Huey‐Nan Wu

1.7k total citations
38 papers, 1.4k citations indexed

About

Huey‐Nan Wu is a scholar working on Molecular Biology, Hepatology and Infectious Diseases. According to data from OpenAlex, Huey‐Nan Wu has authored 38 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 15 papers in Hepatology and 12 papers in Infectious Diseases. Recurrent topics in Huey‐Nan Wu's work include RNA and protein synthesis mechanisms (16 papers), Hepatitis C virus research (15 papers) and Mosquito-borne diseases and control (11 papers). Huey‐Nan Wu is often cited by papers focused on RNA and protein synthesis mechanisms (16 papers), Hepatitis C virus research (15 papers) and Mosquito-borne diseases and control (11 papers). Huey‐Nan Wu collaborates with scholars based in Taiwan, United States and France. Huey‐Nan Wu's co-authors include M M Lai, Michael M. C. Lai, Shinji Makino, Yicheng Lin, Ming‐Fu Chang, Olke C. Uhlenbeck, Dorothy Beckett, Chwan‐Deng Hsiao, Cheng-Yang Huang and Chin-Kai Tseng and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Huey‐Nan Wu

38 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Huey‐Nan Wu Taiwan 22 713 378 362 333 325 38 1.4k
Eric Ferrari United States 13 470 0.7× 864 2.3× 492 1.4× 82 0.2× 581 1.8× 17 1.4k
Kevin Whitby United Kingdom 14 348 0.5× 423 1.1× 775 2.1× 484 1.5× 529 1.6× 20 1.7k
Rainer Ziermann United States 20 341 0.5× 221 0.6× 430 1.2× 53 0.2× 239 0.7× 31 1.0k
Peter Skewes-Cox United States 14 530 0.7× 65 0.2× 343 0.9× 89 0.3× 346 1.1× 20 1.4k
Isabelle Imbert France 19 869 1.2× 215 0.6× 2.1k 5.8× 77 0.2× 304 0.9× 32 3.0k
B. Wolanski United States 15 478 0.7× 304 0.8× 402 1.1× 42 0.1× 399 1.2× 25 1.1k
Musaddeq Hussain United States 14 619 0.9× 279 0.7× 151 0.4× 57 0.2× 283 0.9× 25 1.2k
Eliana G. Acosta Germany 17 259 0.4× 140 0.4× 698 1.9× 1.0k 3.0× 287 0.9× 18 1.5k
Nagraj Mani United States 18 534 0.7× 199 0.5× 588 1.6× 42 0.1× 309 1.0× 36 1.3k
Francesc Puig‐Basagoiti Spain 18 168 0.2× 254 0.7× 371 1.0× 480 1.4× 277 0.9× 22 1.1k

Countries citing papers authored by Huey‐Nan Wu

Since Specialization
Citations

This map shows the geographic impact of Huey‐Nan Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Huey‐Nan Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Huey‐Nan Wu more than expected).

Fields of papers citing papers by Huey‐Nan Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Huey‐Nan Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Huey‐Nan Wu. The network helps show where Huey‐Nan Wu may publish in the future.

