Hamza Celik

1.4k total citations
23 papers, 929 citations indexed

About

Hamza Celik is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Hamza Celik has authored 23 papers receiving a total of 929 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 18 papers in Hematology and 11 papers in Genetics. Recurrent topics in Hamza Celik's work include Acute Myeloid Leukemia Research (17 papers), Epigenetics and DNA Methylation (12 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (10 papers). Hamza Celik is often cited by papers focused on Acute Myeloid Leukemia Research (17 papers), Epigenetics and DNA Methylation (12 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (10 papers). Hamza Celik collaborates with scholars based in United States, Canada and Cyprus. Hamza Celik's co-authors include Grant A. Challen, Ashley C. Kramer, Andrew Martens, Cates Mallaney, Elizabeth L. Ostrander, Margaret A. Goodell, Blanca Rodríguez, Mira Jeong, James I. McDonald and John R. Edwards and has published in prestigious journals such as Blood, Cancer Cell and Cell stem cell.

In The Last Decade

Hamza Celik

21 papers receiving 922 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hamza Celik United States 12 746 386 163 104 102 23 929
Minh Nguyen United States 13 763 1.0× 333 0.9× 115 0.7× 83 0.8× 189 1.9× 23 1.1k
Joshua T. Trujillo United States 8 852 1.1× 717 1.9× 86 0.5× 78 0.8× 77 0.8× 10 1.1k
Ryoichi Ono Japan 13 584 0.8× 428 1.1× 107 0.7× 69 0.7× 49 0.5× 22 815
Tamara Lamprecht United States 13 840 1.1× 836 2.2× 175 1.1× 129 1.2× 147 1.4× 20 1.3k
Tanya Rozovskaia Israel 11 1.5k 2.1× 366 0.9× 61 0.4× 195 1.9× 93 0.9× 11 1.7k
Maike Schwieger Germany 13 426 0.6× 287 0.7× 58 0.4× 116 1.1× 115 1.1× 14 631
Emily Whisenant United States 7 734 1.0× 719 1.9× 87 0.5× 66 0.6× 81 0.8× 11 990
Manami Maeda United States 6 424 0.6× 140 0.4× 236 1.4× 47 0.5× 136 1.3× 13 622
Michael Lie‐A‐Ling United Kingdom 16 650 0.9× 178 0.5× 33 0.2× 94 0.9× 180 1.8× 27 916
Michelle Giehl Germany 10 236 0.3× 206 0.5× 150 0.9× 58 0.6× 99 1.0× 15 549

Countries citing papers authored by Hamza Celik

Since Specialization
Citations

This map shows the geographic impact of Hamza Celik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hamza Celik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hamza Celik more than expected).

Fields of papers citing papers by Hamza Celik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hamza Celik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hamza Celik. The network helps show where Hamza Celik may publish in the future.

