Hengbing Zu

606 total citations
29 papers, 455 citations indexed

About

Hengbing Zu is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Hengbing Zu has authored 29 papers receiving a total of 455 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 10 papers in Physiology and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in Hengbing Zu's work include Alzheimer's disease research and treatments (10 papers), Tryptophan and brain disorders (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Hengbing Zu is often cited by papers focused on Alzheimer's disease research and treatments (10 papers), Tryptophan and brain disorders (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Hengbing Zu collaborates with scholars based in China and United States. Hengbing Zu's co-authors include Kai Yao, Kai Yao, Yongfei Zhao, Jianfeng Zhang, Xiaojing Bai, Lijie Duan, Xiong Lu, Wenbin Wan, Yue Huang and Zigao Wang and has published in prestigious journals such as SHILAP Revista de lepidopterología, Journal of Lipid Research and Life Sciences.

In The Last Decade

Hengbing Zu

25 papers receiving 449 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hengbing Zu China 13 199 158 124 68 61 29 455
Anaïs Mounier United States 10 207 1.0× 135 0.9× 85 0.7× 32 0.5× 36 0.6× 10 407
Kenji Kawabe Japan 13 161 0.8× 117 0.7× 128 1.0× 71 1.0× 23 0.4× 23 486
André F. Batista Brazil 7 238 1.2× 356 2.3× 189 1.5× 69 1.0× 51 0.8× 9 690
Evangelina Ávila-Muñoz Mexico 8 159 0.8× 173 1.1× 133 1.1× 65 1.0× 27 0.4× 9 447
Oscar Hidalgo‐Lanussa Colombia 14 251 1.3× 184 1.2× 109 0.9× 31 0.5× 19 0.3× 20 617
Joseph M. Valentine United States 9 267 1.3× 396 2.5× 139 1.1× 33 0.5× 28 0.5× 11 669
Hanwoong Woo South Korea 9 176 0.9× 98 0.6× 127 1.0× 67 1.0× 13 0.2× 11 487
Cody M. Wolfe United States 12 288 1.4× 291 1.8× 251 2.0× 49 0.7× 67 1.1× 20 715
Alessandro Di Spiezio Germany 11 158 0.8× 318 2.0× 149 1.2× 31 0.5× 30 0.5× 12 620
Jesús Rodríguez Moreno Spain 12 245 1.2× 217 1.4× 135 1.1× 60 0.9× 23 0.4× 24 681

Countries citing papers authored by Hengbing Zu

Since Specialization
Citations

This map shows the geographic impact of Hengbing Zu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hengbing Zu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hengbing Zu more than expected).

Fields of papers citing papers by Hengbing Zu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hengbing Zu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hengbing Zu. The network helps show where Hengbing Zu may publish in the future.