Co-authorship network of co-authors of Huey‐Nan Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Huey‐Nan Wu. A scholar is included among the top collaborators of Huey‐Nan Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Huey‐Nan Wu. Huey‐Nan Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Lu, Jianqiang, et al.. (2017). Epigenetic Silencing of MIR-375 Promotes Catilage Degradation by Targeting JAK2/STAT3 Signaling Pathway in Knee Osteoarthritis. Osteoarthritis and Cartilage. 25. S291–S291. 5 indexed citations
3.
Lee, Jin‐Ching, Chin-Kai Tseng, Yu‐Hsuan Wu, et al.. (2015). Characterization of the activity of 2′-C-methylcytidine against dengue virus replication. Antiviral Research. 116. 1–9. 62 indexed citations
4.
Tseng, Chih-Hua, Chih-Hua Tseng, Chun‐Kuang Lin, et al.. (2014). Synthesis, antiproliferative and anti-dengue virus evaluations of 2-aroyl-3-arylquinoline derivatives. European Journal of Medicinal Chemistry. 79. 66–76. 28 indexed citations
5.
6.
Wu, Szu‐Huei, Shiow‐Ju Lee, Weir‐Torn Jiaang, et al.. (2013). A Novel Dengue Virus Inhibitor, BP13944, Discovered by High-Throughput Screening with Dengue Virus Replicon Cells Selects for Resistance in the Viral NS2B/NS3 Protease. Antimicrobial Agents and Chemotherapy. 58(1). 110–119. 72 indexed citations
7.
Wu, Huey‐Nan, et al.. (2010). HCV NS3 protein helicase domain assists RNA structure conversion. FEBS Letters. 584(11). 2356–2362. 7 indexed citations
8.
Yeh, Trai‐Ming, et al.. (2008). Dengue virus infection induces passive release of high mobility group box 1 protein by epithelial cells. Journal of Infection. 56(2). 143–150. 40 indexed citations
9.
Huang, Cheng-Yang, Che-hsiung Hsu, Yuh‐Ju Sun, Huey‐Nan Wu, & Chwan‐Deng Hsiao. (2006). Complexed crystal structure of replication restart primosome protein PriB reveals a novel single-stranded DNA-binding mode. Nucleic Acids Research. 34(14). 3878–3886. 70 indexed citations
10.
Khanna, May, Huey‐Nan Wu, Carina Johansson, Michèle Caizergues‐Ferrer, & Juli Feigon. (2005). Structural study of the H/ACA snoRNP components Nop10p and the 3′ hairpin of U65 snoRNA. RNA. 12(1). 40–52. 33 indexed citations
11.
Wu, Huey‐Nan, et al.. (2004). Characterization and application of the selective strand annealing activity of the N terminal domain of hepatitis delta antigen. FEBS Letters. 578(3). 345–350. 5 indexed citations
12.
Wu, Huey‐Nan, et al.. (1998). Identification and Characterization of the RNA Chaperone Activity of Hepatitis Delta Antigen Peptides. Journal of Biological Chemistry. 273(41). 26455–26461. 58 indexed citations
13.
Cheng, Jya‐Wei, et al.. (1998). Local helix content and RNA-binding activity of the N-terminal leucine-repeat region of hepatitis delta antigen. Journal of Biomolecular NMR. 12(1). 183–188. 7 indexed citations
14.
Ping, Yueh‐Hsin, et al.. (1997). An AU at the first base pair of helix 3 elevates the catalytic activity of hepatitis delta virus ribozymes. FEBS Letters. 413(2). 299–303. 5 indexed citations
15.
Lee, Bao‐Shiang, Huey‐Nan Wu, & Tai-huang Huang. (1993). The catalytic domain of human hepatitis delta virus RNA. FEBS Letters. 324(3). 296–300. 5 indexed citations
16.
Wu, Huey‐Nan, et al.. (1993). Mutagenesis analysis of a hepatitis delta virus genomic ribozyme. Nucleic Acids Research. 21(18). 4193–4199. 31 indexed citations
17.
Wu, Huey‐Nan, et al.. (1992). Mutagenesis analysis of the self-cleavage domain of hepatitis delta virus antigenomic RNA. Nucleic Acids Research. 20(22). 5937–5941. 33 indexed citations
18.
Wu, Huey‐Nan, et al.. (1992). Sequence and structure of the catalytic RNA of hepatitis delta virus genomic RNA. Journal of Molecular Biology. 223(1). 233–245. 66 indexed citations
19.
Wu, Huey‐Nan & Michael M. C. Lai. (1989). Reversible Cleavage and Ligation of Hepatitis Delta Virus RNA. Science. 243(4891). 652–654. 70 indexed citations
20.
Beckett, Dorothy, Huey‐Nan Wu, & Olke C. Uhlenbeck. (1988). Roles of operator and non-operator RNA sequences in bacteriophage R17 capsid assembly. Journal of Molecular Biology. 204(4). 939–947. 82 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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