Hitoshi Sawa

3.1k total citations
50 papers, 2.5k citations indexed

About

Hitoshi Sawa is a scholar working on Aging, Molecular Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Hitoshi Sawa has authored 50 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Aging, 38 papers in Molecular Biology and 17 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Hitoshi Sawa's work include Genetics, Aging, and Longevity in Model Organisms (39 papers), Reproductive Biology and Fertility (17 papers) and RNA Research and Splicing (9 papers). Hitoshi Sawa is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (39 papers), Reproductive Biology and Fertility (17 papers) and RNA Research and Splicing (9 papers). Hitoshi Sawa collaborates with scholars based in Japan, United States and Canada. Hitoshi Sawa's co-authors include Kota Mizumoto, Hideyuki Okano, Hisako Takeshita, John Abelson, Y. Shimura, Leslie Lobel, H. Robert Horvitz, Hiroko Kouike, Kenji Sugioka and Akinori Yoda and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Hitoshi Sawa

49 papers receiving 2.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hitoshi Sawa Japan 28 1.9k 1.2k 482 378 268 50 2.5k
James R. Priess United States 8 1.3k 0.7× 1.4k 1.2× 415 0.9× 391 1.0× 273 1.0× 8 2.0k
Jeffrey S. Simske United States 13 729 0.4× 894 0.8× 222 0.5× 319 0.8× 324 1.2× 20 1.4k
Kiyoji Nishiwaki Japan 23 1.0k 0.5× 804 0.7× 99 0.2× 558 1.5× 222 0.8× 48 1.9k
Anna P. Newman United States 18 1.0k 0.5× 561 0.5× 232 0.5× 728 1.9× 189 0.7× 30 1.6k
Olaf Bossinger Germany 22 1.1k 0.6× 892 0.8× 105 0.2× 685 1.8× 199 0.7× 36 1.8k
Maria Gallegos United States 9 1.1k 0.6× 691 0.6× 278 0.6× 100 0.3× 169 0.6× 12 1.5k
Marco C. Betist Netherlands 16 892 0.5× 410 0.3× 83 0.2× 439 1.2× 107 0.4× 22 1.2k
Maria Doitsidou United States 12 735 0.4× 351 0.3× 52 0.1× 216 0.6× 102 0.4× 17 1.3k
Bruce Wightman United States 15 3.2k 1.7× 593 0.5× 119 0.2× 85 0.2× 139 0.5× 23 4.0k
Rongwen Xi China 24 1.5k 0.8× 199 0.2× 120 0.2× 322 0.9× 33 0.1× 54 2.4k

Countries citing papers authored by Hitoshi Sawa

Since Specialization
Citations

This map shows the geographic impact of Hitoshi Sawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hitoshi Sawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hitoshi Sawa more than expected).

Fields of papers citing papers by Hitoshi Sawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hitoshi Sawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hitoshi Sawa. The network helps show where Hitoshi Sawa may publish in the future.

