clinical Laboratory Analysis. 32(2). 18 indexed citations
2.
Rejeb, Jihène, Asma Omezzine, Lamia Rebhi, et al.. (2014). Association Between Haptoglobin 2-2 Genotype and Coronary Artery Disease and Its Severity in a Tunisian Population. Biochemical Genetics. 52(5-6). 269–282. 14 indexed citations
3.
Grissa, Oussama, et al.. (2014). Placental infiltration of inflammatory markers in gestational diabetic women. General Physiology and Biophysics. 33(2). 169–176. 62 indexed citations
4.
Boumaiza, Imen, Asma Omezzine, Jihène Rejeb, et al.. (2014). Metabolic Syndrome according to Three Definitions in Hammam-Sousse Sahloul Heart Study: A City Based Tunisian Study. 2014. 1–10. 2 indexed citations
5.
Boumaiza, Imen, et al.. (2013). Inflammation and impaired endothelium-dependant vasodilatation in non obese women with gestational diabetes mellitus: preliminary results. Lipids in Health and Disease. 12(1). 93–93. 35 indexed citations
6.
Boumaiza, Imen, Asma Omezzine, Jihène Rejeb, et al.. (2012). Association Between Four Resistin Polymorphisms, Obesity, and Metabolic Syndrome Parameters in Tunisian Volunteers. Genetic Testing and Molecular Biomarkers. 16(12). 1356–1362. 26 indexed citations
7.
Boumaiza, Imen, Asma Omezzine, Jihène Rejeb, et al.. (2012). Relationship Between Leptin G2548A and Leptin Receptor Q223R Gene Polymorphisms and Obesity and Metabolic Syndrome Risk in Tunisian Volunteers. Genetic Testing and Molecular Biomarkers. 16(7). 726–733. 96 indexed citations
8.
Omezzine, Asma, et al.. (2012). Association of four apolipoprotein B polymorphisms with lipid profile and stenosis in Tunisian coronary patients. Journal of Genetics. 91(1). 75–79. 16 indexed citations
9.
Rebhi, Lamia, Asma Omezzine, Jihène Rejeb, et al.. (2012). Six lipoprotein lipase gene polymorphisms, lipid profile and coronary stenosis in a Tunisian population. Molecular Biology Reports. 39(11). 9893–9901. 17 indexed citations
10.
Rejeb, Jihène, Asma Omezzine, Lamia Rebhi, et al.. (2012). Association of PON1 and PON2 Polymorphisms with PON1 Activity and Significant Coronary Stenosis in a Tunisian Population. Biochemical Genetics. 51(1-2). 76–91. 9 indexed citations
11.
Rejeb, Jihène, Asma Omezzine, Imen Boumaiza, et al.. (2012). Four polymorphisms of cholesteryl ester transfer protein gene and coronary stenosis in a Tunisian population. Journal of Cardiovascular Medicine. Publish Ahead of Print(9). 546–53. 8 indexed citations
12.
Rejeb, Jihène, Asma Omezzine, Imen Boumaiza, et al.. (2012). Association of three polymorphisms of scavenger receptor class BI gene (exon8, exon1, intron5) with coronary stenosis in a coronary Tunisian population. Gene. 511(2). 383–388. 14 indexed citations
13.
Boumaiza, Imen, et al.. (2012). Effects of dietetic WHO's recommendations on HDL-C level in a Tunisian obese group. Immuno-analyse & Biologie Spécialisée. 27(3). 97–103. 1 indexed citations
14.
Boumaiza, Imen, Asma Omezzine, Jihène Rejeb, et al.. (2011). Single-nucleotide polymorphisms at the adiponectin locus and risk of coronary artery disease in Tunisian coronaries. Journal of Cardiovascular Medicine. 12(9). 619–624. 12 indexed citations
15.
Boumaiza, Imen, Asma Omezzine, Jihène Rejeb, et al.. (2011). Association Between Eight Adiponectin Polymorphisms, Obesity, and Metabolic Syndrome Parameters in Tunisian Volunteers. Metabolic Syndrome and Related Disorders. 9(6). 419–426. 21 indexed citations
16.
Rejeb, Jihène, Asma Omezzine, Lamia Rebhi, et al.. (2010). Associations between common polymorphisms of adenosine triphosphate-binding cassette transporter A1 and coronary artery disease in a Tunisian population. Archives of cardiovascular diseases. 103(10). 530–537. 8 indexed citations
17.
Boumaiza, Imen, Asma Omezzine, Jihène Rejeb, et al.. (2010). Apolipoprotein B and Non-High-Density Lipoprotein Cholesterol Are Better Risk Markers for Coronary Artery Disease than Low-Density Lipoprotein Cholesterol in Hypertriglyceridemic Metabolic Syndrome Patients. Metabolic Syndrome and Related Disorders. 8(6). 515–522. 13 indexed citations
18.
Rejeb, Jihène, Asma Omezzine, Imen Boumaiza, et al.. (2010). Elevated Liver Enzymes in Metabolic Syndrome Are Associated with Coronary Stenosis in a Tunisian Population. Metabolic Syndrome and Related Disorders. 8(3). 249–254. 2 indexed citations
19.
Boumaiza, Imen, et al.. (2010). MS502 SINGLE NUCLEOTIDE POLYMORPHISMS AT THE ADIPONECTIN LOCUS AND RISK OF CORONARY ARTERY DISEASE IN A TUNISIAN POPULATION. Atherosclerosis Supplements. 11(2). 211–211. 1 indexed citations
20.
Rejeb, Jihène, Asma Omezzine, Imen Boumaiza, et al.. (2009). Metabolic Syndrome Is a Risk Factor for Coronary Artery Disease in a Tunisian Population. Metabolic Syndrome and Related Disorders. 8(2). 105–112. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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