Hui-Kuan Lin

2.1k total citations
10 papers, 1.6k citations indexed

About

Hui-Kuan Lin is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Hui-Kuan Lin has authored 10 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Oncology and 1 paper in Pulmonary and Respiratory Medicine. Recurrent topics in Hui-Kuan Lin's work include Ubiquitin and proteasome pathways (6 papers), Cancer-related Molecular Pathways (4 papers) and Cancer-related gene regulation (2 papers). Hui-Kuan Lin is often cited by papers focused on Ubiquitin and proteasome pathways (6 papers), Cancer-related Molecular Pathways (4 papers) and Cancer-related gene regulation (2 papers). Hui-Kuan Lin collaborates with scholars based in United States, Taiwan and Japan. Hui-Kuan Lin's co-authors include Chia‐Hsin Chan, Pier Paolo Pandolfi, Keiichi I. Nakayama, Guocan Wang, Zhenbang Chen, Julie Teruya‐Feldstein, Jing Wang, Juan Wu, Caterina Nardella and Carlos Cordon‐Cardo and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Hui-Kuan Lin

10 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hui-Kuan Lin United States 10 1.3k 456 290 274 192 10 1.6k
Jonathan A. Fletcher United States 8 1.2k 0.9× 517 1.1× 435 1.5× 327 1.2× 162 0.8× 12 2.0k
Matthias Drosten Spain 22 1.4k 1.1× 777 1.7× 261 0.9× 291 1.1× 254 1.3× 43 2.0k
Carlotta Costa United States 19 1.2k 0.9× 669 1.5× 430 1.5× 188 0.7× 253 1.3× 28 1.9k
Kiran Mahajan United States 24 1.5k 1.1× 495 1.1× 384 1.3× 273 1.0× 197 1.0× 38 1.9k
Bárbara Sousa Portugal 19 841 0.7× 550 1.2× 185 0.6× 493 1.8× 133 0.7× 43 1.4k
Xiaoqi Liu United States 25 1.2k 0.9× 469 1.0× 436 1.5× 414 1.5× 301 1.6× 77 1.6k
Alina Molchadsky Israel 21 1.3k 1.0× 797 1.7× 130 0.4× 518 1.9× 107 0.6× 28 1.8k
Rachel Bikoff United States 5 865 0.7× 247 0.5× 315 1.1× 334 1.2× 70 0.4× 7 1.3k
Damien Gerald United States 10 973 0.8× 376 0.8× 102 0.4× 553 2.0× 130 0.7× 16 1.5k
Yoon Jong Choi United States 11 1.3k 1.0× 813 1.8× 449 1.5× 240 0.9× 286 1.5× 19 1.9k

Countries citing papers authored by Hui-Kuan Lin

Since Specialization
Citations

This map shows the geographic impact of Hui-Kuan Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hui-Kuan Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hui-Kuan Lin more than expected).

Fields of papers citing papers by Hui-Kuan Lin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hui-Kuan Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hui-Kuan Lin. The network helps show where Hui-Kuan Lin may publish in the future.

Co-authorship network of co-authors of Hui-Kuan Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Hui-Kuan Lin. A scholar is included among the top collaborators of Hui-Kuan Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hui-Kuan Lin. Hui-Kuan Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Wagner, Klaus W., Hunain Alam, Shilpa S. Dhar, et al.. (2013). KDM2A promotes lung tumorigenesis by epigenetically enhancing ERK1/2 signaling. Journal of Clinical Investigation. 123(12). 5231–5246. 149 indexed citations
2.
Wang, Guocan, Yuan Gao, Li Li, et al.. (2012). K63-Linked Ubiquitination in Kinase Activation and Cancer. SHILAP Revista de lepidopterología. 2. 5–5. 77 indexed citations
3.
Lamothe, Betty, Jing Wang, Alejandro D. Campos, et al.. (2012). Deletion of TAK1 in the Myeloid Lineage Results in the Spontaneous Development of Myelomonocytic Leukemia in Mice. PLoS ONE. 7(12). e51228–e51228. 29 indexed citations
4.
Chan, Chia‐Hsin, Chien‐Feng Li, Jing Wang, et al.. (2010). Deciphering the transcriptional complex critical for RhoA gene expression and cancer metastasis. Nature Cell Biology. 12(5). 457–467. 178 indexed citations
5.
Lin, Hui-Kuan, Zhenbang Chen, Guocan Wang, et al.. (2010). Skp2 targeting suppresses tumorigenesis by Arf-p53-independent cellular senescence. Nature. 464(7287). 374–379. 316 indexed citations
6.
Wu, Juan, et al.. (2010). Regulation of Akt signaling activation by ubiquitination. Cell Cycle. 9(3). 486–497. 119 indexed citations
7.
Lin, Hui-Kuan, Guocan Wang, Zhenbang Chen, et al.. (2009). Phosphorylation-dependent regulation of cytosolic localization and oncogenic function of Skp2 by Akt/PKB. Nature Cell Biology. 11(4). 420–432. 203 indexed citations
8.
Wang, Liang, Hui-Kuan Lin, Yueh‐Chiang Hu, et al.. (2004). Suppression of Androgen Receptor-mediated Transactivation and Cell Growth by the Glycogen Synthase Kinase 3β in Prostate Cells. Journal of Biological Chemistry. 279(31). 32444–32452. 77 indexed citations
9.
Yang, Lin, Hui-Kuan Lin, Saleh Altuwaijri, et al.. (2003). APPL Suppresses Androgen Receptor Transactivation via Potentiating Akt Activity. Journal of Biological Chemistry. 278(19). 16820–16827. 51 indexed citations
10.
Lin, Hui-Kuan. (2002). Phosphorylation-dependent ubiquitylation and degradation of androgen receptor by Akt require Mdm2 E3 ligase. The EMBO Journal. 21(15). 4037–4048. 381 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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