Hengjiang Guo

842 total citations
14 papers, 647 citations indexed

About

Hengjiang Guo is a scholar working on Nephrology, Molecular Biology and Cell Biology. According to data from OpenAlex, Hengjiang Guo has authored 14 papers receiving a total of 647 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Nephrology, 5 papers in Molecular Biology and 4 papers in Cell Biology. Recurrent topics in Hengjiang Guo's work include Chronic Kidney Disease and Diabetes (6 papers), Endoplasmic Reticulum Stress and Disease (4 papers) and Autophagy in Disease and Therapy (3 papers). Hengjiang Guo is often cited by papers focused on Chronic Kidney Disease and Diabetes (6 papers), Endoplasmic Reticulum Stress and Disease (4 papers) and Autophagy in Disease and Therapy (3 papers). Hengjiang Guo collaborates with scholars based in China and United States. Hengjiang Guo's co-authors include Aili Cao, Yunman Wang, Wen Peng, Li Wang, Xuemei Zhang, Shuang Chu, Yi Wang, Cheng Liu, Bingbing Zhu and Xingmei Yao and has published in prestigious journals such as Scientific Reports, Biochemical and Biophysical Research Communications and Free Radical Biology and Medicine.

In The Last Decade

Hengjiang Guo

14 papers receiving 643 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hengjiang Guo China 13 247 196 135 89 76 14 647
Yuan Fa-huan China 16 393 1.6× 204 1.0× 162 1.2× 137 1.5× 121 1.6× 33 886
Jinying Wei China 13 331 1.3× 128 0.7× 92 0.7× 48 0.5× 76 1.0× 22 647
Yi‐Gang Wan China 18 472 1.9× 332 1.7× 107 0.8× 37 0.4× 75 1.0× 65 999
Yanbin Gao China 16 446 1.8× 197 1.0× 94 0.7× 36 0.4× 64 0.8× 29 788
Shanhua Xu South Korea 9 285 1.2× 131 0.7× 123 0.9× 75 0.8× 131 1.7× 10 695
Lianbo Wei China 15 340 1.4× 112 0.6× 121 0.9× 44 0.5× 55 0.7× 33 731
Lihua Li China 17 306 1.2× 81 0.4× 104 0.8× 32 0.4× 79 1.0× 42 717
Guijun Qin China 19 527 2.1× 167 0.9× 107 0.8× 26 0.3× 83 1.1× 49 936

Countries citing papers authored by Hengjiang Guo

Since Specialization
Citations

This map shows the geographic impact of Hengjiang Guo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hengjiang Guo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hengjiang Guo more than expected).

Fields of papers citing papers by Hengjiang Guo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hengjiang Guo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hengjiang Guo. The network helps show where Hengjiang Guo may publish in the future.

Co-authorship network of co-authors of Hengjiang Guo

This figure shows the co-authorship network connecting the top 25 collaborators of Hengjiang Guo. A scholar is included among the top collaborators of Hengjiang Guo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hengjiang Guo. Hengjiang Guo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Xie, Hongyan, Li Lu, Li Lu, et al.. (2024). Uremic Toxin Receptor AhR Facilitates Renal Senescence and Fibrosis via Suppressing Mitochondrial Biogenesis. Advanced Science. 11(33). e2402066–e2402066. 17 indexed citations
2.
Shen, Qian, Ji Fang, Hengjiang Guo, et al.. (2023). Astragaloside IV attenuates podocyte apoptosis through ameliorating mitochondrial dysfunction by up-regulated Nrf2-ARE/TFAM signaling in diabetic kidney disease. Free Radical Biology and Medicine. 203. 45–57. 60 indexed citations
3.
4.
Li, Jingyao, Xiang Dong Sun, Hongyan Xie, et al.. (2022). Phosphoglycerate mutase 5 initiates inflammation in acute kidney injury by triggering mitochondrial DNA release by dephosphorylating the pro-apoptotic protein Bax. Kidney International. 103(1). 115–133. 50 indexed citations
5.
Yao, Xingmei, et al.. (2022). Klotho Ameliorates Podocyte Injury through Targeting TRPC6 Channel in Diabetic Nephropathy. Journal of Diabetes Research. 2022. 1–13. 12 indexed citations
6.
7.
Guo, Hengjiang, Bingbing Zhu, Ji Fang, et al.. (2020). Klotho ameliorates diabetic nephropathy by activating Nrf2 signaling pathway in podocytes. Biochemical and Biophysical Research Communications. 534. 450–456. 64 indexed citations
8.
9.
Han, Haiyan, Aili Cao, Li Wang, et al.. (2017). Huangqi Decoction Ameliorates Streptozotocin-Induced Rat Diabetic Nephropathy through Antioxidant and Regulation of the TGF-β/MAPK/PPAR-γ Signaling. Cellular Physiology and Biochemistry. 42(5). 1934–1944. 45 indexed citations
10.
Yuan, Zeting, Aili Cao, Hua Liu, et al.. (2017). Calcium Uptake via Mitochondrial Uniporter Contributes to Palmitic Acid-Induced Apoptosis in Mouse Podocytes. Journal of Cellular Biochemistry. 118(9). 2809–2818. 29 indexed citations
11.
Chu, Shuang, Xiaodong Mao, Hengjiang Guo, et al.. (2017). Indoxyl sulfate potentiates endothelial dysfunction via reciprocal role for reactive oxygen species and RhoA/ROCK signaling in 5/6 nephrectomized rats. Free Radical Research. 51(3). 237–252. 28 indexed citations
12.
Cao, Aili, Li Wang, Xia Chen, et al.. (2016). Ursodeoxycholic acid and 4-phenylbutyrate prevent endoplasmic reticulum stress-induced podocyte apoptosis in diabetic nephropathy. Laboratory Investigation. 96(6). 610–622. 107 indexed citations
13.
14.
Cao, Aili, Li Wang, Hengjiang Guo, et al.. (2016). Ursodeoxycholic Acid Ameliorated Diabetic Nephropathy by Attenuating Hyperglycemia-Mediated Oxidative Stress. Biological and Pharmaceutical Bulletin. 39(8). 1300–1308. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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