Co-authorship network of co-authors of Hamza Celik

This figure shows the co-authorship network connecting the top 25 collaborators of Hamza Celik. A scholar is included among the top collaborators of Hamza Celik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hamza Celik. Hamza Celik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wilson, William C., Andrew L. Young, Eunice S. Wang, et al.. (2024). BIRC5 upregulation enhances DNMT3A-mutant T-ALL cell survival and pathogenesis. PubMed. 1(4). 100040–100040. 1 indexed citations
2.
Celik, Hamza, Maryanne Covington, Katherine Drake, et al.. (2023). Comparison of the Enzymatic and Cellular Profiles of Clinical JAK2 Inhibitors for the Treatment of Myelofibrosis. Blood. 142(Supplement 1). 4532–4532. 2 indexed citations
3.
Zhang, Christine, Elizabeth L. Ostrander, Cates Mallaney, et al.. (2022). Txnip Enhances Fitness of Dnmt3a -Mutant Hematopoietic Stem Cells via p21. Blood Cancer Discovery. 3(3). 220–239. 13 indexed citations
4.
Kong, Tim, Angelo B. A. Laranjeira, Daniel A.C. Fisher, et al.. (2022). DUSP6 mediates resistance to JAK2 inhibition and drives leukemic progression. Nature Cancer. 4(1). 108–127. 25 indexed citations
5.
Reis, Edimara S., Rebecca A. Buonpane, Hamza Celik, et al.. (2022). Discovery of INCA033989, a Monoclonal Antibody That Selectively Antagonizes Mutant Calreticulin Oncogenic Function in Myeloproliferative Neoplasms (MPNs). Blood. 140(Supplement 1). 14–15. 24 indexed citations
6.
Celik, Hamza, et al.. (2021). The Histone Demethylase KDM6B Is a Genetic Dependency of NOTCH1-Driven T-ALL. Blood. 138(Supplement 1). 782–782. 1 indexed citations
7.
Kong, Tim, Angelo B. A. Laranjeira, Mary C. Fulbright, et al.. (2021). Pevonedistat targets malignant cells in myeloproliferative neoplasms in vitro and in vivo via NFκB pathway inhibition. Blood Advances. 6(2). 611–623. 12 indexed citations
8.
Marinaccio, Christian, Praveen Suraneni, Hamza Celik, et al.. (2020). Loss of LKB1/STK11 Facilitates Leukemic Progression of the Myeloproliferative Neoplasms. Blood. 136(Supplement 1). 1–1. 3 indexed citations
9.
Mallaney, Cates, Elizabeth L. Ostrander, Hamza Celik, et al.. (2019). Kdm6b regulates context-dependent hematopoietic stem cell self-renewal and leukemogenesis. Leukemia. 33(10). 2506–2521. 45 indexed citations
10.
Celik, Hamza, Ashley C. Kramer, Elizabeth L. Ostrander, et al.. (2018). JARID2 Functions as a Tumor Suppressor in Myeloid Neoplasms by Repressing Self-Renewal in Hematopoietic Progenitor Cells. Cancer Cell. 34(5). 741–756.e8. 39 indexed citations
11.
Jeong, Mira, Hyun Jung Park, Hamza Celik, et al.. (2018). Loss of Dnmt3a Immortalizes Hematopoietic Stem Cells In Vivo. Cell Reports. 23(1). 1–10. 151 indexed citations
13.
Kramer, Ashley C., A Kothari, William C. Wilson, et al.. (2017). Dnmt3a regulates T-cell development and suppresses T-ALL transformation. Leukemia. 31(11). 2479–2490. 34 indexed citations
14.
McDonald, James I., Hamza Celik, Ashley C. Kramer, et al.. (2016). Reprogrammable CRISPR/Cas9-based system for inducing site-specific DNA methylation. Biology Open. 5(6). 866–874. 208 indexed citations
15.
Challen, Grant A., et al.. (2016). DNMT3A regulates T-cell development and suppresses T-all transformation. Experimental Hematology. 44(9). S41–S41. 2 indexed citations
16.
Celik, Hamza, et al.. (2016). The Role of JARID2 in Normal and Malignant Hematopoiesis. Blood. 128(22). 793–793. 1 indexed citations
17.
Celik, Hamza, Ashley C. Kramer, & Grant A. Challen. (2016). DNA methylation in normal and malignant hematopoiesis. International Journal of Hematology. 103(6). 617–626. 27 indexed citations
18.
Challen, Grant A., Deqiang Sun, Allison Mayle, et al.. (2014). Dnmt3a and Dnmt3b Have Overlapping and Distinct Functions in Hematopoietic Stem Cells. Cell stem cell. 15(3). 350–364. 259 indexed citations
19.
Celik, Hamza, Cates Mallaney, Christopher A. Miller, et al.. (2014). Enforced Differentiation of Dnmt3a-Null Bone Marrow Leads to Failure with c-Kit Mutations Driving Leukemic Transformation. Blood. 124(21). 837–837. 2 indexed citations
20.
Celik, Hamza, Cates Mallaney, Elizabeth L. Ostrander, et al.. (2014). Enforced differentiation of Dnmt3a-null bone marrow leads to failure with c-Kit mutations driving leukemic transformation. Blood. 125(4). 619–628. 77 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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