Heng Wei
- Cancer Research top 5%
- MicroRNA in disease regulation 5
- Cancer Genomics and Diagnostics 2
- Genetics top 10%
- Glioma Diagnosis and Treatment 4
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- Extracellular vesicles in disease 7
- Ginseng Biological Effects and Applications 2
- TGF-β signaling in diseases 2
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- Immune cells in cancer 2
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- Meningioma and schwannoma management 2
- Co-authors
- Michael E. BucklandSusannah HallalKimberley L. KaufmanSaeideh EbrahimkhaniBrindha ShivalingamEugene MaraskovskyJonathan CebonNeil C. Robson
- Journals
- Blood (2 papers)Cancer Research (1 paper)International Journal of Molecular Sciences (2 papers)
- Partner nations
- AustraliaChinaUnited States
In The Last Decade
Heng Wei
18 papers receiving 664 citations
Peers
Comparison fields: 5 of 69
- Cancer Research 317
- Genetics 120
- Molecular Biology 493
- Structural Biology 9
- Immunology 113
Countries citing papers authored by Heng Wei
This map shows the geographic impact of Heng Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heng Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heng Wei more than expected).
Fields of papers citing papers by Heng Wei
This network shows the impact of papers produced by Heng Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heng Wei. The network helps show where Heng Wei may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Heng Wei, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 0 | |
| 3 | 2024 | 3 | |
| 4 | 2024 | 1 | |
| 5 | 2023 | 8 | |
| 6 | 2022 | 18 | |
| 7 | 2022 | 6 | |
| 8 | 2020 | 38 | |
| 9 | 2020 | 41 | |
| 10 | 2020 | 8 | |
| 11 | 2019 | 6 | |
| 12 | 2018 | 62 | |
| 13 | 2018 | 88 | |
| 14 | 2018 | 118 | |
| 15 | 2018 | 1 | |
| 16 | 2016 | 96 | |
| 17 | 2014 | 21 | |
| 18 | [Effects of paeoniflorin on expressions of CTGF, PDGF and TNF-alpha in mice with hepatic fibrosis due to Schistosoma japonicum infection]. | 2011 | 1 |
| 19 | 2009 | 63 | |
| 20 | 2007 | 88 |
About Heng Wei
Heng Wei is a scholar working on Cancer Research, Genetics and Immunology, having authored 20 papers that have together received 667 indexed citations. Recurring topics across this work include Extracellular vesicles in disease (7 papers), MicroRNA in disease regulation (5 papers), Glioma Diagnosis and Treatment (4 papers), Ginseng Biological Effects and Applications (2 papers), TGF-β signaling in diseases (2 papers), Meningioma and schwannoma management (2 papers), Immune cells in cancer (2 papers) and Cancer Genomics and Diagnostics (2 papers). The work is most often cited by research in Cancer Research (317 citations), Genetics (120 citations) and Molecular Biology (493 citations). Heng Wei has collaborated with scholars based in Australia, China and United States. Frequent co-authors include Michael E. Buckland, Susannah Hallal, Kimberley L. Kaufman, Saeideh Ebrahimkhani, Brindha Shivalingam, Eugene Maraskovsky, Jonathan Cebon, Neil C. Robson, Joanne Sy and Richard I. Christopherson. Their work appears in journals such as Blood, Cancer Research and International Journal of Molecular Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.