Hiroko Ishii

530 total citations
19 papers, 355 citations indexed

About

Hiroko Ishii is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Hiroko Ishii has authored 19 papers receiving a total of 355 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Oncology and 4 papers in Cancer Research. Recurrent topics in Hiroko Ishii's work include Cancer Cells and Metastasis (5 papers), Muscle Physiology and Disorders (4 papers) and Tissue Engineering and Regenerative Medicine (2 papers). Hiroko Ishii is often cited by papers focused on Cancer Cells and Metastasis (5 papers), Muscle Physiology and Disorders (4 papers) and Tissue Engineering and Regenerative Medicine (2 papers). Hiroko Ishii collaborates with scholars based in Japan, Egypt and United States. Hiroko Ishii's co-authors include Yukiko Hayashi, Kiichi Arahata, Man Woo, Ikuya Nonaka, Ikuya Nonaka, Masaharu Seno, Ritsuko Koga, Shin’ichi Takeda, Toshifumi Tsukahara and Hideo Sugita and has published in prestigious journals such as International Journal of Molecular Sciences, Journal of Antimicrobial Chemotherapy and Journal of the Neurological Sciences.

In The Last Decade

Hiroko Ishii

19 papers receiving 348 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hiroko Ishii Japan 12 271 66 57 44 35 19 355
Yoshihiro Taniguchi Japan 9 155 0.6× 37 0.6× 37 0.6× 42 1.0× 12 0.3× 15 353
Maria Julia Diacovo United States 6 106 0.4× 37 0.6× 31 0.5× 33 0.8× 31 0.9× 11 349
Alice Witsell United States 9 163 0.6× 70 1.1× 55 1.0× 13 0.3× 14 0.4× 13 491
Bryan A. Haines United States 8 232 0.9× 84 1.3× 78 1.4× 29 0.7× 10 0.3× 8 468
Inna Uliyakina Portugal 12 334 1.2× 65 1.0× 57 1.0× 59 1.3× 7 0.2× 13 625
Lisa W. Ru United States 9 157 0.6× 45 0.7× 30 0.5× 24 0.5× 19 0.5× 10 341
Trisha M. Finlay Canada 9 230 0.8× 44 0.7× 41 0.7× 33 0.8× 18 0.5× 11 403
Andrew Hibbert United Kingdom 8 178 0.7× 25 0.4× 59 1.0× 71 1.6× 13 0.4× 12 401
Sumit Ghosh United States 11 191 0.7× 122 1.8× 36 0.6× 18 0.4× 50 1.4× 24 464
Rosângela P. da Silva United Kingdom 7 138 0.5× 38 0.6× 34 0.6× 22 0.5× 16 0.5× 8 410

Countries citing papers authored by Hiroko Ishii

Since Specialization
Citations

This map shows the geographic impact of Hiroko Ishii's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroko Ishii with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroko Ishii more than expected).

Fields of papers citing papers by Hiroko Ishii

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroko Ishii. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroko Ishii. The network helps show where Hiroko Ishii may publish in the future.

