Hanako Ishida
- Molecular Biology
- Immunology top 10%
- Epidemiology
- Plant Science
- Ecology, Evolution, Behavior and Systematics top 10%
- Co-authors
- Toshiyuki ShimizuUmeharu OhtoKensuke MiyakeTakuma ShibataElena KrayukhinaSusumu UchiyamaHiromi TanjiZhikuan Zhang
- Topics
- Immune Response and Inflammation (5 papers)Protein Structure and Dynamics (4 papers)interferon and immune responses (2 papers)
- Journals
- NatureProceedings of the National Academy of SciencesAngewandte Chemie International Edition
- Partner nations
- JapanSwedenUnited States
In The Last Decade
Hanako Ishida
17 papers receiving 954 citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 435
- Immunology 403
- Epidemiology 153
- Plant Science 149
- Ecology, Evolution, Behavior and Systematics 70
Countries citing papers authored by Hanako Ishida
This map shows the geographic impact of Hanako Ishida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hanako Ishida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hanako Ishida more than expected).
Fields of papers citing papers by Hanako Ishida
This network shows the impact of papers produced by Hanako Ishida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hanako Ishida. The network helps show where Hanako Ishida may publish in the future.
Co-authorship network of co-authors of Hanako Ishida
This figure shows the co-authorship network connecting the top 25 collaborators of Hanako Ishida. A scholar is included among the top collaborators of Hanako Ishida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hanako Ishida. Hanako Ishida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 81 | |
| 4 | 72 | |
| 5 | 57 | |
| 6 | 5 | |
| 7 | 67 | |
| 8 | 115 | |
| 9 | 15 | |
| 10 | 1 | |
| 11 | 106 | |
| 12 | 290 | |
| 13 | 5 | |
| 14 | 39 | |
| 15 | 14 | |
| 16 | 22 | |
| 17 | 66 | |
| 18 | [CpG motif in DNA from immune complexes of SLE patients augments expression of intercellular adhesion molecule-1 on endothelial cells]. | 4 |
About Hanako Ishida
Hanako Ishida is a scholar working on Immunology, Microbiology and Molecular Biology, having authored 18 papers that have together received 962 indexed citations. Recurring topics across this work include Immune Response and Inflammation (5 papers), Protein Structure and Dynamics (4 papers) and interferon and immune responses (2 papers). The work is most often cited by research in Immunology (403 citations), Microbiology (57 citations) and Reproductive Medicine (59 citations). Hanako Ishida has collaborated with scholars based in Japan, Sweden and United States. Frequent co-authors include Toshiyuki Shimizu, Umeharu Ohto, Kensuke Miyake, Takuma Shibata, Elena Krayukhina, Susumu Uchiyama, Hiromi Tanji, Zhikuan Zhang, Ryota Sato and Naokazu Inoue. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Angewandte Chemie International Edition.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.