Hilla Weidberg

3.2k total citations · 2 hit papers
17 papers, 2.5k citations indexed

About

Hilla Weidberg is a scholar working on Molecular Biology, Epidemiology and Cell Biology. According to data from OpenAlex, Hilla Weidberg has authored 17 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Epidemiology and 6 papers in Cell Biology. Recurrent topics in Hilla Weidberg's work include Autophagy in Disease and Therapy (10 papers), Mitochondrial Function and Pathology (4 papers) and Cellular transport and secretion (4 papers). Hilla Weidberg is often cited by papers focused on Autophagy in Disease and Therapy (10 papers), Mitochondrial Function and Pathology (4 papers) and Cellular transport and secretion (4 papers). Hilla Weidberg collaborates with scholars based in Israel, Canada and United States. Hilla Weidberg's co-authors include Zvulun Elazar, Elena Shvets, Tomer Shpilka, Frida Shimron, Angelika Amon, Shmuel Pietrokovski, Vera Shinder, Adi Abada, Ruth Scherz‐Shouval and Moshe Oren and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Hilla Weidberg

17 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

LC3 and GATE‐16/GABARAP subfamilies are both essential yet act differently in autophagosome biogenesis

2010 597 citations Hilla Weidberg, Elena Shvets et al. The EMBO Journal profile →
Atg8: an autophagy-related ubiquitin-like protein family

2011 405 citations Tomer Shpilka, Hilla Weidberg et al. Genome Biology profile →

Peers

Hilla Weidberg

EPIDE

MB

CB

PHYSI

Tomer Shpilka Israel

Anne Petiot France

Jiefei Geng United States

Wei‐Pang Huang Taiwan

Doris Popovic Germany

Alessandra Romagnoli Italy

Chieko Kishi‐Itakura United Kingdom

Alexandra Stolz Germany

Tomohiro Yorimitsu Japan

Maria Manifava United Kingdom

Tomer Shpilka Israel

Hilla Weidberg

1.7k ×1.0

EPIDE

1.3k ×1.0

MB

813 ×1.1

CB

300 ×1.0

PHYSI

355 ×1.5

PHYSI

Citations per year, relative to Hilla Weidberg Hilla Weidberg (= 1×) peers Tomer Shpilka

Countries citing papers authored by Hilla Weidberg

Since Specialization

Citations

This map shows the geographic impact of Hilla Weidberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hilla Weidberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hilla Weidberg more than expected).

Fields of papers citing papers by Hilla Weidberg

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hilla Weidberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hilla Weidberg. The network helps show where Hilla Weidberg may publish in the future.

Co-authorship network of co-authors of Hilla Weidberg

This figure shows the co-authorship network connecting the top 25 collaborators of Hilla Weidberg. A scholar is included among the top collaborators of Hilla Weidberg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hilla Weidberg. Hilla Weidberg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

17 of 17 papers shown

1.

Weidberg, Hilla, et al.. (2025). Quality control of un-imported mitochondrial proteins at a glance. Journal of Cell Science. 138(9). 1 indexed citations

2.

Maxwell, Christopher A., et al.. (2024). ATAD1 prevents clogging of TOM and damage caused by un-imported mitochondrial proteins. Cell Reports. 43(8). 114473–114473. 16 indexed citations

3.

Shpilka, Tomer, Yunguang Du, Qiyuan Yang, et al.. (2021). UPRmt scales mitochondrial network expansion with protein synthesis via mitochondrial import in Caenorhabditis elegans. Nature Communications. 12(1). 479–479. 73 indexed citations

4.

Weidberg, Hilla & Angelika Amon. (2018). MitoCPR—A surveillance pathway that protects mitochondria in response to protein import stress. Science. 360(6385). 255 indexed citations

5.

Rapaport, Debora, Boris Fichtman, Hilla Weidberg, Eli Sprecher, & Mia Horowitz. (2018). NEK3-mediated SNAP29 phosphorylation modulates its membrane association and SNARE fusion dependent processes. Biochemical and Biophysical Research Communications. 497(2). 605–611. 7 indexed citations

6.

Weidberg, Hilla, Fabien Moretto, Gianpiero Spedale, Angelika Amon, & Folkert J. van Werven. (2016). Nutrient Control of Yeast Gametogenesis Is Mediated by TORC1, PKA and Energy Availability. PLoS Genetics. 12(6). e1006075–e1006075. 34 indexed citations

7.

Benjamin, Sigi, et al.. (2012). EHD2 shuttles to the nucleus and represses transcription. Biochemical Journal. 444(3). 383–394. 25 indexed citations

8.

Weidberg, Hilla, Tomer Shpilka, Elena Shvets, et al.. (2011). LC3 and GATE-16 N Termini Mediate Membrane Fusion Processes Required for Autophagosome Biogenesis. Developmental Cell. 20(4). 444–454. 257 indexed citations

9.

Shvets, Elena, Adi Abada, Hilla Weidberg, & Zvulun Elazar. (2011). Dissecting the involvement of LC3B and GATE-16 in p62 recruitment into autophagosomes. Autophagy. 7(7). 683–688. 53 indexed citations

10.

Shpilka, Tomer, Hilla Weidberg, Shmuel Pietrokovski, & Zvulun Elazar. (2011). Atg8: an autophagy-related ubiquitin-like protein family. Genome Biology. 12(7). 226–226. 405 indexed citations breakdown →

11.

Benjamin, Sigi, Hilla Weidberg, Debora Rapaport, et al.. (2011). EHD2 mediates trafficking from the plasma membrane by modulating Rac1 activity. Biochemical Journal. 439(3). 433–445. 26 indexed citations

12.

Weidberg, Hilla, Elena Shvets, & Zvulun Elazar. (2011). Biogenesis and Cargo Selectivity of Autophagosomes. Annual Review of Biochemistry. 80(1). 125–156. 374 indexed citations

13.

Weidberg, Hilla & Zvulun Elazar. (2011). TBK1 Mediates Crosstalk Between the Innate Immune Response and Autophagy. Science Signaling. 4(187). pe39–pe39. 117 indexed citations

14.

Weidberg, Hilla, Elena Shvets, Tomer Shpilka, et al.. (2010). LC3 and GATE‐16/GABARAP subfamilies are both essential yet act differently in autophagosome biogenesis. The EMBO Journal. 29(11). 1792–1802. 597 indexed citations breakdown →

15.

Weidberg, Hilla, Tomer Shpilka, Elena Shvets, & Zvulun Elazar. (2010). Mammalian Atg8s: One is simply not enough. Autophagy. 6(6). 808–809. 11 indexed citations

16.

Scherz‐Shouval, Ruth, et al.. (2010). p53-dependent regulation of autophagy protein LC3 supports cancer cell survival under prolonged starvation. Proceedings of the National Academy of Sciences. 107(43). 18511–18516. 193 indexed citations

17.

Weidberg, Hilla, Elena Shvets, & Zvulun Elazar. (2009). Lipophagy: Selective Catabolism Designed for Lipids. Developmental Cell. 16(5). 628–630. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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