Hans Christian Probst

5.9k total citations · 1 hit paper
104 papers, 4.1k citations indexed

About

Hans Christian Probst is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Hans Christian Probst has authored 104 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Immunology, 40 papers in Molecular Biology and 16 papers in Oncology. Recurrent topics in Hans Christian Probst's work include Immunotherapy and Immune Responses (36 papers), Immune Cell Function and Interaction (32 papers) and T-cell and B-cell Immunology (30 papers). Hans Christian Probst is often cited by papers focused on Immunotherapy and Immune Responses (36 papers), Immune Cell Function and Interaction (32 papers) and T-cell and B-cell Immunology (30 papers). Hans Christian Probst collaborates with scholars based in Germany, Switzerland and Japan. Hans Christian Probst's co-authors include Maries van den Broek, Volker Gekeler, Kathy D. McCoy, Tasuku Honjo, Taku Okazaki, Reto A. Schwendener, Sabine Muth, George Kollias, Hansjörg Schild and Hideo Yagita∥ and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Hans Christian Probst

103 papers receiving 4.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Resting dendritic cells induce peripheral CD8+ T cell tolerance through PD-1 and CTLA-4

2005 432 citations Hans Christian Probst, Maries van den Broek et al. Nature Immunology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hans Christian Probst Germany	30	2.3k	1.4k	1.1k	337	311	104	4.1k
Maurizio Zanetti United States	36	2.4k 1.0×	1.5k 1.1×	780 0.7×	597 1.8×	308 1.0×	158	4.1k
P C Familletti United States	19	2.5k 1.1×	1.5k 1.1×	981 0.9×	474 1.4×	321 1.0×	19	4.5k
Hironobu Asao Japan	35	2.6k 1.1×	1.4k 1.0×	1.2k 1.2×	314 0.9×	338 1.1×	85	4.5k
Martin R. Hodge United States	29	2.9k 1.3×	1.7k 1.3×	947 0.9×	249 0.7×	391 1.3×	41	4.7k
Junichiro Mizuguchi Japan	39	3.6k 1.6×	1.8k 1.3×	1.4k 1.3×	398 1.2×	283 0.9×	129	5.5k
Siquan Sun United States	26	2.5k 1.1×	947 0.7×	545 0.5×	344 1.0×	249 0.8×	43	3.7k
Subburaj Ilangumaran Canada	35	1.5k 0.6×	1.6k 1.1×	1.1k 1.0×	475 1.4×	306 1.0×	101	3.6k
S F Schlossman United States	38	3.3k 1.4×	1.5k 1.1×	1.3k 1.3×	418 1.2×	252 0.8×	60	5.6k
Ramila Philip United States	28	1.5k 0.6×	1.3k 0.9×	580 0.5×	423 1.3×	160 0.5×	81	3.0k
Viktor Steimle Switzerland	31	3.4k 1.5×	1.3k 0.9×	847 0.8×	323 1.0×	344 1.1×	45	4.4k

Countries citing papers authored by Hans Christian Probst

Since Specialization

Citations

This map shows the geographic impact of Hans Christian Probst's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hans Christian Probst with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hans Christian Probst more than expected).

Fields of papers citing papers by Hans Christian Probst

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hans Christian Probst. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hans Christian Probst. The network helps show where Hans Christian Probst may publish in the future.

Co-authorship network of co-authors of Hans Christian Probst

This figure shows the co-authorship network connecting the top 25 collaborators of Hans Christian Probst. A scholar is included among the top collaborators of Hans Christian Probst based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hans Christian Probst. Hans Christian Probst is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Backer, Ronald A., Hans Christian Probst, & Björn E. Clausen. (2023). Classical DC2 subsets and monocyte‐derived DC: Delineating the developmental and functional relationship. European Journal of Immunology. 53(3). e2149548–e2149548. 24 indexed citations

Stein, Lara, Danielle Arnold-Schild, Matthias Klein, et al.. (2023). Tumor-infiltrating CCR2+ inflammatory monocytes counteract specific immunotherapy. Frontiers in Immunology. 14. 1267866–1267866. 3 indexed citations

Steinbrink, Kerstin, et al.. (2022). Topical Application of Adenosine A2-Type Receptor Agonists Prevents Contact Hypersensitivity Reactions in Mice by Affecting Skin Dendritic Cells. Journal of Investigative Dermatology. 143(3). 408–418.e6. 1 indexed citations

Hahlbrock, Jennifer, Michael Stassen, Matthias Klein, et al.. (2022). Dithranol as novel co-adjuvant for non-invasive dermal vaccination. npj Vaccines. 7(1). 112–112. 5 indexed citations

