Hannah Lohner

443 total citations
17 papers, 321 citations indexed

About

Hannah Lohner is a scholar working on Molecular Biology, Immunology and Epidemiology. According to data from OpenAlex, Hannah Lohner has authored 17 papers receiving a total of 321 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Epidemiology. Recurrent topics in Hannah Lohner's work include Inflammasome and immune disorders (6 papers), Autophagy in Disease and Therapy (3 papers) and Cholesterol and Lipid Metabolism (2 papers). Hannah Lohner is often cited by papers focused on Inflammasome and immune disorders (6 papers), Autophagy in Disease and Therapy (3 papers) and Cholesterol and Lipid Metabolism (2 papers). Hannah Lohner collaborates with scholars based in United States, Germany and Hungary. Hannah Lohner's co-authors include Owais M. Bhat, Xinxu Yuan, Pin‐Lan Li, Lei Wang, Huizhi Wang, Junling Ren, Yang Zhang, Ruqiang Liang, Pin‐Lan Li and Shuang Liang and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and The FASEB Journal.

In The Last Decade

Hannah Lohner

17 papers receiving 317 citations

Peers

Hannah Lohner

IMMUN

PHYSI

EPIDE

ONCOL

Bo Qiao China

Fernando Henrique Galvão Tessaro Brazil

Katarzyna Jobin Germany

Yongxu Zhao China

Christopher R. Dunston United Kingdom

Rie Miyamoto Japan

Cynthia Weigel United States

Woon‐Hae Kim South Korea

Yangyan Xiao China

Parastoo Boroumand Canada

Bo Qiao China

Hannah Lohner

218 ×1.2

54 ×0.7

IMMUN

45 ×0.8

PHYSI

31 ×0.6

EPIDE

47 ×1.5

ONCOL

Citations per year, relative to Hannah Lohner Hannah Lohner (= 1×) peers Bo Qiao

Countries citing papers authored by Hannah Lohner

Since Specialization

Citations

This map shows the geographic impact of Hannah Lohner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hannah Lohner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hannah Lohner more than expected).

Fields of papers citing papers by Hannah Lohner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hannah Lohner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hannah Lohner. The network helps show where Hannah Lohner may publish in the future.

Co-authorship network of co-authors of Hannah Lohner

This figure shows the co-authorship network connecting the top 25 collaborators of Hannah Lohner. A scholar is included among the top collaborators of Hannah Lohner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hannah Lohner. Hannah Lohner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Ren, Junling, et al.. (2023). P. gingivalis Infection Upregulates PD-L1 Expression on Dendritic Cells, Suppresses CD8+ T-cell Responses, and Aggravates Oral Cancer. Cancer Immunology Research. 11(3). 290–305. 30 indexed citations

Ren, Junling, et al.. (2022). SGK1 negatively regulates inflammatory immune responses and protects against alveolar bone loss through modulation of TRAF3 activity. Journal of Biological Chemistry. 298(6). 102036–102036. 17 indexed citations

Lohner, Hannah, et al.. (2022). Unbiased analysis of the dorsal root ganglion after peripheral nerve injury: no neuronal loss, no gliosis, but satellite glial cell plasticity. Pain. 164(4). 728–740. 11 indexed citations

Ren, Junling, et al.. (2021). Serum- and Glucocorticoid-Inducible Kinase 1 Promotes Alternative Macrophage Polarization and Restrains Inflammation through FoxO1 and STAT3 Signaling. The Journal of Immunology. 207(1). 268–280. 37 indexed citations

Yuan, Xinxu, Owais M. Bhat, Hannah Lohner, Yang Zhang, & Pin‐Lan Li. (2020). Downregulation of Lysosomal Acid Ceramidase Mediates HMGB1-Induced Migration and Proliferation of Mouse Coronary Arterial Myocytes. Frontiers in Cell and Developmental Biology. 8. 111–111. 13 indexed citations