Co-authorship network of co-authors of Hitoshi Sawa

This figure shows the co-authorship network connecting the top 25 collaborators of Hitoshi Sawa. A scholar is included among the top collaborators of Hitoshi Sawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hitoshi Sawa. Hitoshi Sawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Sugioka, Kenji, Kota Mizumoto, Bruce Bowerman, et al.. (2018). Tumor suppressor APC is an attenuator of spindle-pulling forces during C. elegans asymmetric cell division. Proceedings of the National Academy of Sciences. 115(5). E954–E963. 19 indexed citations
3.
Arata, Yukinobu, Hiroaki Takagi, Yasushi Sako, & Hitoshi Sawa. (2015). Power law relationship between cell cycle duration and cell volume in the early embryonic development of Caenorhabditis elegans. Frontiers in Physiology. 5. 529–529. 40 indexed citations
4.
Sawa, Hitoshi & Hendrik C. Korswagen. (2013). Wnt signaling in C. elegans. WormBook. 1–30. 74 indexed citations
5.
Sugioka, Kenji & Hitoshi Sawa. (2012). Formation and functions of asymmetric microtubule organization in polarized cells. Current Opinion in Cell Biology. 24(4). 517–525. 27 indexed citations
6.
Sawa, Hitoshi. (2012). Control of Cell Polarity and Asymmetric Division in C. elegans. Current topics in developmental biology. 101. 55–76. 45 indexed citations
7.
Shibata, Yukimasa, Masahiro Uchida, Hisako Takeshita, Kiyoji Nishiwaki, & Hitoshi Sawa. (2011). Multiple functions of PBRM-1/Polybromo- and LET-526/Osa-containing chromatin remodeling complexes in C. elegans development. Developmental Biology. 361(2). 349–357. 22 indexed citations
8.
Yamamoto, Yuko, Hisako Takeshita, & Hitoshi Sawa. (2011). Multiple Wnts Redundantly Control Polarity Orientation in Caenorhabditis elegans Epithelial Stem Cells. PLoS Genetics. 7(10). e1002308–e1002308. 42 indexed citations
9.
Sugioka, Kenji & Hitoshi Sawa. (2010). Regulation of asymmetric positioning of nuclei by Wnt and Src signaling and its roles in POP‐1/TCF nuclear asymmetry in Caenorhabditis elegans. Genes to Cells. 15(4). 397–407. 12 indexed citations
10.
Kanamori, Takahiro, Takao Inoue, Taro Sakamoto, et al.. (2008). β-Catenin asymmetry is regulated by PLA1 and retrograde traffic in C. elegans stem cell divisions. The EMBO Journal. 27(12). 1647–1657. 36 indexed citations
11.
Mizumoto, Kota & Hitoshi Sawa. (2007). Two βs or not two βs: regulation of asymmetric division by β-catenin. Trends in Cell Biology. 17(10). 465–473. 101 indexed citations
12.
Fujita, Masaki, Hisako Takeshita, & Hitoshi Sawa. (2007). Cyclin E and CDK2 Repress the Terminal Differentiation of Quiescent Cells after Asymmetric Division in C. elegans. PLoS ONE. 2(5). e407–e407. 21 indexed citations
13.
Kidd, Ambrose R., et al.. (2005). A β-Catenin Identified by Functional Rather Than Sequence Criteria and Its Role in Wnt/MAPK Signaling. Cell. 121(5). 761–772. 114 indexed citations
14.
Takeshita, Hisako & Hitoshi Sawa. (2005). Asymmetric cortical and nuclear localizations of WRM-1/β-catenin during asymmetric cell division in C. elegans. Genes & Development. 19(15). 1743–1748. 75 indexed citations
15.
Zhao, Xiaojun, Hitoshi Sawa, & Michael A. Herman. (2003). Tcl-2 encodes a novel protein that acts synergistically with Wnt signaling pathways in C. elegans. Developmental Biology. 256(2). 276–289. 8 indexed citations
16.
Sawa, Hitoshi, Hiroko Kouike, & Hideyuki Okano. (2000). Components of the SWI/SNF Complex Are Required for Asymmetric Cell Division in C. elegans. Molecular Cell. 6(3). 617–624. 85 indexed citations
17.
Yoda, Akinori, Hitoshi Sawa, & Hideyuki Okano. (2000). MSI‐1, a neural RNA‐binding protein, is involved in male mating behaviour in Caenorhabditis elegans. Genes to Cells. 5(11). 885–895. 37 indexed citations
18.
Rocheleau, Christian E., Jun Yasuda, Tae Ho Shin, et al.. (1999). WRM-1 Activates the LIT-1 Protein Kinase to Transduce Anterior/Posterior Polarity Signals in C. elegans. Cell. 97(6). 717–726. 230 indexed citations
19.
Inoue, Shunsuke, Hiroshi Sakamoto, Hitoshi Sawa, & Yoshiro Shimura. (1992). Nucleotide sequence of a fission yeast gene encoding the DEAH-box RNA helicase. Nucleic Acids Research. 20(21). 5841–5841. 2 indexed citations
20.
Sawa, Hitoshi & Yoshiro Shimura. (1991). Alterations of RNase H sensitivity of the 3′ splice site region during thein vitrosplicing reaction. Nucleic Acids Research. 19(14). 3953–3958. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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