Hui Gai
Impact in
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- Pluripotent Stem Cells Research
- CRISPR and Genetic Engineering
- RNA Research and Splicing
- RNA and protein synthesis mechanisms
- RNA modifications and cancer
- Cell Biology top 10%
- Biotin and Related Studies
Papers in
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- Pluripotent Stem Cells Research 6
- CRISPR and Genetic Engineering 3
- Renal and related cancers 2
- Ion channel regulation and function 1
- Surgery 2
- Tissue Engineering and Regenerative Medicine 2
- Co-authors
- Hui Z. Sheng (4 shared papers)Julien G. Roth (2 shared papers)Theo D. Palmer (2 shared papers)Zhen Fang (3 shared papers)Louis M. Fink (2 shared papers)Brian Zarnegar (1 shared paper)Yupo Ma (2 shared papers)Muthukumar Ramanathan (1 shared paper)
- Journals
- Cell Research (2 papers)eLife (2 papers)Nature Methods (1 paper)Experimental Cell Research (1 paper)Differentiation (1 paper)
- Partner nations
- United StatesChina
In The Last Decade
Hui Gai
9 papers receiving 691 citations
Peers
Comparison fields: 5 of 63
- Molecular Biology 524
- Cell Biology 110
- Cellular and Molecular Neuroscience 74
- Genetics 38
- Surgery 132
Countries citing papers authored by Hui Gai
This map shows the geographic impact of Hui Gai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hui Gai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hui Gai more than expected).
Fields of papers citing papers by Hui Gai
This network shows the impact of papers produced by Hui Gai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hui Gai. The network helps show where Hui Gai may publish in the future.
Co-authors
The 25 scholars most cited alongside Hui Gai, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 233 | |
| 2 | 2016 | 103 | |
| 3 | 2009 | 97 | |
| 4 | 2006 | 77 | |
| 5 | 2005 | 57 | |
| 6 | 2006 | 57 | |
| 7 | 2010 | 39 | |
| 8 | 2020 | 27 | |
| 9 | 2005 | 13 |
About Hui Gai
Hui Gai is a scholar working on Molecular Biology, Surgery, Cellular and Molecular Neuroscience, Public Health, Environmental and Occupational Health and Cell Biology, having authored 9 papers that have together received 703 indexed citations. Recurring topics across this work include Pluripotent Stem Cells Research (6 papers), CRISPR and Genetic Engineering (3 papers), Renal and related cancers (2 papers), Tissue Engineering and Regenerative Medicine (2 papers), Neuroscience and Neural Engineering (1 paper), Genetics and Neurodevelopmental Disorders (1 paper), Epilepsy research and treatment (1 paper) and Ion channel regulation and function (1 paper). The work is most often cited by research in Molecular Biology (524 citations), Cell Biology (110 citations), Cellular and Molecular Neuroscience (74 citations), Genetics (38 citations) and Surgery (132 citations). Hui Gai has collaborated with scholars based in United States and China. Frequent co-authors include Hui Z. Sheng, Julien G. Roth, Theo D. Palmer, Zhen Fang, Louis M. Fink, Brian Zarnegar, Yupo Ma, Muthukumar Ramanathan, Jan E. Carette and David C. Ward. Their work appears in journals such as Cell Research, eLife, Nature Methods, Experimental Cell Research and Differentiation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.