Das, Dipon, Molly L. Bristol, Claire D. James, et al.. (2020). Human Papillomavirus 16 (HPV16) E2 Repression of TWIST1 Transcription Is a Potential Mediator of HPV16 Cancer Outcomes. mSphere. 5(6). 13 indexed citations

Yuan, Xinxu, Owais M. Bhat, Hannah Lohner, Yang Zhang, & Pin‐Lan Li. (2019). Endothelial acid ceramidase in exosome-mediated release of NLRP3 inflammasome products during hyperglycemia: Evidence from endothelium-specific deletion of Asah1 gene. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1864(12). 158532–158532. 22 indexed citations

Yuan, Xinxu, Owais M. Bhat, Hannah Lohner, Dandan Huang, & Pin‐Lan Li. (2019). Reversal of Endothelial Exosomes‐induced Smooth Muscle Phenotype Transition by 7‐ ketocholesterol Stimulation: Role of NLRP3 Inflammasome Activation. The FASEB Journal. 33(S1). 1 indexed citations

Zhang, Qinghua, Guangbi Li, Xinxu Yuan, et al.. (2019). Podocyte‐Specific Rescuing and Deletion of Acid Ceramidase Gene in Mouse Glomeruli. The FASEB Journal. 33(S1). 1 indexed citations

10.

Bhat, Owais M., Xinxu Yuan, Hannah Lohner, & Pin‐Lan Li. (2019). Lysosome Dysfunction and Medial Calcification in the Arterial Wall of Smooth Muscle Cell‐Specific Smpd1 Transgenic Mice: A Ceramide‐Mediated Vasculopathy. The FASEB Journal. 33(S1). 2 indexed citations

11.

Yuan, Xinxu, Lei Wang, Owais M. Bhat, Hannah Lohner, & Pin‐Lan Li. (2018). Differential effects of short chain fatty acids on endothelial Nlrp3 inflammasome activation and neointima formation: Antioxidant action of butyrate. Redox Biology. 16. 21–31. 118 indexed citations

12.

Yuan, Xinxu, Owais M. Bhat, Nan Meng, Hannah Lohner, & Pin‐Lan Li. (2018). Protective Role of Autophagy in Nlrp3 Inflammasome Activation and Medial Thickening of Mouse Coronary Arteries. American Journal Of Pathology. 188(12). 2948–2959. 31 indexed citations

13.

Yuan, Xinxu, Owais M. Bhat, Hannah Lohner, et al.. (2018). Inhibitory effects of growth differentiation factor 11 on autophagy deficiency-induced dedifferentiation of arterial smooth muscle cells. American Journal of Physiology-Heart and Circulatory Physiology. 316(2). H345–H356. 16 indexed citations

14.

Bhat, Owais M., Xinxu Yuan, Yang Zhang, et al.. (2018). Enhanced Arterial Medial Calcification in Mice with Smooth Muscle‐Specific Deletion of Lysosomal Acid Ceramidase.. The FASEB Journal. 32(S1). 1 indexed citations

15.

Yuan, Xinxu, Lei Wang, Owais M. Bhat, et al.. (2017). Differential Effects of Short Chain Fatty Acids on Endothelial NLRP3 Inflammasome Activation and Neointima Formation. The FASEB Journal. 31(S1). 1 indexed citations

16.

Bhat, Owais M., Nan Meng, Xinxu Yuan, Hannah Lohner, & Pin‐Lan Li. (2017). Protective Role of Autophagy in NLRP3 Inflammasome Activation and Medial Thickening of Mouse Coronary Arteries. The FASEB Journal. 31(S1). 1 indexed citations

17.

Gallyas, Ferenc, Hannah Lohner, & Joachim Wolff. (1990). An argyrophil-III method for the selective demonstration of mitochondria in aldehyde-fixed rat brain. Journal of Neuroscience Methods. 35(3). 223–228. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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