Han-Li Huang

724 total citations
18 papers, 387 citations indexed

About

Han-Li Huang is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Han-Li Huang has authored 18 papers receiving a total of 387 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 8 papers in Oncology and 4 papers in Cancer Research. Recurrent topics in Han-Li Huang's work include Histone Deacetylase Inhibitors Research (8 papers), Peptidase Inhibition and Analysis (5 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). Han-Li Huang is often cited by papers focused on Histone Deacetylase Inhibitors Research (8 papers), Peptidase Inhibition and Analysis (5 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). Han-Li Huang collaborates with scholars based in Taiwan, United States and China. Han-Li Huang's co-authors include Shiow‐Lin Pan, Jing‐Ping Liou, Wei‐Chun HuangFu, Che‐Ming Teng, Mei-Jung Lai, Ting-Yi Sung, Yi-Min Liu, Min‐Wu Chao, Hsueh‐Yun Lee and Kunal Nepali and has published in prestigious journals such as PLoS ONE, Scientific Reports and Journal of Medicinal Chemistry.

In The Last Decade

Han-Li Huang

18 papers receiving 384 citations

Peers

Han-Li Huang
Deepak Bhattarai South Korea
Aakanksha Khandelwal United States
Min‐Wu Chao Taiwan
Jee Sun Yang South Korea
Matthew W. Martin United States
Koc-Kan Ho United States
Shengyong Yang China
Wenjian Min China
Pondy Murugappan Ramanujulu Singapore
Dinesh Chimmanamada United States
Deepak Bhattarai South Korea
Han-Li Huang
Citations per year, relative to Han-Li Huang Han-Li Huang (= 1×) peers Deepak Bhattarai

Countries citing papers authored by Han-Li Huang

Since Specialization
Citations

This map shows the geographic impact of Han-Li Huang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Han-Li Huang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Han-Li Huang more than expected).

Fields of papers citing papers by Han-Li Huang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Han-Li Huang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Han-Li Huang. The network helps show where Han-Li Huang may publish in the future.