Co-authorship network of co-authors of Hengbing Zu

This figure shows the co-authorship network connecting the top 25 collaborators of Hengbing Zu. A scholar is included among the top collaborators of Hengbing Zu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hengbing Zu. Hengbing Zu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zu, Hengbing, et al.. (2025). Cellular Cholesterol Loss Impairs Synaptic Vesicle Mobility via the CAMK2/Synapsin-1 Signaling Pathway. Frontiers in Bioscience-Landmark. 30(1). 27111–27111. 2 indexed citations
2.
Mengqi, Zhang, et al.. (2025). DHCR24 deficiency causes AD-like pathology and cognitive decline via the cGAS-STING signaling pathway. Brain Behavior and Immunity. 131. 106150–106150.
3.
4.
Yang, Xiaobo, Kai Yao, Mengqi Zhang, Wenbin Zhang, & Hengbing Zu. (2025). New insight into the role of altered brain cholesterol metabolism in the pathogenesis of AD: A unifying cholesterol hypothesis and new therapeutic approach for AD. Brain Research Bulletin. 224. 111321–111321. 1 indexed citations
5.
Zu, Hengbing, et al.. (2024). The association between plasma trans-fatty acids level and migraine: A cross-sectional study from NHANES 1999–2000. Prostaglandins Leukotrienes and Essential Fatty Acids. 201. 102624–102624.
6.
Zhang, Wenbin, et al.. (2023). DHCR24 reverses Alzheimer’s disease-related pathology and cognitive impairment via increasing hippocampal cholesterol levels in 5xFAD mice. Acta Neuropathologica Communications. 11(1). 102–102. 19 indexed citations
7.
Huang, Yue, et al.. (2023). Cellular cholesterol loss by DHCR24 knockdown leads to Aβ production by changing APP intracellular localization. Journal of Lipid Research. 64(5). 100367–100367. 5 indexed citations
8.
Zhao, Xiuqin, Hengbing Zu, & Kai Yao. (2023). A case report of Andersen-Tawil syndrome misdiagnosed with myodystrophy. Frontiers in Neurology. 14. 1170693–1170693. 1 indexed citations
9.
Bai, Xiaojing, Kai Yao, Yue Huang, et al.. (2022). The role of DHCR24 in the pathogenesis of AD: re-cognition of the relationship between cholesterol and AD pathogenesis. Acta Neuropathologica Communications. 10(1). 35–35. 34 indexed citations
10.
11.
Huang, Fengmin, et al.. (2022). Clinical efficacy and safety of low-dose doxepin in Chinese patients with generalized anxiety disorder: A before–after study. Medicine. 101(42). e31201–e31201. 1 indexed citations
12.
Zhang, Ying, Kai Yao, Yixuan Xu, et al.. (2021). DHCR24 Knockdown Lead to Hyperphosphorylation of Tau at Thr181, Thr231, Ser262, Ser396, and Ser422 Sites by Membrane Lipid-Raft Dependent PP2A Signaling in SH-SY5Y Cells. Neurochemical Research. 46(7). 1627–1640. 13 indexed citations
13.
Zu, Hengbing, Xinying Liu, & Kai Yao. (2020). DHCR24 overexpression modulates microglia polarization and inflammatory response via Akt/GSK3β signaling in Aβ25–35 treated BV-2 cells. Life Sciences. 260. 118470–118470. 21 indexed citations
14.
Tao, Feng, Jie Zhu, Lijie Duan, et al.. (2019). Anti‐inflammatory effects of doxepin hydrochloride against LPS‐induced C6‐glioma cell inflammatory reaction by PI3K‐mediated Akt signaling. Journal of Biochemical and Molecular Toxicology. 34(2). e22424–e22424. 15 indexed citations
15.
Yao, Kai, Yongfei Zhao, & Hengbing Zu. (2019). <p>Melatonin receptor stimulation by agomelatine prevents A&beta;-induced tau phosphorylation and oxidative damage in PC12 cells</p>. Drug Design Development and Therapy. Volume 13. 387–396. 36 indexed citations
16.
Yao, Kai & Hengbing Zu. (2019). Microglial polarization: novel therapeutic mechanism against Alzheimer’s disease. Inflammopharmacology. 28(1). 95–110. 119 indexed citations
17.
Wang, Zigao, Xiong Lu, Guanqun Wang, et al.. (2016). Insulin-like growth factor-1 protects SH-SY5Y cells against β-amyloid-induced apoptosis via the PI3K/Akt-Nrf2 pathway. Experimental Gerontology. 87(Pt A). 23–32. 30 indexed citations
18.
Zu, Hengbing, et al.. (2015). Efficacy and safety evaluation of citalopram and doxepin on sleep quality in comorbid insomnia and anxiety disorders. Experimental and Therapeutic Medicine. 10(4). 1303–1308. 8 indexed citations
19.
20.
Zu, Hengbing, et al.. (2012). Testosterone up-regulates seladin-1 expression by iAR and PI3-K/Akt signaling pathway in C6 cells. Neuroscience Letters. 514(1). 122–126. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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