Ian Dainty
- Physiology top 0.5%
- Molecular Biology
- Physiology top 10%
- Cellular and Molecular Neuroscience top 10%
- Cardiology and Cardiovascular Medicine
- Co-authors
- Paul LeffStephen E. O'ConnorIain G. DougallTania E. WebbDuncan J. HendersonG M SmithGraeme WilkinsonP. Leff
- Topics
- Asthma and respiratory diseases (6 papers)Adenosine and Purinergic Signaling (5 papers)Receptor Mechanisms and Signaling (5 papers)
- Journals
- Biochemical and Biophysical Research CommunicationsJournal of Pharmacology and Experimental TherapeuticsNature Structural & Molecular Biology
- Partner nations
- United KingdomSwedenUnited States
In The Last Decade
Ian Dainty
19 papers receiving 851 citations
Peers
Comparison fields: 5 of 83
- Physiology 455
- Molecular Biology 409
- Physiology 259
- Cellular and Molecular Neuroscience 180
- Cardiology and Cardiovascular Medicine 127
Countries citing papers authored by Ian Dainty
This map shows the geographic impact of Ian Dainty's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ian Dainty with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ian Dainty more than expected).
Fields of papers citing papers by Ian Dainty
This network shows the impact of papers produced by Ian Dainty. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ian Dainty. The network helps show where Ian Dainty may publish in the future.
Co-authorship network of co-authors of Ian Dainty
This figure shows the co-authorship network connecting the top 25 collaborators of Ian Dainty. A scholar is included among the top collaborators of Ian Dainty based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ian Dainty. Ian Dainty is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 20 | |
| 2 | 7 | |
| 3 | 3 | |
| 4 | 22 | |
| 5 | 12 | |
| 6 | 67 | |
| 7 | 1 | |
| 8 | 12 | |
| 9 | 20 | |
| 10 | 28 | |
| 11 | 89 | |
| 12 | 95 | |
| 13 | 46 | |
| 14 | 317 | |
| 15 | 25 | |
| 16 | 48 | |
| 17 | 13 | |
| 18 | 7 | |
| 19 | 47 |
About Ian Dainty
Ian Dainty is a scholar working on Physiology, Physiology and Animal Science and Zoology, having authored 19 papers that have together received 879 indexed citations. Recurring topics across this work include Asthma and respiratory diseases (6 papers), Adenosine and Purinergic Signaling (5 papers) and Receptor Mechanisms and Signaling (5 papers). The work is most often cited by research in Physiology (455 citations), Endocrine and Autonomic Systems (116 citations) and Physiology (259 citations). Ian Dainty has collaborated with scholars based in United Kingdom, Sweden and United States. Frequent co-authors include Paul Leff, Stephen E. O'Connor, Iain G. Dougall, Tania E. Webb, Duncan J. Henderson, G M Smith, Graeme Wilkinson, P. Leff, Michael Spedding and J.C. McGrath. Their work appears in journals such as Biochemical and Biophysical Research Communications, Journal of Pharmacology and Experimental Therapeutics and Nature Structural & Molecular Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.