Co-authorship network of co-authors of Han-Li Huang

This figure shows the co-authorship network connecting the top 25 collaborators of Han-Li Huang. A scholar is included among the top collaborators of Han-Li Huang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Han-Li Huang. Han-Li Huang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Bagchi, Sreya, Robert Yuan, Han-Li Huang, et al.. (2024). The acid-sensing receptor GPR65 on tumor macrophages drives tumor growth in obesity. Science Immunology. 9(100). eadg6453–eadg6453. 10 indexed citations
2.
Sung, Ting-Yi, Han-Li Huang, Po‐Li Wei, et al.. (2021). EGFL6 promotes colorectal cancer cell growth and mobility and the anti‐cancer property of anti-EGFL6 antibody. Cell & Bioscience. 11(1). 53–53. 11 indexed citations
3.
Chen, Liang‐Chieh, Han-Li Huang, Tony Eight Lin, et al.. (2021). Investigation of Selected Flavonoid Derivatives as Potent FLT3 Inhibitors for the Potential Treatment of Acute Myeloid Leukemia. Journal of Natural Products. 84(1). 1–10. 18 indexed citations
4.
Ojha, Ritu, Kunal Nepali, Chun‐Han Chen, et al.. (2020). Isoindoline scaffold-based dual inhibitors of HDAC6 and HSP90 suppressing the growth of lung cancer in vitro and in vivo. European Journal of Medicinal Chemistry. 190. 112086–112086. 35 indexed citations
5.
Chen, Liang‐Chieh, Han-Li Huang, Wei‐Chun HuangFu, et al.. (2020). Biological Evaluation of Selected Flavonoids as Inhibitors of MNKs Targeting Acute Myeloid Leukemia. Journal of Natural Products. 83(10). 2967–2975. 24 indexed citations
6.
Huang, Han-Li, Yi-Min Liu, Ting-Yi Sung, et al.. (2019). TIMP3 expression associates with prognosis in colorectal cancer and its novel arylsulfonamide inducer, MPT0B390, inhibits tumor growth, metastasis and angiogenesis. Theranostics. 9(22). 6676–6689. 30 indexed citations
7.
Ojha, Ritu, Han-Li Huang, Wei‐Chun HuangFu, et al.. (2018). 1-Aroylindoline-hydroxamic acids as anticancer agents, inhibitors of HSP90 and HDAC. European Journal of Medicinal Chemistry. 150. 667–677. 48 indexed citations
8.
Chao, Min‐Wu, Han-Li Huang, Yu-Wei Chang, et al.. (2017). Lanatoside C, a cardiac glycoside, acts through protein kinase Cδ to cause apoptosis of human hepatocellular carcinoma cells. Scientific Reports. 7(1). 46134–46134. 35 indexed citations
9.
Liu, Yi-Min, Wei‐Chun HuangFu, Han-Li Huang, et al.. (2017). 1,4-Naphthoquinones as inhibitors of Itch, a HECT domain-E3 ligase, and tumor growth suppressors in multiple myeloma. European Journal of Medicinal Chemistry. 140. 84–91. 16 indexed citations
10.
Mehndiratta, Samir, et al.. (2017). 4-Indolyl- N -hydroxyphenylacrylamides as potent HDAC class I and IIB inhibitors in vitro and in vivo. European Journal of Medicinal Chemistry. 134. 13–23. 20 indexed citations
11.
Chao, Min‐Wu, Han-Li Huang, Wei‐Chun HuangFu, et al.. (2017). An oral quinoline derivative, MPT0B392, causes leukemic cells mitotic arrest and overcomes drug resistant cancer cells. Oncotarget. 8(17). 27772–27785. 7 indexed citations
12.
Hsu, En‐Chi, Samuel K. Kulp, Han-Li Huang, et al.. (2016). Integrin-linked kinase as a novel molecular switch of the IL-6-NF-κB signaling loop in breast cancer. Carcinogenesis. 37(4). 430–442. 20 indexed citations
13.
Huang, Han-Li, Min‐Wu Chao, Chun‐Han Chen, et al.. (2016). MPT0G066, a novel anti-mitotic drug, induces JNK-independent mitotic arrest, JNK-mediated apoptosis and potentiates antineoplastic effect of cisplatin in ovarian cancer. Scientific Reports. 6(1). 31664–31664. 15 indexed citations
14.
Huang, Han-Li, Min‐Wu Chao, Mei-Chuan Chen, et al.. (2016). LTP-1, a novel antimitotic agent and Stat3 inhibitor, inhibits human pancreatic carcinomas in vitro and in vivo. Scientific Reports. 6(1). 27794–27794. 9 indexed citations
15.
Lee, Hsueh‐Yun, C.S. Chang, Han-Li Huang, et al.. (2016). 2-(Phenylsulfonyl)quinoline N -hydroxyacrylamides as potent anticancer agents inhibiting histone deacetylase. European Journal of Medicinal Chemistry. 122. 92–101. 32 indexed citations
16.
Huang, Han-Li, et al.. (2016). TW-01, a piperazinedione-derived compound, inhibits Ras-mediated cell proliferation and angioplasty-induced vascular restenosis. Toxicology and Applied Pharmacology. 305. 194–202. 1 indexed citations
17.
Lai, Mei-Jung, Han-Li Huang, Shiow‐Lin Pan, et al.. (2012). Synthesis and Biological Evaluation of 1-Arylsulfonyl-5-(N-hydroxyacrylamide)indoles as Potent Histone Deacetylase Inhibitors with Antitumor Activity in Vivo. Journal of Medicinal Chemistry. 55(8). 3777–3791. 55 indexed citations
18.
Huang, Han-Li, Hsueh‐Yun Lee, Chieh-Yu Peng, et al.. (2012). Correction: Anticancer Activity of MPT0E028, a Novel Potent Histone Deacetylase Inhibitor, in Human Colorectal Cancer HCT116 Cells In Vitro and In Vivo. PLoS ONE. 7(9). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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