Hannes Todt
- Molecular Biology
- Cellular and Molecular Neuroscience top 5%
- Cardiology and Cardiovascular Medicine top 5%
- Psychiatry and Mental health top 10%
- Pediatrics, Perinatology and Child Health top 10%
- Co-authors
- Karlheinz HilberHarry A. FozzardSamuel C. DudleyXaver KoenigWalter SandtnerGregory M. LipkindRobert J. FrenchJohn W. Kyle
- Topics
- Cardiac electrophysiology and arrhythmias (32 papers)Ion channel regulation and function (30 papers)Neuroscience and Neural Engineering (13 papers)
- Partner nations
- AustriaUnited StatesGermany
In The Last Decade
Hannes Todt
71 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 89
- Molecular Biology 653
- Cellular and Molecular Neuroscience 387
- Cardiology and Cardiovascular Medicine 373
- Psychiatry and Mental health 141
- Pediatrics, Perinatology and Child Health 111
Countries citing papers authored by Hannes Todt
This map shows the geographic impact of Hannes Todt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hannes Todt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hannes Todt more than expected).
Fields of papers citing papers by Hannes Todt
This network shows the impact of papers produced by Hannes Todt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hannes Todt. The network helps show where Hannes Todt may publish in the future.
Co-authorship network of co-authors of Hannes Todt
This figure shows the co-authorship network connecting the top 25 collaborators of Hannes Todt. A scholar is included among the top collaborators of Hannes Todt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hannes Todt. Hannes Todt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 1 | |
| 5 | 4 | |
| 6 | 9 | |
| 7 | 27 | |
| 8 | 7 | |
| 9 | 20 | |
| 10 | 9 | |
| 11 | 49 | |
| 12 | 5 | |
| 13 | 77 | |
| 14 | 3 | |
| 15 | 3 | |
| 16 | 21 | |
| 17 | 4 | |
| 18 | 1 | |
| 19 | 1 | |
| 20 | 118 |
About Hannes Todt
Hannes Todt is a scholar working on Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience and Sensory Systems, having authored 78 papers that have together received 1.0k indexed citations. Recurring topics across this work include Cardiac electrophysiology and arrhythmias (32 papers), Ion channel regulation and function (30 papers) and Neuroscience and Neural Engineering (13 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (387 citations), Cardiology and Cardiovascular Medicine (373 citations) and Sensory Systems (48 citations). Hannes Todt has collaborated with scholars based in Austria, United States and Germany. Frequent co-authors include Karlheinz Hilber, Harry A. Fozzard, Samuel C. Dudley, Xaver Koenig, Walter Sandtner, Gregory M. Lipkind, Robert J. French, John W. Kyle, Vaibhavkumar S. Gawali and Eva Zebedin. Their work appears in journals such as Journal of Biological Chemistry, PLoS ONE and Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.