Ida Rasmusson
- Genetics top 0.05%
- Surgery top 1%
- Molecular Biology top 10%
- Hematology top 0.5%
- Immunology top 5%
- Co-authors
- Olle RingdénKatarina Le BlancBerit SundbergMehmet UzunelCecilia GötherströmMoustapha HassanKatarina LeBlancBjörn Sundberg
- Topics
- Mesenchymal stem cell research (10 papers)Hematopoietic Stem Cell Transplantation (5 papers)Immune cells in cancer (2 papers)
- Cited by
- GeneticsHematologyRehabilitation
- Partner nations
- SwedenPanamaLuxembourg
In The Last Decade
Ida Rasmusson
11 papers receiving 4.9k citations
Hit Papers
Peers
Comparison fields: 5 of 84
- Genetics 4.2k
- Surgery 1.8k
- Molecular Biology 1.2k
- Hematology 1.1k
- Immunology 884
Countries citing papers authored by Ida Rasmusson
This map shows the geographic impact of Ida Rasmusson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ida Rasmusson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ida Rasmusson more than expected).
Fields of papers citing papers by Ida Rasmusson
This network shows the impact of papers produced by Ida Rasmusson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ida Rasmusson. The network helps show where Ida Rasmusson may publish in the future.
Co-authorship network of co-authors of Ida Rasmusson
This figure shows the co-authorship network connecting the top 25 collaborators of Ida Rasmusson. A scholar is included among the top collaborators of Ida Rasmusson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ida Rasmusson. Ida Rasmusson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 112 | |
| 2 | 222 | |
| 3 | 111 | |
| 4 | Mesenchymal Stem Cells for Treatment of Therapy-Resistant Graft-versus-Host Diseasebreakdown → | 893 |
| 5 | 77 | |
| 6 | 291 | |
| 7 | 7 | |
| 8 | 415 | |
| 9 | 264 | |
| 10 | Treatment of severe acute graft-versus-host disease with third party haploidentical mesenchymal stem cellsbreakdown → | 2129 |
| 11 | Mesenchymal stem cells inhibit the formation of cytotoxic T lymphocytes, but not activated cytotoxic T lymphocytes or natural killer cellsbreakdown → | 519 |
About Ida Rasmusson
Ida Rasmusson is a scholar working on Genetics, Hematology and Transplantation, having authored 11 papers that have together received 5.0k indexed citations. Recurring topics across this work include Mesenchymal stem cell research (10 papers), Hematopoietic Stem Cell Transplantation (5 papers) and Immune cells in cancer (2 papers). The work is most often cited by research in Genetics (4.2k citations), Hematology (1.1k citations) and Rehabilitation (339 citations). Ida Rasmusson has collaborated with scholars based in Sweden, Panama and Luxembourg. Frequent co-authors include Olle Ringdén, Katarina Le Blanc, Berit Sundberg, Mehmet Uzunel, Cecilia Götherström, Moustapha Hassan, Katarina LeBlanc, Björn Sundberg, J Aschan and Hanns–Ulrich Marschall. Their work appears in journals such as The Lancet, Diabetes and Experimental Cell Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.