Co-authorship network of co-authors of Hiroko Ishii

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroko Ishii. A scholar is included among the top collaborators of Hiroko Ishii based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroko Ishii. Hiroko Ishii is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Afify, Said M., Ghmkin Hassan, Hiroko Ishii, et al.. (2022). Functional and Molecular Characters of Cancer Stem Cells Through Development to Establishment. Advances in experimental medicine and biology. 1393. 83–101. 3 indexed citations
2.
Ishii, Hiroko, et al.. (2021). Isolation and characterization of cancer stem cells derived from human glioblastoma.. American Journal of Cancer Research. 11(2). 441–457. 14 indexed citations
3.
Ishii, Hiroko, Said M. Afify, Ghmkin Hassan, David S. Salomon, & Masaharu Seno. (2021). Cripto-1 as a Potential Target of Cancer Stem Cells for Immunotherapy. Cancers. 13(10). 2491–2491. 13 indexed citations
4.
Ishii, Hiroko, et al.. (2021). A Novel Artificially Humanized Anti-Cripto-1 Antibody Suppressing Cancer Cell Growth. International Journal of Molecular Sciences. 22(4). 1709–1709. 10 indexed citations
5.
Hassan, Ghmkin, Said M. Afify, Yoshihiro Kodama, et al.. (2020). Cancer Stem Cell Microenvironment Models with Biomaterial Scaffolds In Vitro. Processes. 9(1). 45–45. 11 indexed citations
6.
Ishii, Hiroko, et al.. (2016). A high-fat diet increases the incidence of mammary cancer inc-Ha-<i>ras</i> proto-oncogene transgenic rats. Journal of Toxicologic Pathology. 30(2). 145–152. 6 indexed citations
7.
Ishii, Hiroko, Shigeshi Kamikawa, Satoshi Hirohata, et al.. (2015). Eosinophil Cationic Protein Shows Survival Effect on H9c2 Cardiac Myoblast Cells with Enhanced Phosphorylation of ERK and Akt/GSK-3β under Oxidative Stress.. PubMed. 69(3). 145–53. 4 indexed citations
8.
Kawai, Katsuhisa, Masaki Yamaga, Hiroko Ishii, et al.. (2009). Focal adhesion‐localization of START‐GAP1/DLC1 is essential for cell motility and morphology. Genes to Cells. 14(2). 227–241. 22 indexed citations
9.
Tanooka, Hiroshi, Hideo Tsuji, Takanori Katsube, et al.. (2009). Mutant mouse p53 transgene elevates the chemical induction of tumors that respond to gene silencing with siRNA. Cancer Gene Therapy. 17(1). 1–10. 1 indexed citations
10.
Nishimura, Masaharu, Shigeharu Wakana, Shizuko Kakinuma, et al.. (1999). Low Frequency of Ras Gene Mutation in Spontaneous and Gamma-Ray-Induced Thymic Lymphomas of Scid Mice. Radiation Research. 151(2). 142–142. 13 indexed citations
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13.
Ishii, Hiroko, Yukiko Hayashi, Ikuya Nonaka, & Kiichi Arahata. (1997). Electron microscopic examination of basal lamina in Fukuyama congenital muscular dystrophy. Neuromuscular Disorders. 7(3). 191–197. 45 indexed citations
14.
Ogiu, Toshiaki, et al.. (1996). Relation of Leakiness and Development of Thymic Lymphomas in Radiosensitive C. B-17/scid Mice. Journal of Radiation Research. 37(4). 380. 3 indexed citations
15.
Iijima, Takehiko, Hiroki Sawa, Yoshiaki Shiokawa, et al.. (1996). Thromboxane Synthetase Inhibitor Ameliorates Delayed Neuronal Death in the CA1 Subfield of the Hippocampus after Transient Global Ischemia in Gerbils. Journal of Neurosurgical Anesthesiology. 8(3). 237–242. 11 indexed citations
16.
Arahata, Kiichi, Hiroko Ishii, & Yukiko Hayashi. (1995). Congenital muscular dystrophies. Current Opinion in Neurology. 8(5). 385–390. 26 indexed citations
17.
Arahata, Kiichi, Yukiko Hayashi, Ritsuko Koga, et al.. (1993). Laminin in Animal Models for Muscular Dystrophy Defect of Laminin M in Skeletal and Cardiac Muscles and Peripheral Nerve of the Homozygous Dystrophic dy/dy Mice.. Proceedings of the Japan Academy Series B. 69(10). 259–264. 84 indexed citations
18.
Ishii, Hiroko, Kenichi Sato, Kazuki Hoshino, et al.. (1991). Active efflux of ofloxacin by a highly quinolone-resistant strain of Proteus vulgaris. Journal of Antimicrobial Chemotherapy. 28(6). 827–836. 31 indexed citations
19.
Woo, Man, et al.. (1987). Muscle fiber growth and necrosis in dystrophic muscles: a comparative study between dy and mdx mice. Journal of the Neurological Sciences. 82(1-3). 111–122. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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