Ferreira, Cristina, Marta Baptista, Birte Blankenhaus, et al.. (2020). Type 1 Treg cells promote the generation of CD8+ tissue-resident memory T cells. Nature Immunology. 21(7). 766–776. 78 indexed citations

Uhrín, Pavel, Gernot Schabbauer, Dirk Eulberg, et al.. (2019). Intervention of Inflammatory Monocyte Activity Limits Dermal Fibrosis. Journal of Investigative Dermatology. 139(10). 2144–2153. 15 indexed citations

Muth, Sabine, Björn E. Clausen, Karsten Mahnke, et al.. (2018). Dermal CD207-Negative Migratory Dendritic Cells Are Fully Competent to Prime Protective, Skin Homing Cytotoxic T-Lymphocyte Responses. Journal of Investigative Dermatology. 139(2). 422–429. 10 indexed citations

Stein, Pamela, Gerd Rechtsteiner, Tobias Warger, et al.. (2010). UV Exposure Boosts Transcutaneous Immunization and Improves Tumor Immunity: Cytotoxic T-Cell Priming through the Skin. Journal of Investigative Dermatology. 131(1). 211–219. 22 indexed citations

Garbi, Natalio, Günter J. Hämmerling, Hans Christian Probst, & Maries van den Broek. (2010). Tonic T cell signalling and T cell tolerance as opposite effects of self-recognition on dendritic cells. Current Opinion in Immunology. 22(5). 601–608. 35 indexed citations

10.

Schildknecht, Anita, Katharina Lahl, Hansjörg Schild, et al.. (2009). FoxP3 ⁺ regulatory T cells essentially contribute to peripheral CD8 ⁺ T-cell tolerance induced by steady-state dendritic cells. Proceedings of the National Academy of Sciences. 107(1). 199–203. 79 indexed citations

11.

Lang, Karl S., Mike Recher, Alexander A. Navarini, et al.. (2005). Inverse correlation between IL‐7 receptor expression and CD8 T cell exhaustion during persistent antigen stimulation. European Journal of Immunology. 35(3). 738–745. 146 indexed citations

12.

Probst, Hans Christian, Awen Gallimore, Marianne M. Martinic, et al.. (2003). Immunodominance of an Antiviral Cytotoxic T Cell Response Is Shaped by the Kinetics of Viral Protein Expression. The Journal of Immunology. 171(10). 5415–5422. 90 indexed citations

13.

Steinbach, Jörg, et al.. (2003). Hypoxia-induced cell death in human malignant glioma cells: energy deprivation promotes decoupling of mitochondrial cytochrome c release from caspase processing and necrotic cell death. Cell Death and Differentiation. 10(7). 823–832. 97 indexed citations

14.

Probst, Hans Christian, Tilman Dumrese, & Maries van den Broek. (2002). Cutting Edge: Competition for APC by CTLs of Different Specificities Is Not Functionally Important During Induction of Antiviral Responses. The Journal of Immunology. 168(11). 5387–5391. 37 indexed citations

15.

Probst, Hans Christian, et al.. (2002). Re‐oxygenation of hypoxic simian virus 40 (SV40)‐infected CV1 cells causes distinct changes of SV40 minichromosome‐associated replication proteins. European Journal of Biochemistry. 269(9). 2383–2393. 7 indexed citations

16.

Gekeler, Volker, James F. Beck, A Wilisch, et al.. (1994). Drug-induced changes in the expression of MDR-associated genes: Investigations on cultured cell lines and chemotherapeutically treated leukemias. Annals of Hematology. 69(1). S19–S24. 29 indexed citations

17.

Schulte, Dorothea, et al.. (1992). Cycloheximide inhibits cellular, but not SV40, DNA replication. FEBS Letters. 299(2). 149–154. 10 indexed citations

18.

Gekeler, Volker, et al.. (1992). Reversible shutdown of replicon initiation by transient hypoxia in Ehrlich ascites cells. European Journal of Biochemistry. 210(2). 389–398. 17 indexed citations

19.

Probst, Hans Christian, et al.. (1989). No significant overreplication occurs in Ehrlich ascites cells during and after reversal of hypoxia. Experimental Cell Research. 180(2). 563–568. 6 indexed citations

20.

Probst, Hans Christian, Klaus Hamprecht, & Volker Gekeler. (1983). Replicon initiation frequency and intracellular levels of ATP, ADP, AMP and of diadenosine 5′,5‴-P1,P4-tetraphosphate in Ehrlich ascites cells cultured aerobically and anaerobically. Biochemical and Biophysical Research Communications. 110(2). 688–693. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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